These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

234 related items for PubMed ID: 16732486

  • 1. Functional role of phosphatidylinositol 3-kinase/Akt pathway on cell growth and lytic cycle of Epstein-Barr virus in the Burkitt's lymphoma cell line, P3HR-1.
    Mori T, Sairenji T.
    Virus Genes; 2006 Jun; 32(3):327-34. PubMed ID: 16732486
    [Abstract] [Full Text] [Related]

  • 2. (-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.
    Liu S, Li H, Chen L, Yang L, Li L, Tao Y, Li W, Li Z, Liu H, Tang M, Bode AM, Dong Z, Cao Y.
    Carcinogenesis; 2013 Mar; 34(3):627-37. PubMed ID: 23180656
    [Abstract] [Full Text] [Related]

  • 3. LY294002-mediated inhibition of phosphatidylinositol 3-kinase activity triggers growth inhibition and apoptosis in CD40-triggered Ramos-Burkitt lymphoma B cells.
    Curnock AP, Knox KA.
    Cell Immunol; 1998 Aug 01; 187(2):77-87. PubMed ID: 9732695
    [Abstract] [Full Text] [Related]

  • 4. Inhibition of phosphatidylinositol 3'-kinase/AKT signaling promotes apoptosis of primary effusion lymphoma cells.
    Uddin S, Hussain AR, Al-Hussein KA, Manogaran PS, Wickrema A, Gutierrez MI, Bhatia KG.
    Clin Cancer Res; 2005 Apr 15; 11(8):3102-8. PubMed ID: 15837766
    [Abstract] [Full Text] [Related]

  • 5. Epstein-Barr virus latent membrane protein 2A mediates transformation through constitutive activation of the Ras/PI3-K/Akt Pathway.
    Fukuda M, Longnecker R.
    J Virol; 2007 Sep 15; 81(17):9299-306. PubMed ID: 17582000
    [Abstract] [Full Text] [Related]

  • 6. Phosphatidylinositol 3-kinase is a determinant of responsiveness to B cell antigen receptor-mediated Epstein-Barr virus activation.
    Iwakiri D, Takada K.
    J Immunol; 2004 Feb 01; 172(3):1561-6. PubMed ID: 14734735
    [Abstract] [Full Text] [Related]

  • 7. Epstein-Barr virus immediate-early protein BRLF1 induces the lytic form of viral replication through a mechanism involving phosphatidylinositol-3 kinase activation.
    Darr CD, Mauser A, Kenney S.
    J Virol; 2001 Jul 01; 75(13):6135-42. PubMed ID: 11390615
    [Abstract] [Full Text] [Related]

  • 8. hTERT inhibition triggers Epstein-Barr virus lytic cycle and apoptosis in immortalized and transformed B cells: a basis for new therapies.
    Giunco S, Dolcetti R, Keppel S, Celeghin A, Indraccolo S, Dal Col J, Mastorci K, De Rossi A.
    Clin Cancer Res; 2013 Apr 15; 19(8):2036-47. PubMed ID: 23444223
    [Abstract] [Full Text] [Related]

  • 9. IRF4 promotes Epstein-Barr virus activation in Burkitt's lymphoma cells.
    Gao Y, Wang L, Lei Z, Li J, Forrest JC, Liang X.
    J Gen Virol; 2019 May 15; 100(5):851-862. PubMed ID: 30907723
    [Abstract] [Full Text] [Related]

  • 10. Resveratrol inhibits proliferation and survival of Epstein Barr virus-infected Burkitt's lymphoma cells depending on viral latency program.
    De Leo A, Arena G, Stecca C, Raciti M, Mattia E.
    Mol Cancer Res; 2011 Oct 15; 9(10):1346-55. PubMed ID: 21856773
    [Abstract] [Full Text] [Related]

  • 11.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 12.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 13. Induction of Epstein-Barr virus (EBV) reactivation in Raji cells by doxorubicin and cisplatin.
    Hsu CH, Hergenhahn M, Chuang SE, Yeh PY, Wu TC, Gao M, Cheng AL.
    Anticancer Res; 2002 Oct 15; 22(6C):4065-71. PubMed ID: 12553034
    [Abstract] [Full Text] [Related]

  • 14. Activation of the lytic program of the Epstein-Barr virus in Burkitt's lymphoma cells leads to a two steps downregulation of expression of the proapoptotic protein BimEL, one of which is EBV-late-gene expression dependent.
    Oussaief L, Hippocrate A, Clybouw C, Rampanou A, Ramirez V, Desgranges C, Vazquez A, Khelifa R, Joab I.
    Virology; 2009 Apr 25; 387(1):41-9. PubMed ID: 19254802
    [Abstract] [Full Text] [Related]

  • 15.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 16.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 17. Down-regulation of spontaneous Epstein-Barr virus reactivation in the P3HR-1 cell line by L-arginine.
    Agawa H, Ikuta K, Minamiyama Y, Inoue M, Sairenji T.
    Virology; 2002 Dec 05; 304(1):114-24. PubMed ID: 12490409
    [Abstract] [Full Text] [Related]

  • 18.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 19. Activation of mammalian target of rapamycin in transformed B lymphocytes is nutrient dependent but independent of Akt, mitogen-activated protein kinase/extracellular signal-regulated kinase kinase, insulin growth factor-I, and serum.
    Wlodarski P, Kasprzycka M, Liu X, Marzec M, Robertson ES, Slupianek A, Wasik MA.
    Cancer Res; 2005 Sep 01; 65(17):7800-8. PubMed ID: 16140948
    [Abstract] [Full Text] [Related]

  • 20. TLR9 triggering in Burkitt's lymphoma cell lines suppresses the EBV BZLF1 transcription via histone modification.
    Zauner L, Melroe GT, Sigrist JA, Rechsteiner MP, Dorner M, Arnold M, Berger C, Bernasconi M, Schaefer BW, Speck RF, Nadal D.
    Oncogene; 2010 Aug 12; 29(32):4588-98. PubMed ID: 20514021
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 12.