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Journal Abstract Search

445 related items for PubMed ID: 16735477

  • 21. Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy.
    Schroeder BC, Kubisch C, Stein V, Jentsch TJ.
    Nature; 1998 Dec 17; 396(6712):687-90. PubMed ID: 9872318
    [Abstract] [Full Text] [Related]

  • 22. Mouse models of human KCNQ2 and KCNQ3 mutations for benign familial neonatal convulsions show seizures and neuronal plasticity without synaptic reorganization.
    Singh NA, Otto JF, Dahle EJ, Pappas C, Leslie JD, Vilaythong A, Noebels JL, White HS, Wilcox KS, Leppert MF.
    J Physiol; 2008 Jul 15; 586(14):3405-23. PubMed ID: 18483067
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  • 23. Reduced axonal surface expression and phosphoinositide sensitivity in Kv7 channels disrupts their function to inhibit neuronal excitability in Kcnq2 epileptic encephalopathy.
    Kim EC, Zhang J, Pang W, Wang S, Lee KY, Cavaretta JP, Walters J, Procko E, Tsai NP, Chung HJ.
    Neurobiol Dis; 2018 Oct 15; 118():76-93. PubMed ID: 30008368
    [Abstract] [Full Text] [Related]

  • 24. The KCNQ2/3 selective channel opener ICA-27243 binds to a novel voltage-sensor domain site.
    Padilla K, Wickenden AD, Gerlach AC, McCormack K.
    Neurosci Lett; 2009 Nov 13; 465(2):138-42. PubMed ID: 19733209
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  • 25. KCNQ2 and KCNQ3 potassium channel genes in benign familial neonatal convulsions: expansion of the functional and mutation spectrum.
    Singh NA, Westenskow P, Charlier C, Pappas C, Leslie J, Dillon J, Anderson VE, Sanguinetti MC, Leppert MF, BFNC Physician Consortium.
    Brain; 2003 Dec 13; 126(Pt 12):2726-37. PubMed ID: 14534157
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  • 26. Sequence determinants of subtype-specific actions of KCNQ channel openers.
    Wang AW, Yang R, Kurata HT.
    J Physiol; 2017 Feb 01; 595(3):663-676. PubMed ID: 27506413
    [Abstract] [Full Text] [Related]

  • 27. Lack of potassium current in W309R mutant KCNQ3 channel causing benign familial neonatal convulsions (BFNC).
    Sugiura Y, Nakatsu F, Hiroyasu K, Ishii A, Hirose S, Okada M, Jibiki I, Ohno H, Kaneko S, Ugawa Y.
    Epilepsy Res; 2009 Mar 01; 84(1):82-5. PubMed ID: 19167866
    [Abstract] [Full Text] [Related]

  • 28. Functional expression of two KvLQT1-related potassium channels responsible for an inherited idiopathic epilepsy.
    Yang WP, Levesque PC, Little WA, Conder ML, Ramakrishnan P, Neubauer MG, Blanar MA.
    J Biol Chem; 1998 Jul 31; 273(31):19419-23. PubMed ID: 9677360
    [Abstract] [Full Text] [Related]

  • 29. KCNQ2 is a nodal K+ channel.
    Devaux JJ, Kleopa KA, Cooper EC, Scherer SS.
    J Neurosci; 2004 Feb 04; 24(5):1236-44. PubMed ID: 14762142
    [Abstract] [Full Text] [Related]

  • 30. Surface expression and single channel properties of KCNQ2/KCNQ3, M-type K+ channels involved in epilepsy.
    Schwake M, Pusch M, Kharkovets T, Jentsch TJ.
    J Biol Chem; 2000 May 05; 275(18):13343-8. PubMed ID: 10788442
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  • 33. M-channels: neurological diseases, neuromodulation, and drug development.
    Cooper EC, Jan LY.
    Arch Neurol; 2003 Apr 05; 60(4):496-500. PubMed ID: 12707061
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  • 36. Neuronal KCNQ potassium channels: physiology and role in disease.
    Jentsch TJ.
    Nat Rev Neurosci; 2000 Oct 05; 1(1):21-30. PubMed ID: 11252765
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  • 37. Expression and localization of K channels KCNQ2 and KCNQ3 in the mammalian cochlea.
    Jin Z, Liang GH, Cooper EC, Jarlebark L.
    Audiol Neurootol; 2009 Oct 05; 14(2):98-105. PubMed ID: 18827480
    [Abstract] [Full Text] [Related]

  • 38. Atypical gating of M-type potassium channels conferred by mutations in uncharged residues in the S4 region of KCNQ2 causing benign familial neonatal convulsions.
    Soldovieri MV, Cilio MR, Miceli F, Bellini G, Miraglia del Giudice E, Castaldo P, Hernandez CC, Shapiro MS, Pascotto A, Annunziato L, Taglialatela M.
    J Neurosci; 2007 May 02; 27(18):4919-28. PubMed ID: 17475800
    [Abstract] [Full Text] [Related]

  • 39. Triclosan is a KCNQ3 potassium channel activator.
    De la Rosa V, Guzmán-Hernández ML, Carrillo E.
    Pflugers Arch; 2022 Jul 02; 474(7):721-732. PubMed ID: 35459955
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