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Journal Abstract Search

349 related items for PubMed ID: 16513752

  • 1. Evidence that the promoter can influence assembly of antitermination complexes at downstream RNA sites.
    Zhou Y, Shi T, Mozola MA, Olson ER, Henthorn K, Brown S, Gussin GN, Friedman DI.
    J Bacteriol; 2006 Mar; 188(6):2222-32. PubMed ID: 16513752
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  • 3. Bacteriophage lambda N-dependent transcription antitermination. Competition for an RNA site may regulate antitermination.
    Patterson TA, Zhang Z, Baker T, Johnson LL, Friedman DI, Court DL.
    J Mol Biol; 1994 Feb 11; 236(1):217-28. PubMed ID: 8107107
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  • 6. Genetic analysis of bacteriophage lambdaN-dependent antitermination suggests a possible role for the RNA polymerase alpha subunit in facilitating specific functions of NusA and NusE.
    Szalewska-Pałasz A, Strzelczyk B, Herman-Antosiewicz A, Wegrzyn G, Thomas MS.
    Arch Microbiol; 2003 Sep 11; 180(3):161-8. PubMed ID: 12845423
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  • 7. Translation repression by an RNA polymerase elongation complex.
    Wilson HR, Zhou JG, Yu D, Court DL.
    Mol Microbiol; 2004 Aug 11; 53(3):821-8. PubMed ID: 15255895
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  • 8. Escherichia coli RNA polymerase mutations located near the upstream edge of an RNA:DNA hybrid and the beginning of the RNA-exit channel are defective for transcription antitermination by the N protein from lambdoid phage H-19B.
    Cheeran A, Babu Suganthan R, Swapna G, Bandey I, Achary MS, Nagarajaram HA, Sen R.
    J Mol Biol; 2005 Sep 09; 352(1):28-43. PubMed ID: 16061258
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  • 16. Clustered arginine residues of bacteriophage lambda N protein are essential to antitermination of transcription, but their locale cannot compensate for boxB loop defects.
    Franklin NC.
    J Mol Biol; 1993 May 20; 231(2):343-60. PubMed ID: 8510151
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