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762 related items for PubMed ID: 16442737
1. Early signs of motoneuron vulnerability in a disease model system: Characterization of transverse slice cultures of spinal cord isolated from embryonic ALS mice. Avossa D, Grandolfo M, Mazzarol F, Zatta M, Ballerini L. Neuroscience; 2006; 138(4):1179-94. PubMed ID: 16442737 [Abstract] [Full Text] [Related]
2. Neural stem cells LewisX+ CXCR4+ modify disease progression in an amyotrophic lateral sclerosis model. Corti S, Locatelli F, Papadimitriou D, Del Bo R, Nizzardo M, Nardini M, Donadoni C, Salani S, Fortunato F, Strazzer S, Bresolin N, Comi GP. Brain; 2007 May; 130(Pt 5):1289-305. PubMed ID: 17439986 [Abstract] [Full Text] [Related]
3. Excitotoxic motoneuron degeneration induced by glutamate receptor agonists and mitochondrial toxins in organotypic cultures of chick embryo spinal cord. Brunet N, Tarabal O, Esquerda JE, Calderó J. J Comp Neurol; 2009 Oct 01; 516(4):277-90. PubMed ID: 19634179 [Abstract] [Full Text] [Related]
4. Chronic elevation of extracellular glutamate due to transport blockade is innocuous for spinal motoneurons in vivo. Tovar-Y-Romo LB, Santa-Cruz LD, Zepeda A, Tapia R. Neurochem Int; 2009 Oct 01; 54(3-4):186-91. PubMed ID: 19100799 [Abstract] [Full Text] [Related]
5. Ca2+-permeable AMPA receptors and intracellular Ca2+ determine motoneuron vulnerability in rat spinal cord in vivo. Corona JC, Tapia R. Neuropharmacology; 2007 Apr 01; 52(5):1219-28. PubMed ID: 17320918 [Abstract] [Full Text] [Related]
6. Increased internalisation and degradation of GLT-1 glial glutamate transporter in a cell model for familial amyotrophic lateral sclerosis (ALS). Vanoni C, Massari S, Losa M, Carrega P, Perego C, Conforti L, Pietrini G. J Cell Sci; 2004 Oct 15; 117(Pt 22):5417-26. PubMed ID: 15466883 [Abstract] [Full Text] [Related]
7. ALS-linked Cu/Zn-SOD mutation increases vulnerability of motor neurons to excitotoxicity by a mechanism involving increased oxidative stress and perturbed calcium homeostasis. Kruman II, Pedersen WA, Springer JE, Mattson MP. Exp Neurol; 1999 Nov 15; 160(1):28-39. PubMed ID: 10630188 [Abstract] [Full Text] [Related]
8. A systematic study of brainstem motor nuclei in a mouse model of ALS, the effects of lithium. Ferrucci M, Spalloni A, Bartalucci A, Cantafora E, Fulceri F, Nutini M, Longone P, Paparelli A, Fornai F. Neurobiol Dis; 2010 Feb 15; 37(2):370-83. PubMed ID: 19874893 [Abstract] [Full Text] [Related]
9. Compensatory changes in degenerating spinal motoneurons sustain functional sparing in the SOD1-G93A mouse model of amyotrophic lateral sclerosis. Giusto E, Codrich M, de Leo G, Francardo V, Coradazzi M, Parenti R, Gulisano M, Vicario N, Gulino R, Leanza G. J Comp Neurol; 2020 Feb 01; 528(2):231-243. PubMed ID: 31364764 [Abstract] [Full Text] [Related]
10. Altered distributions of nucleocytoplasmic transport-related proteins in the spinal cord of a mouse model of amyotrophic lateral sclerosis. Zhang J, Ito H, Wate R, Ohnishi S, Nakano S, Kusaka H. Acta Neuropathol; 2006 Dec 01; 112(6):673-80. PubMed ID: 16957927 [Abstract] [Full Text] [Related]
11. Expression of heat shock transcription factor 1 and its downstream target protein T-cell death associated gene 51 in the spinal cord of a mouse model of amyotrophic lateral sclerosis. Mimoto T, Morimoto N, Miyazaki K, Kurata T, Sato K, Ikeda Y, Abe K. Brain Res; 2012 Dec 07; 1488():123-31. PubMed ID: 23063459 [Abstract] [Full Text] [Related]
12. Altered development in GABA co-release shapes glycinergic synaptic currents in cultured spinal slices of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis. Medelin M, Rancic V, Cellot G, Laishram J, Veeraraghavan P, Rossi C, Muzio L, Sivilotti L, Ballerini L. J Physiol; 2016 Jul 01; 594(13):3827-40. PubMed ID: 27098371 [Abstract] [Full Text] [Related]
13. Protein retention in the endoplasmic reticulum, blockade of programmed cell death and autophagy selectively occur in spinal cord motoneurons after glutamate receptor-mediated injury. Tarabal O, Calderó J, Casas C, Oppenheim RW, Esquerda JE. Mol Cell Neurosci; 2005 Jun 01; 29(2):283-98. PubMed ID: 15911352 [Abstract] [Full Text] [Related]
14. Serotonergic mechanisms in amyotrophic lateral sclerosis. Sandyk R. Int J Neurosci; 2006 Jul 01; 116(7):775-826. PubMed ID: 16861147 [Abstract] [Full Text] [Related]
15. Slow and selective death of spinal motor neurons in vivo by intrathecal infusion of kainic acid: implications for AMPA receptor-mediated excitotoxicity in ALS. Sun H, Kawahara Y, Ito K, Kanazawa I, Kwak S. J Neurochem; 2006 Aug 01; 98(3):782-91. PubMed ID: 16893420 [Abstract] [Full Text] [Related]
16. GAB(A) receptors present higher affinity and modified subunit composition in spinal motor neurons from a genetic model of amyotrophic lateral sclerosis. Carunchio I, Mollinari C, Pieri M, Merlo D, Zona C. Eur J Neurosci; 2008 Oct 01; 28(7):1275-85. PubMed ID: 18973555 [Abstract] [Full Text] [Related]
17. Oxidative and excitotoxic insults exert differential effects on spinal motoneurons and astrocytic glutamate transporters: Implications for the role of astrogliosis in amyotrophic lateral sclerosis. Zagami CJ, Beart PM, Wallis N, Nagley P, O'Shea RD. Glia; 2009 Jan 15; 57(2):119-35. PubMed ID: 18661557 [Abstract] [Full Text] [Related]
18. Selective loss of alpha motoneurons innervating the medial gastrocnemius muscle in a mouse model of amyotrophic lateral sclerosis. Mohajeri MH, Figlewicz DA, Bohn MC. Exp Neurol; 1998 Apr 15; 150(2):329-36. PubMed ID: 9527903 [Abstract] [Full Text] [Related]
19. Survival and death of mature avian motoneurons in organotypic slice culture: trophic requirements for survival and different types of degeneration. Brunet N, Tarabal O, Portero-Otín M, Oppenheim RW, Esquerda JE, Calderó J. J Comp Neurol; 2007 Apr 10; 501(5):669-90. PubMed ID: 17299760 [Abstract] [Full Text] [Related]
20. Characterization of chronic glutamate-mediated motor neuron toxicity in organotypic spinal cord culture prepared from ALS model mice. Kosuge Y, Sekikawa-Nishida K, Negi H, Ishige K, Ito Y. Neurosci Lett; 2009 Apr 24; 454(2):165-9. PubMed ID: 19429077 [Abstract] [Full Text] [Related] Page: [Next] [New Search]