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293 related items for PubMed ID: 16467499

  • 1. Physiological role of the alpha1- and alpha2-isoforms of the Na+-K+-ATPase and biological significance of their cardiac glycoside binding site.
    Dostanic-Larson I, Lorenz JN, Van Huysse JW, Neumann JC, Moseley AE, Lingrel JB.
    Am J Physiol Regul Integr Comp Physiol; 2006 Mar; 290(3):R524-8. PubMed ID: 16467499
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  • 2. The highly conserved cardiac glycoside binding site of Na,K-ATPase plays a role in blood pressure regulation.
    Dostanic-Larson I, Van Huysse JW, Lorenz JN, Lingrel JB.
    Proc Natl Acad Sci U S A; 2005 Nov 01; 102(44):15845-50. PubMed ID: 16243970
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  • 3. Cardiac glycosides induced toxicity in human cells expressing α1-, α2-, or α3-isoforms of Na-K-ATPase.
    Cherniavsky Lev M, Karlish SJ, Garty H.
    Am J Physiol Cell Physiol; 2015 Jul 15; 309(2):C126-35. PubMed ID: 25994790
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  • 7. The ouabain-binding site of the α2 isoform of Na,K-ATPase plays a role in blood pressure regulation during pregnancy.
    Oshiro N, Dostanic-Larson I, Neumann JC, Lingrel JB.
    Am J Hypertens; 2010 Dec 15; 23(12):1279-85. PubMed ID: 20940714
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  • 9. The alpha2-isoform of Na-K-ATPase mediates ouabain-induced hypertension in mice and increased vascular contractility in vitro.
    Dostanic I, Paul RJ, Lorenz JN, Theriault S, Van Huysse JW, Lingrel JB.
    Am J Physiol Heart Circ Physiol; 2005 Feb 15; 288(2):H477-85. PubMed ID: 15458945
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  • 10. ACTH-induced hypertension is dependent on the ouabain-binding site of the alpha2-Na+-K+-ATPase subunit.
    Lorenz JN, Loreaux EL, Dostanic-Larson I, Lasko V, Schnetzer JR, Paul RJ, Lingrel JB.
    Am J Physiol Heart Circ Physiol; 2008 Jul 15; 295(1):H273-80. PubMed ID: 18487447
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  • 11. DOCA-salt hypertension does not require the ouabain-sensitive binding site of the α2 Na,K-ATPase.
    Lorenz JN, Oshiro N, Loreaux EL, Lingrel JB.
    Am J Hypertens; 2012 Apr 15; 25(4):421-9. PubMed ID: 22258333
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  • 12. New molecular determinants controlling the accessibility of ouabain to its binding site in human Na,K-ATPase alpha isoforms.
    Crambert G, Schaer D, Roy S, Geering K.
    Mol Pharmacol; 2004 Feb 15; 65(2):335-41. PubMed ID: 14742675
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  • 13. The cardiac sodium pump: structure and function.
    McDonough AA, Velotta JB, Schwinger RH, Philipson KD, Farley RA.
    Basic Res Cardiol; 2002 Feb 15; 97 Suppl 1():I19-24. PubMed ID: 12479229
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  • 15. Renovascular hypertension using a modified two-kidney, one-clip approach in mice is not dependent on the α1 or α2 Na-K-ATPase ouabain-binding site.
    Lorenz JN, Lasko VM, Nieman ML, Damhoff T, Prasad V, Beierwaltes WH, Lingrel JB.
    Am J Physiol Renal Physiol; 2011 Sep 15; 301(3):F615-21. PubMed ID: 21632957
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  • 16. Cardiac Oxidative Signaling and Physiological Hypertrophy in the Na/K-ATPase α1s/sα2s/s Mouse Model of High Affinity for Cardiotonic Steroids.
    Marck PV, Pessoa MT, Xu Y, Kutz LC, Collins DM, Yan Y, King C, Wang X, Duan Q, Cai L, Xie JX, Lingrel JB, Xie Z, Tian J, Pierre SV.
    Int J Mol Sci; 2021 Mar 27; 22(7):. PubMed ID: 33801629
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  • 17. The alpha 1 isoform of Na,K-ATPase regulates cardiac contractility and functionally interacts and co-localizes with the Na/Ca exchanger in heart.
    Dostanic I, Schultz Jel J, Lorenz JN, Lingrel JB.
    J Biol Chem; 2004 Dec 24; 279(52):54053-61. PubMed ID: 15485817
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  • 18. Ouabain-Sensitive alpha1 Na,K-ATPase enhances natriuretic response to saline load.
    Loreaux EL, Kaul B, Lorenz JN, Lingrel JB.
    J Am Soc Nephrol; 2008 Oct 24; 19(10):1947-54. PubMed ID: 18667729
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  • 19. Marinobufagenin enhances cardiac contractility in mice with ouabain-sensitive alpha1 Na+-K+-ATPase.
    Wansapura AN, Lasko V, Xie Z, Fedorova OV, Bagrov AY, Lingrel JB, Lorenz JN.
    Am J Physiol Heart Circ Physiol; 2009 Jun 24; 296(6):H1833-9. PubMed ID: 19376809
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  • 20. Kinetic analysis of ouabain binding to native and mutated forms of Na,K-ATPase and identification of a new region involved in cardiac glycoside interactions.
    Schultheis PJ, Wallick ET, Lingrel JB.
    J Biol Chem; 1993 Oct 25; 268(30):22686-94. PubMed ID: 8226778
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