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1053 related items for PubMed ID: 16504408

  • 1. Inhibition of calpain and caspase-3 prevented apoptosis and preserved electrophysiological properties of voltage-gated and ligand-gated ion channels in rat primary cortical neurons exposed to glutamate.
    Ray SK, Karmakar S, Nowak MW, Banik NL.
    Neuroscience; 2006 May 12; 139(2):577-95. PubMed ID: 16504408
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  • 2. 17beta-estradiol attenuates glutamate-induced apoptosis and preserves electrophysiologic function in primary cortical neurons.
    Sribnick EA, Ray SK, Nowak MW, Li L, Banik NL.
    J Neurosci Res; 2004 Jun 01; 76(5):688-96. PubMed ID: 15139027
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  • 3. Opposing roles for caspase and calpain death proteases in L-glutamate-induced oxidative neurotoxicity.
    Elphick LM, Hawat M, Toms NJ, Meinander A, Mikhailov A, Eriksson JE, Kass GE.
    Toxicol Appl Pharmacol; 2008 Oct 15; 232(2):258-67. PubMed ID: 18687350
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  • 4. Evidence that non-caspase proteases are required for chromatin degradation during apoptosis.
    Hughes FM, Evans-Storms RB, Cidlowski JA.
    Cell Death Differ; 1998 Dec 15; 5(12):1017-27. PubMed ID: 9894608
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  • 5. Inhibition of N-methyl-D-aspartate receptors increases paraoxon-induced apoptosis in cultured neurons.
    Wu X, Tian F, Okagaki P, Marini AM.
    Toxicol Appl Pharmacol; 2005 Oct 01; 208(1):57-67. PubMed ID: 16164961
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  • 6. Novel characteristics of glutamate-induced cell death in primary septohippocampal cultures: relationship to calpain and caspase-3 protease activation.
    Zhao X, Newcomb JK, Pike BR, Wang KK, d'Avella D, Hayes RL.
    J Cereb Blood Flow Metab; 2000 Mar 01; 20(3):550-62. PubMed ID: 10724120
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  • 7. Role of the NR2A/2B subunits of the N-methyl-D-aspartate receptor in glutamate-induced glutamic acid decarboxylase alteration in cortical GABAergic neurons in vitro.
    Monnerie H, Hsu FC, Coulter DA, Le Roux PD.
    Neuroscience; 2010 Dec 29; 171(4):1075-90. PubMed ID: 20923697
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  • 11. Apoptosis is not an invariable component of in vitro models of cortical cerebral ischaemia.
    Jones PA, May GR, McLuckie JA, Iwashita A, Sharkey J.
    Cell Res; 2004 Jun 29; 14(3):241-50. PubMed ID: 15225418
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  • 14. Serofendic acid, a neuroprotective substance derived from fetal calf serum, inhibits mitochondrial membrane depolarization and caspase-3 activation.
    Kume T, Taguchi R, Katsuki H, Akao M, Sugimoto H, Kaneko S, Akaike A.
    Eur J Pharmacol; 2006 Aug 07; 542(1-3):69-76. PubMed ID: 16806165
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  • 15. Evidence for synaptic apoptosis.
    Mattson MP, Keller JN, Begley JG.
    Exp Neurol; 1998 Sep 07; 153(1):35-48. PubMed ID: 9743565
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  • 16. Attenuation of delayed neuronal death after mild focal ischemia in mice by inhibition of the caspase family.
    Endres M, Namura S, Shimizu-Sasamata M, Waeber C, Zhang L, Gómez-Isla T, Hyman BT, Moskowitz MA.
    J Cereb Blood Flow Metab; 1998 Mar 07; 18(3):238-47. PubMed ID: 9498840
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  • 17. Inhibition of nuclear factor kappaB is associated with neuroprotective effects of glycyrrhizic acid on glutamate-induced excitotoxicity in primary neurons.
    Cherng JM, Lin HJ, Hung MS, Lin YR, Chan MH, Lin JC.
    Eur J Pharmacol; 2006 Oct 10; 547(1-3):10-21. PubMed ID: 16952351
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  • 18. Caspase-mediated suppression of glutamate (AMPA) receptor channel activity in hippocampal neurons in response to DNA damage promotes apoptosis and prevents necrosis: implications for neurological side effects of cancer therapy and neurodegenerative disorders.
    Lu C, Fu W, Mattson MP.
    Neurobiol Dis; 2001 Apr 10; 8(2):194-206. PubMed ID: 11300717
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