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23. A herpes simplex virus type 1 variant, deleted in the promoter region of the latency-associated transcripts, does not produce any detectable minor RNA species during latency in the mouse trigeminal ganglion. Mitchell WJ, Steiner I, Brown SM, MacLean AR, Subak-Sharpe JH, Fraser NW. J Gen Virol; 1990 Apr; 71 ( Pt 4)():953-7. PubMed ID: 2157803 [Abstract] [Full Text] [Related]
32. A herpes simplex virus type 1 mutant with a deletion immediately upstream of the LAT locus establishes latency and reactivates from latently infected mice with normal kinetics. Maggioncalda J, Mehta A, Bagasra O, Fraser NW, Block TM. J Neurovirol; 1996 Aug; 2(4):268-78. PubMed ID: 8799218 [Abstract] [Full Text] [Related]
36. The abundant latency-associated transcripts of herpes simplex virus type 1 are bound to polyribosomes in cultured neuronal cells and during latent infection in mouse trigeminal ganglia. Goldenberg D, Mador N, Ball MJ, Panet A, Steiner I. J Virol; 1997 Apr; 71(4):2897-904. PubMed ID: 9060647 [Abstract] [Full Text] [Related]
38. Mapping of low abundance latency-associated RNA in the trigeminal ganglia of mice latently infected with herpes simplex virus type 1. Mitchell WJ, Lirette RP, Fraser NW. J Gen Virol; 1990 Jan; 71 ( Pt 1)():125-32. PubMed ID: 2154530 [Abstract] [Full Text] [Related]
39. The region of the herpes simplex virus type 1 LAT gene involved in spontaneous reactivation does not encode a functional protein. Drolet BS, Perng GC, Cohen J, Slanina SM, Yukht A, Nesburn AB, Wechsler SL. Virology; 1998 Mar 01; 242(1):221-32. PubMed ID: 9501054 [Abstract] [Full Text] [Related]