These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

1659 related items for PubMed ID: 16551656

  • 1. Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression.
    Manczak M, Anekonda TS, Henson E, Park BS, Quinn J, Reddy PH.
    Hum Mol Genet; 2006 May 01; 15(9):1437-49. PubMed ID: 16551656
    [Abstract] [Full Text] [Related]

  • 2. Protective effects of reduced dynamin-related protein 1 against amyloid beta-induced mitochondrial dysfunction and synaptic damage in Alzheimer's disease.
    Manczak M, Kandimalla R, Fry D, Sesaki H, Reddy PH.
    Hum Mol Genet; 2016 Dec 01; 25(23):5148-5166. PubMed ID: 27677309
    [Abstract] [Full Text] [Related]

  • 3. Amyloid precursor protein-mediated free radicals and oxidative damage: implications for the development and progression of Alzheimer's disease.
    Reddy PH.
    J Neurochem; 2006 Jan 01; 96(1):1-13. PubMed ID: 16305625
    [Abstract] [Full Text] [Related]

  • 4.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 5.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 6. Mutations in amyloid precursor protein and presenilin-1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid beta-peptide (1-42), HO and kainic acid: implications for Alzheimer's disease.
    Mohmmad Abdul H, Sultana R, Keller JN, St Clair DK, Markesbery WR, Butterfield DA.
    J Neurochem; 2006 Mar 01; 96(5):1322-35. PubMed ID: 16478525
    [Abstract] [Full Text] [Related]

  • 7.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 8. Mitochondrial Abeta: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease.
    Caspersen C, Wang N, Yao J, Sosunov A, Chen X, Lustbader JW, Xu HW, Stern D, McKhann G, Yan SD.
    FASEB J; 2005 Dec 01; 19(14):2040-1. PubMed ID: 16210396
    [Abstract] [Full Text] [Related]

  • 9. The H63D HFE gene variant promotes activation of the intrinsic apoptotic pathway via mitochondria dysfunction following β-amyloid peptide exposure.
    Mairuae N, Hall Ii EC, Cheepsunthorn P, Lee SY, Connor JR.
    J Neurosci Res; 2010 Nov 01; 88(14):3079-89. PubMed ID: 20734416
    [Abstract] [Full Text] [Related]

  • 10. Pyroglutamate-modified amyloid-β protein demonstrates similar properties in an Alzheimer's disease familial mutant knock-in mouse and Alzheimer's disease brain.
    Wu G, Miller RA, Connolly B, Marcus J, Renger J, Savage MJ.
    Neurodegener Dis; 2014 Nov 01; 14(2):53-66. PubMed ID: 24158021
    [Abstract] [Full Text] [Related]

  • 11. Mitochondria-targeted small molecule SS31: a potential candidate for the treatment of Alzheimer's disease.
    Reddy PH, Manczak M, Kandimalla R.
    Hum Mol Genet; 2017 Apr 15; 26(8):1483-1496. PubMed ID: 28186562
    [Abstract] [Full Text] [Related]

  • 12.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 13. Hippocampal mutant APP and amyloid beta-induced cognitive decline, dendritic spine loss, defective autophagy, mitophagy and mitochondrial abnormalities in a mouse model of Alzheimer's disease.
    Manczak M, Kandimalla R, Yin X, Reddy PH.
    Hum Mol Genet; 2018 Apr 15; 27(8):1332-1342. PubMed ID: 29408999
    [Abstract] [Full Text] [Related]

  • 14. Alzheimer's disease.
    De-Paula VJ, Radanovic M, Diniz BS, Forlenza OV.
    Subcell Biochem; 2012 Apr 15; 65():329-52. PubMed ID: 23225010
    [Abstract] [Full Text] [Related]

  • 15. Effects of the superoxide dismutase/catalase mimetic EUK-207 in a mouse model of Alzheimer's disease: protection against and interruption of progression of amyloid and tau pathology and cognitive decline.
    Clausen A, Xu X, Bi X, Baudry M.
    J Alzheimers Dis; 2012 Apr 15; 30(1):183-208. PubMed ID: 22406441
    [Abstract] [Full Text] [Related]

  • 16. Mitochondrial targeting and a novel transmembrane arrest of Alzheimer's amyloid precursor protein impairs mitochondrial function in neuronal cells.
    Anandatheerthavarada HK, Biswas G, Robin MA, Avadhani NG.
    J Cell Biol; 2003 Apr 14; 161(1):41-54. PubMed ID: 12695498
    [Abstract] [Full Text] [Related]

  • 17. Mitochondria-targeted catalase reduces abnormal APP processing, amyloid β production and BACE1 in a mouse model of Alzheimer's disease: implications for neuroprotection and lifespan extension.
    Mao P, Manczak M, Calkins MJ, Truong Q, Reddy TP, Reddy AP, Shirendeb U, Lo HH, Rabinovitch PS, Reddy PH.
    Hum Mol Genet; 2012 Jul 01; 21(13):2973-90. PubMed ID: 22492996
    [Abstract] [Full Text] [Related]

  • 18. Mitochondrial dysfunction and accumulation of the β-secretase-cleaved C-terminal fragment of APP in Alzheimer's disease transgenic mice.
    Devi L, Ohno M.
    Neurobiol Dis; 2012 Jan 01; 45(1):417-24. PubMed ID: 21933711
    [Abstract] [Full Text] [Related]

  • 19. Early Presymptomatic Changes in the Proteome of Mitochondria-Associated Membrane in the APP/PS1 Mouse Model of Alzheimer's Disease.
    Völgyi K, Badics K, Sialana FJ, Gulyássy P, Udvari EB, Kis V, Drahos L, Lubec G, Kékesi KA, Juhász G.
    Mol Neurobiol; 2018 Oct 01; 55(10):7839-7857. PubMed ID: 29468564
    [Abstract] [Full Text] [Related]

  • 20.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 83.