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Journal Abstract Search

266 related items for PubMed ID: 16303321

  • 1. Advances in understanding the molecular causes of diabetes-induced birth defects.
    Loeken MR.
    J Soc Gynecol Investig; 2006 Jan; 13(1):2-10. PubMed ID: 16303321
    [Abstract] [Full Text] [Related]

  • 2. Hypoxic stress in diabetic pregnancy contributes to impaired embryo gene expression and defective development by inducing oxidative stress.
    Li R, Chase M, Jung SK, Smith PJ, Loeken MR.
    Am J Physiol Endocrinol Metab; 2005 Oct; 289(4):E591-9. PubMed ID: 15928021
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  • 3. Current perspectives on the causes of neural tube defects resulting from diabetic pregnancy.
    Loeken MR.
    Am J Med Genet C Semin Med Genet; 2005 May 15; 135C(1):77-87. PubMed ID: 15800853
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  • 4. Oxidant regulation of gene expression and neural tube development: Insights gained from diabetic pregnancy on molecular causes of neural tube defects.
    Chang TI, Horal M, Jain SK, Wang F, Patel R, Loeken MR.
    Diabetologia; 2003 Apr 15; 46(4):538-45. PubMed ID: 12739027
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  • 5. AMP-activated protein kinase mediates effects of oxidative stress on embryo gene expression in a mouse model of diabetic embryopathy.
    Wu Y, Viana M, Thirumangalathu S, Loeken MR.
    Diabetologia; 2012 Jan 15; 55(1):245-54. PubMed ID: 21993711
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  • 7. Oxidative stress during diabetic pregnancy disrupts cardiac neural crest migration and causes outflow tract defects.
    Morgan SC, Relaix F, Sandell LL, Loeken MR.
    Birth Defects Res A Clin Mol Teratol; 2008 Jun 15; 82(6):453-63. PubMed ID: 18435457
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  • 10. [Expression of Pax3 and Cx43 in NTD embryos induced by hyperglycemia].
    Mao DW, Zhang YJ, Li QM, Li SR.
    Zhonghua Yi Xue Za Zhi; 2003 Apr 10; 83(7):593-7. PubMed ID: 12887752
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  • 11. Activation of the hexosamine pathway causes oxidative stress and abnormal embryo gene expression: involvement in diabetic teratogenesis.
    Horal M, Zhang Z, Stanton R, Virkamäki A, Loeken MR.
    Birth Defects Res A Clin Mol Teratol; 2004 Aug 10; 70(8):519-27. PubMed ID: 15329829
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  • 12. [Study of teratogenicity of hyperglycemia on neural tube defects and antagonistic effect of taurine].
    Mao DW, Zhao YP, Li SR, Che JH, Tan WH.
    Zhonghua Fu Chan Ke Za Zhi; 2004 Mar 10; 39(3):169-72. PubMed ID: 15130376
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  • 13. Deficiency of the oxidative stress-responsive kinase p70S6K1 restores autophagy and ameliorates neural tube defects in diabetic embryopathy.
    Cao S, Shen WB, Reece EA, Yang P.
    Am J Obstet Gynecol; 2020 Nov 10; 223(5):753.e1-753.e14. PubMed ID: 32416155
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  • 14. Increased DNA methyltransferase 3b (Dnmt3b)-mediated CpG island methylation stimulated by oxidative stress inhibits expression of a gene required for neural tube and neural crest development in diabetic pregnancy.
    Wei D, Loeken MR.
    Diabetes; 2014 Oct 10; 63(10):3512-22. PubMed ID: 24834974
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  • 15. The green tea polyphenol EGCG alleviates maternal diabetes-induced neural tube defects by inhibiting DNA hypermethylation.
    Zhong J, Xu C, Reece EA, Yang P.
    Am J Obstet Gynecol; 2016 Sep 10; 215(3):368.e1-368.e10. PubMed ID: 26979632
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  • 16. Polymorphic susceptibility to the molecular causes of neural tube defects during diabetic embryopathy.
    Pani L, Horal M, Loeken MR.
    Diabetes; 2002 Sep 10; 51(9):2871-4. PubMed ID: 12196484
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  • 17. Free radicals and birth defects.
    Loeken MR.
    J Matern Fetal Neonatal Med; 2004 Jan 10; 15(1):6-14. PubMed ID: 15101606
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  • 18. Global gene expression analysis of cranial neural tubes in embryos of diabetic mice.
    Jiang B, Kumar SD, Loh WT, Manikandan J, Ling EA, Tay SS, Dheen ST.
    J Neurosci Res; 2008 Dec 10; 86(16):3481-93. PubMed ID: 18655203
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  • 19. Modulation of nuclear factor-κB signaling and reduction of neural tube defects by quercetin-3-glucoside in embryos of diabetic mice.
    Tan C, Meng F, Reece EA, Zhao Z.
    Am J Obstet Gynecol; 2018 Aug 10; 219(2):197.e1-197.e8. PubMed ID: 29733843
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