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Journal Abstract Search


322 related items for PubMed ID: 16330765

  • 1. The multidomain proapoptotic molecules Bax and Bak are directly activated by heat.
    Pagliari LJ, Kuwana T, Bonzon C, Newmeyer DD, Tu S, Beere HM, Green DR.
    Proc Natl Acad Sci U S A; 2005 Dec 13; 102(50):17975-80. PubMed ID: 16330765
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  • 2. Bid chimeras indicate that most BH3-only proteins can directly activate Bak and Bax, and show no preference for Bak versus Bax.
    Hockings C, Anwari K, Ninnis RL, Brouwer J, O'Hely M, Evangelista M, Hinds MG, Czabotar PE, Lee EF, Fairlie WD, Dewson G, Kluck RM.
    Cell Death Dis; 2015 Apr 23; 6(4):e1735. PubMed ID: 25906158
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  • 3. Bax/Bak-dependent, Drp1-independent Targeting of X-linked Inhibitor of Apoptosis Protein (XIAP) into Inner Mitochondrial Compartments Counteracts Smac/DIABLO-dependent Effector Caspase Activation.
    Hamacher-Brady A, Brady NR.
    J Biol Chem; 2015 Sep 04; 290(36):22005-18. PubMed ID: 26134559
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  • 4. Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
    Kandasamy K, Srinivasula SM, Alnemri ES, Thompson CB, Korsmeyer SJ, Bryant JL, Srivastava RK.
    Cancer Res; 2003 Apr 01; 63(7):1712-21. PubMed ID: 12670926
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  • 5. Translocation of a Bak C-terminus mutant from cytosol to mitochondria to mediate cytochrome C release: implications for Bak and Bax apoptotic function.
    Ferrer PE, Frederick P, Gulbis JM, Dewson G, Kluck RM.
    PLoS One; 2012 Apr 01; 7(3):e31510. PubMed ID: 22442658
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  • 6. Bax- or Bak-induced mitochondrial fission can be uncoupled from cytochrome C release.
    Sheridan C, Delivani P, Cullen SP, Martin SJ.
    Mol Cell; 2008 Aug 22; 31(4):570-585. PubMed ID: 18722181
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  • 9. BH3 domains other than Bim and Bid can directly activate Bax/Bak.
    Du H, Wolf J, Schafer B, Moldoveanu T, Chipuk JE, Kuwana T.
    J Biol Chem; 2011 Jan 07; 286(1):491-501. PubMed ID: 21041309
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  • 12. An interconnected hierarchical model of cell death regulation by the BCL-2 family.
    Chen HC, Kanai M, Inoue-Yamauchi A, Tu HC, Huang Y, Ren D, Kim H, Takeda S, Reyna DE, Chan PM, Ganesan YT, Liao CP, Gavathiotis E, Hsieh JJ, Cheng EH.
    Nat Cell Biol; 2015 Oct 07; 17(10):1270-81. PubMed ID: 26344567
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  • 13. BH3-only proteins target BCL-xL/MCL-1, not BAX/BAK, to initiate apoptosis.
    Huang K, O'Neill KL, Li J, Zhou W, Han N, Pang X, Wu W, Struble L, Borgstahl G, Liu Z, Zhang L, Luo X.
    Cell Res; 2019 Nov 07; 29(11):942-952. PubMed ID: 31551537
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  • 17. Non-canonical function of Bax in stress-induced nuclear protein redistribution.
    Lindenboim L, Ferrando-May E, Borner C, Stein R.
    Cell Mol Life Sci; 2013 Aug 07; 70(16):3013-27. PubMed ID: 23475110
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  • 19. Direct activation of full-length proapoptotic BAK.
    Leshchiner ES, Braun CR, Bird GH, Walensky LD.
    Proc Natl Acad Sci U S A; 2013 Mar 12; 110(11):E986-95. PubMed ID: 23404709
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  • 20. Subtilase cytotoxin induces apoptosis in HeLa cells by mitochondrial permeabilization via activation of Bax/Bak, independent of C/EBF-homologue protein (CHOP), Ire1alpha or JNK signaling.
    Yahiro K, Morinaga N, Moss J, Noda M.
    Microb Pathog; 2010 Oct 12; 49(4):153-63. PubMed ID: 20561923
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