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242 related items for PubMed ID: 16423699

  • 1. Contrasting effects of the persistent Na+ current on neuronal excitability and spike timing.
    Vervaeke K, Hu H, Graham LJ, Storm JF.
    Neuron; 2006 Jan 19; 49(2):257-70. PubMed ID: 16423699
    [Abstract] [Full Text] [Related]

  • 2. Persistent sodium currents in mesencephalic v neurons participate in burst generation and control of membrane excitability.
    Wu N, Enomoto A, Tanaka S, Hsiao CF, Nykamp DQ, Izhikevich E, Chandler SH.
    J Neurophysiol; 2005 May 19; 93(5):2710-22. PubMed ID: 15625100
    [Abstract] [Full Text] [Related]

  • 3. Dendritic Na+ current inactivation can increase cell excitability by delaying a somatic depolarizing afterpotential.
    Fernandez FR, Mehaffey WH, Turner RW.
    J Neurophysiol; 2005 Dec 19; 94(6):3836-48. PubMed ID: 16120659
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  • 4. Phasic stimuli evoke precisely timed spikes in intermittently discharging mitral cells.
    Balu R, Larimer P, Strowbridge BW.
    J Neurophysiol; 2004 Aug 19; 92(2):743-53. PubMed ID: 15277594
    [Abstract] [Full Text] [Related]

  • 5. Reduction of cortical pyramidal neuron excitability by the action of phenytoin on persistent Na+ current.
    Lampl I, Schwindt P, Crill W.
    J Pharmacol Exp Ther; 1998 Jan 19; 284(1):228-37. PubMed ID: 9435183
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  • 6. Role of axonal NaV1.6 sodium channels in action potential initiation of CA1 pyramidal neurons.
    Royeck M, Horstmann MT, Remy S, Reitze M, Yaari Y, Beck H.
    J Neurophysiol; 2008 Oct 19; 100(4):2361-80. PubMed ID: 18650312
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  • 9. Inhibitory synaptic plasticity regulates pyramidal neuron spiking in the rodent hippocampus.
    Saraga F, Balena T, Wolansky T, Dickson CT, Woodin MA.
    Neuroscience; 2008 Jul 31; 155(1):64-75. PubMed ID: 18562122
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  • 11. SK (KCa2) channels do not control somatic excitability in CA1 pyramidal neurons but can be activated by dendritic excitatory synapses and regulate their impact.
    Gu N, Hu H, Vervaeke K, Storm JF.
    J Neurophysiol; 2008 Nov 31; 100(5):2589-604. PubMed ID: 18684909
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  • 12. Ionic mechanisms in the generation of subthreshold oscillations and action potential clustering in entorhinal layer II stellate neurons.
    Fransén E, Alonso AA, Dickson CT, Magistretti J, Hasselmo ME.
    Hippocampus; 2004 Nov 31; 14(3):368-84. PubMed ID: 15132436
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  • 13. Contribution of persistent Na+ current and M-type K+ current to somatic bursting in CA1 pyramidal cells: combined experimental and modeling study.
    Golomb D, Yue C, Yaari Y.
    J Neurophysiol; 2006 Oct 31; 96(4):1912-26. PubMed ID: 16807352
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  • 15. Intrinsic bursting of immature CA3 pyramidal neurons and consequent giant depolarizing potentials are driven by a persistent Na+ current and terminated by a slow Ca2+ -activated K+ current.
    Sipilä ST, Huttu K, Voipio J, Kaila K.
    Eur J Neurosci; 2006 May 31; 23(9):2330-8. PubMed ID: 16706841
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  • 16. Voltage-activated sodium channels amplify inhibition in neocortical pyramidal neurons.
    Stuart G.
    Nat Neurosci; 1999 Feb 31; 2(2):144-50. PubMed ID: 10195198
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  • 17. Concentration-dependent effects of anticonvulsant enaminone methyl 4-(4'-bromophenyl)aminocyclohex-3-en-6-methyl-2-oxo-1-oate on neuronal excitability in vitro.
    Ananthalakshmi KV, Edafiogho IO, Kombian SB.
    Neuroscience; 2006 Aug 11; 141(1):345-56. PubMed ID: 16650601
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  • 19. Cell-type specific modulation of intrinsic firing properties and subthreshold membrane oscillations by the M(Kv7)-current in neurons of the entorhinal cortex.
    Yoshida M, Alonso A.
    J Neurophysiol; 2007 Nov 11; 98(5):2779-94. PubMed ID: 17728392
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  • 20. Boosting of neuronal firing evoked with asynchronous and synchronous inputs to the dendrite.
    Oviedo H, Reyes AD.
    Nat Neurosci; 2002 Mar 11; 5(3):261-6. PubMed ID: 11836531
    [Abstract] [Full Text] [Related]

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