These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

161 related items for PubMed ID: 1647923

  • 1. Na+,K(+)-ATPase activity and hormones in single nephron segments from nephrotic rats.
    Vogt B, Favre H.
    Clin Sci (Lond); 1991 Jun; 80(6):599-604. PubMed ID: 1647923
    [Abstract] [Full Text] [Related]

  • 2. Glomerulonephritis and sodium retention: enhancement of Na+/K+-ATPase activity in the collecting duct is shared by rats with puromycin induced nephrotic syndrome and mice with spontaneous lupus-like glomerulonephritis.
    Zolty E, Ibnou-Zekri N, Izui S, Féraille E, Favre H.
    Nephrol Dial Transplant; 1999 Sep; 14(9):2192-5. PubMed ID: 10489230
    [Abstract] [Full Text] [Related]

  • 3. [Role of tubular Na-K-ATPase in nephrotic syndrome induced by puromycin in the rat].
    Féraille E.
    Nephrologie; 1994 Sep; 15(4):301-5. PubMed ID: 7984251
    [Abstract] [Full Text] [Related]

  • 4. Specific activity of Na-K-ATPase after adrenalectomy and hormone replacement along the rabbit nephron.
    El Mernissi G, Doucet A.
    Pflugers Arch; 1984 Nov; 402(3):258-63. PubMed ID: 6097867
    [Abstract] [Full Text] [Related]

  • 5. Modulation of renal sodium-potassium-adenosine triphosphatase by aldosterone. Effect of high physiologic levels on enzyme activity in isolated rat and rabbit tubules.
    Mujais SK, Chekal MA, Jones WJ, Hayslett JP, Katz AI.
    J Clin Invest; 1985 Jul; 76(1):170-6. PubMed ID: 2991336
    [Abstract] [Full Text] [Related]

  • 6. Mechanism of enhanced Na-K-ATPase activity in cortical collecting duct from rats with nephrotic syndrome.
    Féraille E, Vogt B, Rousselot M, Barlet-Bas C, Cheval L, Doucet A, Favre H.
    J Clin Invest; 1993 Apr; 91(4):1295-300. PubMed ID: 8386183
    [Abstract] [Full Text] [Related]

  • 7. Collecting duct (Na+/K+)-ATPase activity is correlated with urinary sodium excretion in rat nephrotic syndromes.
    Deschênes G, Doucet A.
    J Am Soc Nephrol; 2000 Apr; 11(4):604-615. PubMed ID: 10752519
    [Abstract] [Full Text] [Related]

  • 8. Short-term effect of aldosterone on renal sodium transport and tubular Na-K-ATPase in the rat.
    El Mernissi G, Doucet A.
    Pflugers Arch; 1983 Oct; 399(2):139-46. PubMed ID: 6139787
    [Abstract] [Full Text] [Related]

  • 9. Stimulation of Na-K-ATPase in the rat collecting tubule by two diuretics: furosemide and amiloride.
    Mernissi GE, Doucet A.
    Am J Physiol; 1984 Sep; 247(3 Pt 2):F485-90. PubMed ID: 6089591
    [Abstract] [Full Text] [Related]

  • 10. Collecting duct is a site of sodium retention in PAN nephrosis: a rationale for amiloride therapy.
    Deschênes G, Wittner M, Stefano AD, Jounier S, Doucet A.
    J Am Soc Nephrol; 2001 Mar; 12(3):598-601. PubMed ID: 11181809
    [Abstract] [Full Text] [Related]

  • 11. Increased expression but not targeting of ENaC in adrenalectomized rats with PAN-induced nephrotic syndrome.
    de Seigneux S, Kim SW, Hemmingsen SC, Frøkiaer J, Nielsen S.
    Am J Physiol Renal Physiol; 2006 Jul; 291(1):F208-17. PubMed ID: 16403831
    [Abstract] [Full Text] [Related]

  • 12. Indomethacin and sodium retention in the rat: role of inhibition of prostaglandin E2 synthesis.
    Scherzer P, Wald H, Rubinger D, Popovtzer MM.
    Clin Sci (Lond); 1992 Sep; 83(3):307-11. PubMed ID: 1327647
    [Abstract] [Full Text] [Related]

  • 13. Na,K-ATPase in isolated nephron segments in rats with experimental heart failure.
    Wald H, Scherzer P, Popovtzer MM.
    Circ Res; 1991 Apr; 68(4):1051-8. PubMed ID: 1849057
    [Abstract] [Full Text] [Related]

  • 14. Nephrotic syndrome is associated with increased plasma K+ concentration, intestinal K+ losses, and attenuated urinary K+ excretion: a study in rats and humans.
    Ydegaard R, Svenningsen P, Bistrup C, Andersen RF, Stubbe J, Buhl KB, Marcussen N, Hinrichs GR, Iraqi H, Zamani R, Dimke H, Jensen BL.
    Am J Physiol Renal Physiol; 2019 Dec 01; 317(6):F1549-F1562. PubMed ID: 31566427
    [Abstract] [Full Text] [Related]

  • 15. Effect of low potassium-diet on Na-K-ATPase in rat nephron segments.
    Garg LC, Mackie S, Tisher CC.
    Pflugers Arch; 1982 Aug 01; 394(2):113-7. PubMed ID: 6289258
    [Abstract] [Full Text] [Related]

  • 16. Secondary effect of aldosterone on Na-KATPase activity in the rabbit cortical collecting tubule.
    Petty KJ, Kokko JP, Marver D.
    J Clin Invest; 1981 Dec 01; 68(6):1514-21. PubMed ID: 6274911
    [Abstract] [Full Text] [Related]

  • 17. Short-term effects of aldosterone and dexamethasone on Na-K-ATPase along the rabbit nephron.
    El Mernissi G, Doucet A.
    Pflugers Arch; 1983 Oct 01; 399(2):147-51. PubMed ID: 6139788
    [Abstract] [Full Text] [Related]

  • 18. Distal nephron sodium-potassium exchange in children with nephrotic syndrome.
    Donckerwolcke RA, France A, Raes A, Vande Walle J.
    Clin Nephrol; 2003 Apr 01; 59(4):259-66. PubMed ID: 12708565
    [Abstract] [Full Text] [Related]

  • 19. Regulation of renal Na-K-ATPase in the rat. Role of the natural mineralo- and glucocorticoid hormones.
    Mujais SK, Chekal MA, Jones WJ, Hayslett JP, Katz AI.
    J Clin Invest; 1984 Jan 01; 73(1):13-9. PubMed ID: 6317714
    [Abstract] [Full Text] [Related]

  • 20. Jejunal dopamine and Na,K+-ATPase activity in nephrotic syndrome.
    Sampaio-Maia B, Moreira-Rodrigues M, Serrão P, Pestana M.
    Am J Nephrol; 2005 Jan 01; 25(4):382-92. PubMed ID: 16088079
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 9.