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Journal Abstract Search

118 related items for PubMed ID: 16411374

  • 21. A diabetes-like environment increases malformation rate and diminishes prostaglandin E(2) in rat embryos: reversal by administration of vitamin E and folic acid.
    Wentzel P, Eriksson UJ.
    Birth Defects Res A Clin Mol Teratol; 2005 Jul; 73(7):506-11. PubMed ID: 15959876
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  • 22. Modulation of nitric oxide concentration and lipid metabolism by 15-deoxy Delta12,14prostaglandin J2 in embryos from control and diabetic rats during early organogenesis.
    Jawerbaum A, Sinner D, White V, Pustovrh C, Capobianco E, Gonzalez E.
    Reproduction; 2002 Nov; 124(5):625-31. PubMed ID: 12417000
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  • 23. Safflower and olive oil dietary treatments rescue aberrant embryonic arachidonic acid and nitric oxide metabolism and prevent diabetic embryopathy in rats.
    Higa R, White V, Martínez N, Kurtz M, Capobianco E, Jawerbaum A.
    Mol Hum Reprod; 2010 Apr; 16(4):286-95. PubMed ID: 20051498
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  • 27. Maternal diabetes in vivo and high glucose concentration in vitro increases apoptosis in rat embryos.
    Gäreskog M, Cederberg J, Eriksson UJ, Wentzel P.
    Reprod Toxicol; 2007 Jan; 23(1):63-74. PubMed ID: 17034987
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  • 28. Expression of enzymes involved in the synthesis of prostaglandin E2 in bovine in vitro-produced embryos.
    Saint-Dizier M, Guyader-Joly C, Charpigny G, Grimard B, Humblot P, Ponter AA.
    Zygote; 2011 Aug; 19(3):277-83. PubMed ID: 21232167
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  • 30. High glucose levels modulate eicosanoid production in uterine and placental tissue from non-insulin-dependent diabetic rats during late pregnancy.
    Jawerbaum A, Novaro V, Franchi AM, Gimeno M, González E.
    Prostaglandins Leukot Essent Fatty Acids; 1998 Jun; 58(6):389-93. PubMed ID: 10189067
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  • 31. Modulation of PGE2 generation in the diabetic embryo: effect of nitric oxide and superoxide dismutase.
    Jawerbaum A, Sinner D, White V, Pustovrh C, Capobianco E, Gimeno MA, Gonzalez ET.
    Prostaglandins Leukot Essent Fatty Acids; 2001 Feb; 64(2):127-33. PubMed ID: 11237480
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  • 32. Oxidative stress and altered prostanoid production in the placenta of streptozotocin-induced diabetic rats.
    White V, Jawerbaum A, Sinner D, Pustovrh C, Capobianco E, González E.
    Reprod Fertil Dev; 2002 Feb; 14(1-2):117-23. PubMed ID: 12051517
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  • 33. Aberrant patterns of cellular communication in diabetes-induced embryopathy in rats: II, apoptotic pathways.
    Reece EA, Ma XD, Zhao Z, Wu YK, Dhanasekaran D.
    Am J Obstet Gynecol; 2005 Mar; 192(3):967-72. PubMed ID: 15746699
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  • 34. Role of reactive oxygen species (ROS) in the diabetes-induced anomalies in rat embryos in vitro: reduction in antioxidant enzymes and low-molecular-weight antioxidants (LMWA) may be the causative factor for increased anomalies.
    Ornoy A, Zaken V, Kohen R.
    Teratology; 1999 Dec; 60(6):376-86. PubMed ID: 10590399
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  • 36. Delay in pulmonary glycogen degradation in fetuses of streptozotocin diabetic rats.
    Gewolb IH, Barrett C, Wilson CM, Warshaw JB.
    Pediatr Res; 1982 Oct; 16(10):869-73. PubMed ID: 7145509
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  • 39. TNF-alpha acts to prevent occurrence of malformed fetuses in diabetic mice.
    Torchinsky A, Gongadze M, Orenstein H, Savion S, Fein A, Toder V.
    Diabetologia; 2004 Jan; 47(1):132-9. PubMed ID: 14647895
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  • 40. Folic acid supplementation affects ROS scavenging enzymes, enhances Vegf-A, and diminishes apoptotic state in yolk sacs of embryos of diabetic rats.
    Zabihi S, Eriksson UJ, Wentzel P.
    Reprod Toxicol; 2007 Jun; 23(4):486-98. PubMed ID: 17482424
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