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646 related items for PubMed ID: 16807352
1. Contribution of persistent Na+ current and M-type K+ current to somatic bursting in CA1 pyramidal cells: combined experimental and modeling study. Golomb D, Yue C, Yaari Y. J Neurophysiol; 2006 Oct; 96(4):1912-26. PubMed ID: 16807352 [Abstract] [Full Text] [Related]
2. Fast rhythmic bursting can be induced in layer 2/3 cortical neurons by enhancing persistent Na+ conductance or by blocking BK channels. Traub RD, Buhl EH, Gloveli T, Whittington MA. J Neurophysiol; 2003 Feb; 89(2):909-21. PubMed ID: 12574468 [Abstract] [Full Text] [Related]
3. SK (KCa2) channels do not control somatic excitability in CA1 pyramidal neurons but can be activated by dendritic excitatory synapses and regulate their impact. Gu N, Hu H, Vervaeke K, Storm JF. J Neurophysiol; 2008 Nov; 100(5):2589-604. PubMed ID: 18684909 [Abstract] [Full Text] [Related]
9. Persistent sodium currents in mesencephalic v neurons participate in burst generation and control of membrane excitability. Wu N, Enomoto A, Tanaka S, Hsiao CF, Nykamp DQ, Izhikevich E, Chandler SH. J Neurophysiol; 2005 May 15; 93(5):2710-22. PubMed ID: 15625100 [Abstract] [Full Text] [Related]
10. Emergence of intrinsic bursting in trigeminal sensory neurons parallels the acquisition of mastication in weanling rats. Brocard F, Verdier D, Arsenault I, Lund JP, Kolta A. J Neurophysiol; 2006 Nov 15; 96(5):2410-24. PubMed ID: 16914618 [Abstract] [Full Text] [Related]
15. Kv7/KCNQ/M and HCN/h, but not KCa2/SK channels, contribute to the somatic medium after-hyperpolarization and excitability control in CA1 hippocampal pyramidal cells. Gu N, Vervaeke K, Hu H, Storm JF. J Physiol; 2005 Aug 01; 566(Pt 3):689-715. PubMed ID: 15890705 [Abstract] [Full Text] [Related]
17. Mechanisms of neuronal hyperexcitability caused by partial inhibition of Na+-K+-ATPases in the rat CA1 hippocampal region. Vaillend C, Mason SE, Cuttle MF, Alger BE. J Neurophysiol; 2002 Dec 01; 88(6):2963-78. PubMed ID: 12466422 [Abstract] [Full Text] [Related]