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Journal Abstract Search

403 related items for PubMed ID: 16621870

  • 1. Prasugrel achieves greater inhibition of platelet aggregation and a lower rate of non-responders compared with clopidogrel in aspirin-treated patients with stable coronary artery disease.
    Jernberg T, Payne CD, Winters KJ, Darstein C, Brandt JT, Jakubowski JA, Naganuma H, Siegbahn A, Wallentin L.
    Eur Heart J; 2006 May; 27(10):1166-73. PubMed ID: 16621870
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  • 3. Greater reduction of platelet activation markers and platelet-monocyte aggregates by prasugrel compared to clopidogrel in stable coronary artery disease.
    Braun OO, Johnell M, Varenhorst C, James S, Brandt JT, Jakubowski JA, Winters KJ, Wallentin L, Erlinge D, Siegbahn A.
    Thromb Haemost; 2008 Oct; 100(4):626-33. PubMed ID: 18841285
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  • 5. Switching directly to prasugrel from clopidogrel results in greater inhibition of platelet aggregation in aspirin-treated subjects.
    Payne CD, Li YG, Brandt JT, Jakubowski JA, Small DS, Farid NA, Salazar DE, Winters KJ.
    Platelets; 2008 Jun; 19(4):275-81. PubMed ID: 18569863
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  • 7. Dose-dependent inhibition of human platelet aggregation by prasugrel and its interaction with aspirin in healthy subjects.
    Jakubowski JA, Payne CD, Weerakkody GJ, Brandt JT, Farid NA, Li YG, Naganuma H, Lachno DR, Winters KJ.
    J Cardiovasc Pharmacol; 2007 Mar; 49(3):167-73. PubMed ID: 17414229
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  • 11. Recovery of platelet function after discontinuation of prasugrel or clopidogrel maintenance dosing in aspirin-treated patients with stable coronary disease: the recovery trial.
    Price MJ, Walder JS, Baker BA, Heiselman DE, Jakubowski JA, Logan DK, Winters KJ, Li W, Angiolillo DJ.
    J Am Coll Cardiol; 2012 Jun 19; 59(25):2338-43. PubMed ID: 22698488
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  • 12. Increased active metabolite formation explains the greater platelet inhibition with prasugrel compared to high-dose clopidogrel.
    Payne CD, Li YG, Small DS, Ernest CS, Farid NA, Jakubowski JA, Brandt JT, Salazar DE, Winters KJ.
    J Cardiovasc Pharmacol; 2007 Nov 19; 50(5):555-62. PubMed ID: 18030066
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  • 13. A comparison of the antiplatelet effects of prasugrel and high-dose clopidogrel as assessed by VASP-phosphorylation and light transmission aggregometry.
    Jakubowski JA, Payne CD, Li YG, Farid NA, Brandt JT, Small DS, Salazar DE, Winters KJ.
    Thromb Haemost; 2008 Jan 19; 99(1):215-22. PubMed ID: 18217157
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  • 14. The use of the VerifyNow P2Y12 point-of-care device to monitor platelet function across a range of P2Y12 inhibition levels following prasugrel and clopidogrel administration.
    Jakubowski JA, Payne CD, Li YG, Brandt JT, Small DS, Farid NA, Salazar DE, Winters KJ.
    Thromb Haemost; 2008 Feb 19; 99(2):409-15. PubMed ID: 18278193
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  • 16. Patients with poor responsiveness to thienopyridine treatment or with diabetes have lower levels of circulating active metabolite, but their platelets respond normally to active metabolite added ex vivo.
    Erlinge D, Varenhorst C, Braun OO, James S, Winters KJ, Jakubowski JA, Brandt JT, Sugidachi A, Siegbahn A, Wallentin L.
    J Am Coll Cardiol; 2008 Dec 09; 52(24):1968-77. PubMed ID: 19055987
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  • 17. Effect of ranitidine on the pharmacokinetics and pharmacodynamics of prasugrel and clopidogrel.
    Small DS, Farid NA, Li YG, Ernest CS, Payne CD, Salazar DE, Winters KJ.
    Curr Med Res Opin; 2008 Aug 09; 24(8):2251-7. PubMed ID: 18786302
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  • 19. Greater inhibition of platelet aggregation and reduced response variability with prasugrel versus clopidogrel: an integrated analysis.
    Weerakkody GJ, Jakubowski JA, Brandt JT, Payne CD, Naganuma H, Winters KJ.
    J Cardiovasc Pharmacol Ther; 2007 Sep 09; 12(3):205-12. PubMed ID: 17875947
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