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Journal Abstract Search

178 related items for PubMed ID: 16420433

  • 1. LTP in the lateral amygdala during cocaine withdrawal.
    Goussakov I, Chartoff EH, Tsvetkov E, Gerety LP, Meloni EG, Carlezon WA, Bolshakov VY.
    Eur J Neurosci; 2006 Jan; 23(1):239-50. PubMed ID: 16420433
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  • 2. Fear memories induce a switch in stimulus response and signaling mechanisms for long-term potentiation in the lateral amygdala.
    Schroeder BW, Shinnick-Gallagher P.
    Eur J Neurosci; 2004 Jul; 20(2):549-56. PubMed ID: 15233764
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  • 3. Cocaine withdrawal enhances long-term potentiation induced by corticotropin-releasing factor at central amygdala glutamatergic synapses via CRF, NMDA receptors and PKA.
    Pollandt S, Liu J, Orozco-Cabal L, Grigoriadis DE, Vale WW, Gallagher JP, Shinnick-Gallagher P.
    Eur J Neurosci; 2006 Sep; 24(6):1733-43. PubMed ID: 17004937
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  • 4. Fear learning induces persistent facilitation of amygdala synaptic transmission.
    Schroeder BW, Shinnick-Gallagher P.
    Eur J Neurosci; 2005 Oct; 22(7):1775-83. PubMed ID: 16197518
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  • 5. Repeated exposure to amphetamine disrupts dopaminergic modulation of excitatory synaptic plasticity and neurotransmission in nucleus accumbens.
    Li Y, Kauer JA.
    Synapse; 2004 Jan; 51(1):1-10. PubMed ID: 14579420
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  • 6. Repeated ethanol exposure and withdrawal impairs human fear conditioning and depresses long-term potentiation in rat amygdala and hippocampus.
    Stephens DN, Ripley TL, Borlikova G, Schubert M, Albrecht D, Hogarth L, Duka T.
    Biol Psychiatry; 2005 Sep 01; 58(5):392-400. PubMed ID: 16018978
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  • 7. Cocaine withdrawal enhances long-term potentiation in rat hippocampus via changing the activity of corticotropin-releasing factor receptor subtype 2.
    Guan X, Zhang R, Xu Y, Li S.
    Neuroscience; 2009 Jul 07; 161(3):665-70. PubMed ID: 19376201
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  • 8. Role of AMPA receptor trafficking in NMDA receptor-dependent synaptic plasticity in the rat lateral amygdala.
    Yu SY, Wu DC, Liu L, Ge Y, Wang YT.
    J Neurochem; 2008 Jul 07; 106(2):889-99. PubMed ID: 18466342
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  • 10. Long-term potentiation (LTP) in the central amygdala (CeA) is enhanced after prolonged withdrawal from chronic cocaine and requires CRF1 receptors.
    Fu Y, Pollandt S, Liu J, Krishnan B, Genzer K, Orozco-Cabal L, Gallagher JP, Shinnick-Gallagher P.
    J Neurophysiol; 2007 Jan 07; 97(1):937-41. PubMed ID: 17079348
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  • 13. Glutamate and GABA(B) transmissions in lateral amygdala are involved in startle-like electromyographic (EMG) potentiation caused by activation of auditory thalamus.
    He S, Huang J, Wu X, Li L.
    Neurosci Lett; 2005 Feb 10; 374(2):113-8. PubMed ID: 15644275
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  • 14. Stabilization of thalamo-cortical long-term potentiation by the amygdala: cholinergic and transcription-dependent mechanisms.
    Dringenberg HC, Kuo MC, Tomaszek S.
    Eur J Neurosci; 2004 Jul 10; 20(2):557-65. PubMed ID: 15233765
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  • 15. Amygdaloid GABA, not glutamate neurotransmission or mRNA transcription controls footshock-associated fear arousal in the acoustic startle paradigm.
    Van Nobelen M, Kokkinidis L.
    Neuroscience; 2006 Jul 10; 137(2):707-16. PubMed ID: 16289581
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  • 16. Cocaine withdrawal reduces group I mGluR-mediated long-term potentiation via decreased GABAergic transmission in the amygdala.
    Schmidt K, Krishnan B, Xia Y, Sun A, Orozco-Cabal L, Pollandt S, Centeno M, Genzer K, Gallagher JP, Shinnick-Gallagher P, Liu J.
    Eur J Neurosci; 2011 Jul 10; 34(2):177-89. PubMed ID: 21749491
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  • 17. Effects of the mGluR8 agonist (S)-3,4-DCPG in the lateral amygdala on acquisition/expression of fear-potentiated startle, synaptic transmission, and plasticity.
    Schmid S, Fendt M.
    Neuropharmacology; 2006 Feb 10; 50(2):154-64. PubMed ID: 16188284
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