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Journal Abstract Search

289 related items for PubMed ID: 1672117

  • 1. The human t(1;19) translocation in pre-B ALL produces multiple nuclear E2A-Pbx1 fusion proteins with differing transforming potentials.
    Kamps MP, Look AT, Baltimore D.
    Genes Dev; 1991 Mar; 5(3):358-68. PubMed ID: 1672117
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  • 4. Expression of identical E2A/PBX1 fusion transcripts occurs in both pre-B and early pre-B immunological subtypes of childhood acute lymphoblastic leukemia.
    Izraeli S, Henn T, Strobl H, Ludwig WD, Kovar H, Haas OA, Harbott J, Bartram CR, Gadner H, Lion T.
    Leukemia; 1993 Dec; 7(12):2054-6. PubMed ID: 8255105
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  • 6. An inhibitory switch derepressed by pbx, hox, and Meis/Prep1 partners regulates DNA-binding by pbx1 and E2a-pbx1 and is dispensable for myeloid immortalization by E2a-pbx1.
    Calvo KR, Knoepfler P, McGrath S, Kamps MP.
    Oncogene; 1999 Dec 23; 18(56):8033-43. PubMed ID: 10637514
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  • 9. The highest affinity DNA element bound by Pbx complexes in t(1;19) leukemic cells fails to mediate cooperative DNA-binding or cooperative transactivation by E2a-Pbx1 and class I Hox proteins - evidence for selective targetting of E2a-Pbx1 to a subset of Pbx-recognition elements.
    Knoepfler PS, Kamps MP.
    Oncogene; 1997 May 29; 14(21):2521-31. PubMed ID: 9191052
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  • 10. Monoclonal antibodies specific to the acute lymphoblastic leukemia t(1;19)-associated E2A/pbx1 chimeric protein: characterization and diagnostic utility.
    Sang BC, Shi L, Dias P, Liu L, Wei J, Wang ZX, Monell CR, Behm F, Gruenwald S.
    Blood; 1997 Apr 15; 89(8):2909-14. PubMed ID: 9108411
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  • 12. The Hox cooperativity motif of the chimeric oncoprotein E2a-Pbx1 is necessary and sufficient for oncogenesis.
    Chang CP, de Vivo I, Cleary ML.
    Mol Cell Biol; 1997 Jan 15; 17(1):81-8. PubMed ID: 8972188
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  • 14. The t(1;19)(q23;p13) results in consistent fusion of E2A and PBX1 coding sequences in acute lymphoblastic leukemias.
    Hunger SP, Galili N, Carroll AJ, Crist WM, Link MP, Cleary ML.
    Blood; 1991 Feb 15; 77(4):687-93. PubMed ID: 1671560
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  • 15. Site-specific translocation and evidence of postnatal origin of the t(1;19) E2A-PBX1 fusion in childhood acute lymphoblastic leukemia.
    Wiemels JL, Leonard BC, Wang Y, Segal MR, Hunger SP, Smith MT, Crouse V, Ma X, Buffler PA, Pine SR.
    Proc Natl Acad Sci U S A; 2002 Nov 12; 99(23):15101-6. PubMed ID: 12415113
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  • 16. [E2A gene in t(1;19)-ALL].
    Hayashi Y.
    Nihon Rinsho; 1992 Jun 12; 50(6):1369-73. PubMed ID: 1355570
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  • 17. Pbx1 is converted into a transcriptional activator upon acquiring the N-terminal region of E2A in pre-B-cell acute lymphoblastoid leukemia.
    Van Dijk MA, Voorhoeve PM, Murre C.
    Proc Natl Acad Sci U S A; 1993 Jul 01; 90(13):6061-5. PubMed ID: 8327485
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  • 18. The effects of siRNA-mediated inhibition of E2A-PBX1 on EB-1 and Wnt16b expression in the 697 pre-B leukemia cell line.
    Casagrande G, te Kronnie G, Basso G.
    Haematologica; 2006 Jun 01; 91(6):765-71. PubMed ID: 16769578
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  • 19. [Childhood acute lymphoblastic leukemia with t(1;19) lacking E2A-pBX1 chimeric transcripts].
    Matsubara K, Hirata T.
    Rinsho Ketsueki; 2005 Jan 01; 46(1):7-12. PubMed ID: 16708911
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  • 20. DNA-binding by oncoprotein E2a-Pbx1 is important for blocking differentiation but dispensable for fibroblast transformation.
    Kamps MP, Wright DD, Lu Q.
    Oncogene; 1996 Jan 04; 12(1):19-30. PubMed ID: 8552391
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