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272 related items for PubMed ID: 16267158

  • 1. Hyperaldosteronemia and activation of the epithelial sodium channel are not required for sodium retention in puromycin-induced nephrosis.
    Lourdel S, Loffing J, Favre G, Paulais M, Nissant A, Fakitsas P, Créminon C, Féraille E, Verrey F, Teulon J, Doucet A, Deschênes G.
    J Am Soc Nephrol; 2005 Dec; 16(12):3642-50. PubMed ID: 16267158
    [Abstract] [Full Text] [Related]

  • 2. Epithelial sodium channel (ENaC) subunit mRNA and protein expression in rats with puromycin aminonucleoside-induced nephrotic syndrome.
    Audigé A, Yu ZR, Frey BM, Uehlinger DE, Frey FJ, Vogt B.
    Clin Sci (Lond); 2003 Apr; 104(4):389-95. PubMed ID: 12653683
    [Abstract] [Full Text] [Related]

  • 3. Increased expression and apical targeting of renal ENaC subunits in puromycin aminonucleoside-induced nephrotic syndrome in rats.
    Kim SW, Wang W, Nielsen J, Praetorius J, Kwon TH, Knepper MA, Frøkiaer J, Nielsen S.
    Am J Physiol Renal Physiol; 2004 May; 286(5):F922-35. PubMed ID: 15075188
    [Abstract] [Full Text] [Related]

  • 4. GI262570, a peroxisome proliferator-activated receptor {gamma} agonist, changes electrolytes and water reabsorption from the distal nephron in rats.
    Chen L, Yang B, McNulty JA, Clifton LG, Binz JG, Grimes AM, Strum JC, Harrington WW, Chen Z, Balon TW, Stimpson SA, Brown KK.
    J Pharmacol Exp Ther; 2005 Feb; 312(2):718-25. PubMed ID: 15475592
    [Abstract] [Full Text] [Related]

  • 5. Increased expression but not targeting of ENaC in adrenalectomized rats with PAN-induced nephrotic syndrome.
    de Seigneux S, Kim SW, Hemmingsen SC, Frøkiaer J, Nielsen S.
    Am J Physiol Renal Physiol; 2006 Jul; 291(1):F208-17. PubMed ID: 16403831
    [Abstract] [Full Text] [Related]

  • 6. Sodium retention in rats with liver cirrhosis is associated with increased renal abundance of NaCl cotransporter (NCC).
    Yu Z, Serra A, Sauter D, Loffing J, Ackermann D, Frey FJ, Frey BM, Vogt B.
    Nephrol Dial Transplant; 2005 Sep; 20(9):1833-41. PubMed ID: 15956070
    [Abstract] [Full Text] [Related]

  • 7. Increased renal alpha-ENaC and NCC abundance and elevated blood pressure are independent of hyperaldosteronism in vasopressin escape.
    Tiwari S, Packer RK, Hu X, Sugimura Y, Verbalis JG, Ecelbarger CA.
    Am J Physiol Renal Physiol; 2006 Jul; 291(1):F49-57. PubMed ID: 16449357
    [Abstract] [Full Text] [Related]

  • 8. [Role of tubular Na-K-ATPase in nephrotic syndrome induced by puromycin in the rat].
    Féraille E.
    Nephrologie; 1994 Jul; 15(4):301-5. PubMed ID: 7984251
    [Abstract] [Full Text] [Related]

  • 9. Increased synthesis and avp unresponsiveness of Na,K-ATPase in collecting duct from nephrotic rats.
    Deschênes G, Gonin S, Zolty E, Cheval L, Rousselot M, Martin PY, Verbavatz JM, Féraille E, Doucet A.
    J Am Soc Nephrol; 2001 Nov; 12(11):2241-2252. PubMed ID: 11675400
    [Abstract] [Full Text] [Related]

  • 10. Pathogenesis of oedema in nephrotic syndrome: role of epithelial sodium channel.
    Kim SW, Frøkiaer J, Nielsen S.
    Nephrology (Carlton); 2007 Dec; 12 Suppl 3():S8-10. PubMed ID: 17995529
    [Abstract] [Full Text] [Related]

  • 11. Collecting duct (Na+/K+)-ATPase activity is correlated with urinary sodium excretion in rat nephrotic syndromes.
    Deschênes G, Doucet A.
    J Am Soc Nephrol; 2000 Apr; 11(4):604-615. PubMed ID: 10752519
    [Abstract] [Full Text] [Related]

  • 12. Regulation of epithelial sodium channel in puromycin aminonucleoside-induced unilateral experimental nephrotic syndrome in normal and analbuminemic Nagase rats.
    Yu Z, Schumacher M, Frey BM, Frey FJ, Vogt B.
    Nephron Physiol; 2005 Apr; 101(3):p51-62. PubMed ID: 16020936
    [Abstract] [Full Text] [Related]

  • 13. Altered expression of renal apical plasma membrane Na+ transporters in the early phase of genetic hypertension.
    Capasso G, Rizzo M, Evangelista C, Ferrari P, Geelen G, Lang F, Bianchi G.
    Am J Physiol Renal Physiol; 2005 Jun; 288(6):F1173-82. PubMed ID: 15687246
    [Abstract] [Full Text] [Related]

  • 14. Segmental variability of ENaC subunit expression in rat colon during dietary sodium depletion.
    Greig ER, Baker EH, Mathialahan T, Boot-Handford RP, Sandle GI.
    Pflugers Arch; 2002 Jul; 444(4):476-83. PubMed ID: 12136266
    [Abstract] [Full Text] [Related]

  • 15. Na channel expression and activity in the medullary collecting duct of rat kidney.
    Frindt G, Ergonul Z, Palmer LG.
    Am J Physiol Renal Physiol; 2007 Apr; 292(4):F1190-6. PubMed ID: 17200158
    [Abstract] [Full Text] [Related]

  • 16. Modulation of Na+ transport and epithelial sodium channel expression by protein kinase C in rat alveolar epithelial cells.
    Yamagata T, Yamagata Y, Massé C, Tessier MC, Brochiero E, Dagenais A, Berthiaume Y.
    Can J Physiol Pharmacol; 2005 Nov; 83(11):977-87. PubMed ID: 16391706
    [Abstract] [Full Text] [Related]

  • 17. Jejunal dopamine and Na,K+-ATPase activity in nephrotic syndrome.
    Sampaio-Maia B, Moreira-Rodrigues M, Serrão P, Pestana M.
    Am J Nephrol; 2005 Nov; 25(4):382-92. PubMed ID: 16088079
    [Abstract] [Full Text] [Related]

  • 18. Nephrotic syndrome is associated with increased plasma K+ concentration, intestinal K+ losses, and attenuated urinary K+ excretion: a study in rats and humans.
    Ydegaard R, Svenningsen P, Bistrup C, Andersen RF, Stubbe J, Buhl KB, Marcussen N, Hinrichs GR, Iraqi H, Zamani R, Dimke H, Jensen BL.
    Am J Physiol Renal Physiol; 2019 Dec 01; 317(6):F1549-F1562. PubMed ID: 31566427
    [Abstract] [Full Text] [Related]

  • 19. Role of uroguanylin, a Peptide with natriuretic activity, in rats with experimental nephrotic syndrome.
    Kikuchi M, Fujimoto S, Fukae H, Kinoshita H, Kita T, Nakazato M, Eto T.
    J Am Soc Nephrol; 2005 Feb 01; 16(2):392-7. PubMed ID: 15590761
    [Abstract] [Full Text] [Related]

  • 20. Collecting duct is a site of sodium retention in PAN nephrosis: a rationale for amiloride therapy.
    Deschênes G, Wittner M, Stefano AD, Jounier S, Doucet A.
    J Am Soc Nephrol; 2001 Mar 01; 12(3):598-601. PubMed ID: 11181809
    [Abstract] [Full Text] [Related]

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