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Journal Abstract Search

466 related items for PubMed ID: 16741751

  • 1. Modulation of Hsp90 function in neurodegenerative disorders: a molecular-targeted therapy against disease-causing protein.
    Waza M, Adachi H, Katsuno M, Minamiyama M, Tanaka F, Doyu M, Sobue G.
    J Mol Med (Berl); 2006 Aug; 84(8):635-46. PubMed ID: 16741751
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  • 3. 17-AAG, an Hsp90 inhibitor, ameliorates polyglutamine-mediated motor neuron degeneration.
    Waza M, Adachi H, Katsuno M, Minamiyama M, Sang C, Tanaka F, Inukai A, Doyu M, Sobue G.
    Nat Med; 2005 Oct; 11(10):1088-95. PubMed ID: 16155577
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  • 4. Pathogenesis and molecular targeted therapy of spinal and bulbar muscular atrophy.
    Adachi H, Waza M, Katsuno M, Tanaka F, Doyu M, Sobue G.
    Neuropathol Appl Neurobiol; 2007 Apr; 33(2):135-51. PubMed ID: 17359355
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  • 5. 17-DMAG ameliorates polyglutamine-mediated motor neuron degeneration through well-preserved proteasome function in an SBMA model mouse.
    Tokui K, Adachi H, Waza M, Katsuno M, Minamiyama M, Doi H, Tanaka K, Hamazaki J, Murata S, Tanaka F, Sobue G.
    Hum Mol Genet; 2009 Mar 01; 18(5):898-910. PubMed ID: 19066230
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  • 6. Enhanced aggregation of androgen receptor in induced pluripotent stem cell-derived neurons from spinal and bulbar muscular atrophy.
    Nihei Y, Ito D, Okada Y, Akamatsu W, Yagi T, Yoshizaki T, Okano H, Suzuki N.
    J Biol Chem; 2013 Mar 22; 288(12):8043-8052. PubMed ID: 23364790
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  • 7. Heat shock transcription factor 1-activating compounds suppress polyglutamine-induced neurodegeneration through induction of multiple molecular chaperones.
    Fujikake N, Nagai Y, Popiel HA, Okamoto Y, Yamaguchi M, Toda T.
    J Biol Chem; 2008 Sep 19; 283(38):26188-97. PubMed ID: 18632670
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  • 9. 17-AAG increases autophagic removal of mutant androgen receptor in spinal and bulbar muscular atrophy.
    Rusmini P, Simonini F, Crippa V, Bolzoni E, Onesto E, Cagnin M, Sau D, Ferri N, Poletti A.
    Neurobiol Dis; 2011 Jan 19; 41(1):83-95. PubMed ID: 20816782
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  • 10. 17-AAG improves cognitive process and increases heat shock protein response in a model lesion with Aβ25-35.
    Ortega L, Calvillo M, Luna F, Pérez-Severiano F, Rubio-Osornio M, Guevara J, Limón ID.
    Neuropeptides; 2014 Aug 19; 48(4):221-32. PubMed ID: 24819277
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  • 11. Molecular mechanism of 17-allylamino-17-demethoxygeldanamycin (17-AAG)-induced AXL receptor tyrosine kinase degradation.
    Krishnamoorthy GP, Guida T, Alfano L, Avilla E, Santoro M, Carlomagno F, Melillo RM.
    J Biol Chem; 2013 Jun 14; 288(24):17481-94. PubMed ID: 23629654
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  • 12. Heat shock protein 70 chaperone overexpression ameliorates phenotypes of the spinal and bulbar muscular atrophy transgenic mouse model by reducing nuclear-localized mutant androgen receptor protein.
    Adachi H, Katsuno M, Minamiyama M, Sang C, Pagoulatos G, Angelidis C, Kusakabe M, Yoshiki A, Kobayashi Y, Doyu M, Sobue G.
    J Neurosci; 2003 Mar 15; 23(6):2203-11. PubMed ID: 12657679
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  • 15. Heat shock proteins in neurodegenerative diseases: pathogenic roles and therapeutic implications.
    Adachi H, Katsuno M, Waza M, Minamiyama M, Tanaka F, Sobue G.
    Int J Hyperthermia; 2009 Dec 15; 25(8):647-54. PubMed ID: 20021225
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  • 18. Sulforaphane potentiates the efficacy of 17-allylamino 17-demethoxygeldanamycin against pancreatic cancer through enhanced abrogation of Hsp90 chaperone function.
    Li Y, Zhang T, Schwartz SJ, Sun D.
    Nutr Cancer; 2011 Dec 15; 63(7):1151-9. PubMed ID: 21875325
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  • 19. Effects of HSP90 inhibitor 17-allylamino-17-demethoxygeldanamycin (17-AAG) on NEU/HER2 overexpressing mammary tumours in MMTV-NEU-NT mice monitored by Magnetic Resonance Spectroscopy.
    Rodrigues LM, Chung YL, Al Saffar NM, Sharp SY, Jackson LE, Banerji U, Stubbs M, Leach MO, Griffiths JR, Workman P.
    BMC Res Notes; 2012 May 23; 5():250. PubMed ID: 22621282
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  • 20. Pathogenesis and therapy of spinal and bulbar muscular atrophy (SBMA).
    Katsuno M, Tanaka F, Adachi H, Banno H, Suzuki K, Watanabe H, Sobue G.
    Prog Neurobiol; 2012 Dec 23; 99(3):246-56. PubMed ID: 22609045
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