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Journal Abstract Search


2227 related items for PubMed ID: 16478525

  • 1. Mutations in amyloid precursor protein and presenilin-1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid beta-peptide (1-42), HO and kainic acid: implications for Alzheimer's disease.
    Mohmmad Abdul H, Sultana R, Keller JN, St Clair DK, Markesbery WR, Butterfield DA.
    J Neurochem; 2006 Mar; 96(5):1322-35. PubMed ID: 16478525
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  • 2. Acetyl-L-carnitine-induced up-regulation of heat shock proteins protects cortical neurons against amyloid-beta peptide 1-42-mediated oxidative stress and neurotoxicity: implications for Alzheimer's disease.
    Abdul HM, Calabrese V, Calvani M, Butterfield DA.
    J Neurosci Res; 2006 Aug 01; 84(2):398-408. PubMed ID: 16634066
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  • 3. Presenilin 1 regulates the processing of beta-amyloid precursor protein C-terminal fragments and the generation of amyloid beta-protein in endoplasmic reticulum and Golgi.
    Xia W, Zhang J, Ostaszewski BL, Kimberly WT, Seubert P, Koo EH, Shen J, Selkoe DJ.
    Biochemistry; 1998 Nov 24; 37(47):16465-71. PubMed ID: 9843412
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  • 5. Ferulic acid ethyl ester protects neurons against amyloid beta- peptide(1-42)-induced oxidative stress and neurotoxicity: relationship to antioxidant activity.
    Sultana R, Ravagna A, Mohmmad-Abdul H, Calabrese V, Butterfield DA.
    J Neurochem; 2005 Feb 24; 92(4):749-58. PubMed ID: 15686476
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  • 6. Neuropathological characterization of mutant amyloid precursor protein yeast artificial chromosome transgenic mice.
    Kulnane LS, Lamb BT.
    Neurobiol Dis; 2001 Dec 24; 8(6):982-92. PubMed ID: 11741394
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  • 7. Reduced retinal function in amyloid precursor protein-over-expressing transgenic mice via attenuating glutamate-N-methyl-d-aspartate receptor signaling.
    Shimazawa M, Inokuchi Y, Okuno T, Nakajima Y, Sakaguchi G, Kato A, Oku H, Sugiyama T, Kudo T, Ikeda T, Takeda M, Hara H.
    J Neurochem; 2008 Oct 24; 107(1):279-90. PubMed ID: 18691390
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  • 8. Proteolytic processing of the Alzheimer's disease amyloid precursor protein in brain and platelets.
    Evin G, Zhu A, Holsinger RM, Masters CL, Li QX.
    J Neurosci Res; 2003 Nov 01; 74(3):386-92. PubMed ID: 14598315
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  • 9. Sublethal concentrations of prion peptide PrP106-126 or the amyloid beta peptide of Alzheimer's disease activates expression of proapoptotic markers in primary cortical neurons.
    White AR, Guirguis R, Brazier MW, Jobling MF, Hill AF, Beyreuther K, Barrow CJ, Masters CL, Collins SJ, Cappai R.
    Neurobiol Dis; 2001 Apr 01; 8(2):299-316. PubMed ID: 11300725
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  • 10. Central cholinergic functions in human amyloid precursor protein knock-in/presenilin-1 transgenic mice.
    Hartmann J, Erb C, Ebert U, Baumann KH, Popp A, König G, Klein J.
    Neuroscience; 2004 Apr 01; 125(4):1009-17. PubMed ID: 15120860
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  • 12. Familial Alzheimer's disease mutations inhibit gamma-secretase-mediated liberation of beta-amyloid precursor protein carboxy-terminal fragment.
    Wiley JC, Hudson M, Kanning KC, Schecterson LC, Bothwell M.
    J Neurochem; 2005 Sep 01; 94(5):1189-201. PubMed ID: 15992373
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  • 14. Secreted amyloid precursor protein and holo-APP bind amyloid beta through distinct domains eliciting different toxic responses on hippocampal neurons.
    Kedikian G, Heredia F, Salvador VR, Raimunda D, Isoardi N, Heredia L, Lorenzo A.
    J Neurosci Res; 2010 Jun 01; 88(8):1795-803. PubMed ID: 20155808
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  • 16. Decreased Abeta secretion by cells expressing familial Alzheimer's disease-linked mutant presenilin 1.
    Shimojo M, Sahara N, Murayama M, Ichinose H, Takashima A.
    Neurosci Res; 2007 Mar 01; 57(3):446-53. PubMed ID: 17210196
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  • 17. Caspase inhibition therapy abolishes brain trauma-induced increases in Abeta peptide: implications for clinical outcome.
    Abrahamson EE, Ikonomovic MD, Ciallella JR, Hope CE, Paljug WR, Isanski BA, Flood DG, Clark RS, DeKosky ST.
    Exp Neurol; 2006 Feb 01; 197(2):437-50. PubMed ID: 16300758
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  • 18. Comparative analysis of cortical gene expression in mouse models of Alzheimer's disease.
    Wu ZL, Ciallella JR, Flood DG, O'Kane TM, Bozyczko-Coyne D, Savage MJ.
    Neurobiol Aging; 2006 Mar 01; 27(3):377-86. PubMed ID: 15927307
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  • 19. Overexpression of amyloid precursor protein is associated with degeneration, decreased viability, and increased damage caused by neurotoxins (prostaglandins A1 and E2, hydrogen peroxide, and nitric oxide) in differentiated neuroblastoma cells.
    Hanson AJ, Prasad JE, Nahreini P, Andreatta C, Kumar B, Yan XD, Prasad KN.
    J Neurosci Res; 2003 Oct 01; 74(1):148-59. PubMed ID: 13130517
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  • 20. AATF protects neural cells against oxidative damage induced by amyloid beta-peptide.
    Xie J, Guo Q.
    Neurobiol Dis; 2004 Jun 01; 16(1):150-7. PubMed ID: 15207272
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