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242 related items for PubMed ID: 16423699
1. Contrasting effects of the persistent Na+ current on neuronal excitability and spike timing. Vervaeke K, Hu H, Graham LJ, Storm JF. Neuron; 2006 Jan 19; 49(2):257-70. PubMed ID: 16423699 [Abstract] [Full Text] [Related]
2. Persistent sodium currents in mesencephalic v neurons participate in burst generation and control of membrane excitability. Wu N, Enomoto A, Tanaka S, Hsiao CF, Nykamp DQ, Izhikevich E, Chandler SH. J Neurophysiol; 2005 May 19; 93(5):2710-22. PubMed ID: 15625100 [Abstract] [Full Text] [Related]
3. Dendritic Na+ current inactivation can increase cell excitability by delaying a somatic depolarizing afterpotential. Fernandez FR, Mehaffey WH, Turner RW. J Neurophysiol; 2005 Dec 19; 94(6):3836-48. PubMed ID: 16120659 [Abstract] [Full Text] [Related]
5. Reduction of cortical pyramidal neuron excitability by the action of phenytoin on persistent Na+ current. Lampl I, Schwindt P, Crill W. J Pharmacol Exp Ther; 1998 Jan 19; 284(1):228-37. PubMed ID: 9435183 [Abstract] [Full Text] [Related]
6. Role of axonal NaV1.6 sodium channels in action potential initiation of CA1 pyramidal neurons. Royeck M, Horstmann MT, Remy S, Reitze M, Yaari Y, Beck H. J Neurophysiol; 2008 Oct 19; 100(4):2361-80. PubMed ID: 18650312 [Abstract] [Full Text] [Related]
11. SK (KCa2) channels do not control somatic excitability in CA1 pyramidal neurons but can be activated by dendritic excitatory synapses and regulate their impact. Gu N, Hu H, Vervaeke K, Storm JF. J Neurophysiol; 2008 Nov 31; 100(5):2589-604. PubMed ID: 18684909 [Abstract] [Full Text] [Related]
12. Ionic mechanisms in the generation of subthreshold oscillations and action potential clustering in entorhinal layer II stellate neurons. Fransén E, Alonso AA, Dickson CT, Magistretti J, Hasselmo ME. Hippocampus; 2004 Nov 31; 14(3):368-84. PubMed ID: 15132436 [Abstract] [Full Text] [Related]
13. Contribution of persistent Na+ current and M-type K+ current to somatic bursting in CA1 pyramidal cells: combined experimental and modeling study. Golomb D, Yue C, Yaari Y. J Neurophysiol; 2006 Oct 31; 96(4):1912-26. PubMed ID: 16807352 [Abstract] [Full Text] [Related]
15. Intrinsic bursting of immature CA3 pyramidal neurons and consequent giant depolarizing potentials are driven by a persistent Na+ current and terminated by a slow Ca2+ -activated K+ current. Sipilä ST, Huttu K, Voipio J, Kaila K. Eur J Neurosci; 2006 May 31; 23(9):2330-8. PubMed ID: 16706841 [Abstract] [Full Text] [Related]
16. Voltage-activated sodium channels amplify inhibition in neocortical pyramidal neurons. Stuart G. Nat Neurosci; 1999 Feb 31; 2(2):144-50. PubMed ID: 10195198 [Abstract] [Full Text] [Related]
17. Concentration-dependent effects of anticonvulsant enaminone methyl 4-(4'-bromophenyl)aminocyclohex-3-en-6-methyl-2-oxo-1-oate on neuronal excitability in vitro. Ananthalakshmi KV, Edafiogho IO, Kombian SB. Neuroscience; 2006 Aug 11; 141(1):345-56. PubMed ID: 16650601 [Abstract] [Full Text] [Related]
19. Cell-type specific modulation of intrinsic firing properties and subthreshold membrane oscillations by the M(Kv7)-current in neurons of the entorhinal cortex. Yoshida M, Alonso A. J Neurophysiol; 2007 Nov 11; 98(5):2779-94. PubMed ID: 17728392 [Abstract] [Full Text] [Related]
20. Boosting of neuronal firing evoked with asynchronous and synchronous inputs to the dendrite. Oviedo H, Reyes AD. Nat Neurosci; 2002 Mar 11; 5(3):261-6. PubMed ID: 11836531 [Abstract] [Full Text] [Related] Page: [Next] [New Search]