These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

417 related items for PubMed ID: 16822974

  • 1. Impaired firing and cell-specific compensation in neurons lacking nav1.6 sodium channels.
    Van Wart A, Matthews G.
    J Neurosci; 2006 Jul 05; 26(27):7172-80. PubMed ID: 16822974
    [Abstract] [Full Text] [Related]

  • 2. The contribution of resurgent sodium current to high-frequency firing in Purkinje neurons: an experimental and modeling study.
    Khaliq ZM, Gouwens NW, Raman IM.
    J Neurosci; 2003 Jun 15; 23(12):4899-912. PubMed ID: 12832512
    [Abstract] [Full Text] [Related]

  • 3. Functional specialization of the axon initial segment by isoform-specific sodium channel targeting.
    Boiko T, Van Wart A, Caldwell JH, Levinson SR, Trimmer JS, Matthews G.
    J Neurosci; 2003 Mar 15; 23(6):2306-13. PubMed ID: 12657689
    [Abstract] [Full Text] [Related]

  • 4. Paranodal interactions regulate expression of sodium channel subtypes and provide a diffusion barrier for the node of Ranvier.
    Rios JC, Rubin M, St Martin M, Downey RT, Einheber S, Rosenbluth J, Levinson SR, Bhat M, Salzer JL.
    J Neurosci; 2003 Aug 06; 23(18):7001-11. PubMed ID: 12904461
    [Abstract] [Full Text] [Related]

  • 5. Reduced sodium current in Purkinje neurons from Nav1.1 mutant mice: implications for ataxia in severe myoclonic epilepsy in infancy.
    Kalume F, Yu FH, Westenbroek RE, Scheuer T, Catterall WA.
    J Neurosci; 2007 Oct 10; 27(41):11065-74. PubMed ID: 17928448
    [Abstract] [Full Text] [Related]

  • 6. Mice lacking sodium channel beta1 subunits display defects in neuronal excitability, sodium channel expression, and nodal architecture.
    Chen C, Westenbroek RE, Xu X, Edwards CA, Sorenson DR, Chen Y, McEwen DP, O'Malley HA, Bharucha V, Meadows LS, Knudsen GA, Vilaythong A, Noebels JL, Saunders TL, Scheuer T, Shrager P, Catterall WA, Isom LL.
    J Neurosci; 2004 Apr 21; 24(16):4030-42. PubMed ID: 15102918
    [Abstract] [Full Text] [Related]

  • 7.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 8. Single amino acid deletion in transmembrane segment D4S6 of sodium channel Scn8a (Nav1.6) in a mouse mutant with a chronic movement disorder.
    Jones JM, Dionne L, Dell'Orco J, Parent R, Krueger JN, Cheng X, Dib-Hajj SD, Bunton-Stasyshyn RK, Sharkey LM, Dowling JJ, Murphy GG, Shakkottai VG, Shrager P, Meisler MH.
    Neurobiol Dis; 2016 May 21; 89():36-45. PubMed ID: 26807988
    [Abstract] [Full Text] [Related]

  • 9. Expression of sodium channels Nav1.2 and Nav1.6 during postnatal development of the retina.
    Van Wart A, Matthews G.
    Neurosci Lett; 2006 Aug 07; 403(3):315-7. PubMed ID: 16753259
    [Abstract] [Full Text] [Related]

  • 10. Fibroblast growth factor homologous factor 2B: association with Nav1.6 and selective colocalization at nodes of Ranvier of dorsal root axons.
    Wittmack EK, Rush AM, Craner MJ, Goldfarb M, Waxman SG, Dib-Hajj SD.
    J Neurosci; 2004 Jul 28; 24(30):6765-75. PubMed ID: 15282281
    [Abstract] [Full Text] [Related]

  • 11. Where is the spike generator of the cochlear nerve? Voltage-gated sodium channels in the mouse cochlea.
    Hossain WA, Antic SD, Yang Y, Rasband MN, Morest DK.
    J Neurosci; 2005 Jul 20; 25(29):6857-68. PubMed ID: 16033895
    [Abstract] [Full Text] [Related]

  • 12. An ankyrinG-binding motif is necessary and sufficient for targeting Nav1.6 sodium channels to axon initial segments and nodes of Ranvier.
    Gasser A, Ho TS, Cheng X, Chang KJ, Waxman SG, Rasband MN, Dib-Hajj SD.
    J Neurosci; 2012 May 23; 32(21):7232-43. PubMed ID: 22623668
    [Abstract] [Full Text] [Related]

  • 13. Nav1.1 is predominantly expressed in nodes of Ranvier and axon initial segments.
    Duflocq A, Le Bras B, Bullier E, Couraud F, Davenne M.
    Mol Cell Neurosci; 2008 Oct 23; 39(2):180-92. PubMed ID: 18621130
    [Abstract] [Full Text] [Related]

  • 14. Paranodal axoglial junction is required for the maintenance of the Nav1.6-type sodium channel in the node of Ranvier in the optic nerves but not in peripheral nerve fibers in the sulfatide-deficient mice.
    Suzuki A, Hoshi T, Ishibashi T, Hayashi A, Yamaguchi Y, Baba H.
    Glia; 2004 May 23; 46(3):274-83. PubMed ID: 15048850
    [Abstract] [Full Text] [Related]

  • 15. Subunit dependence of Na channel slow inactivation and open channel block in cerebellar neurons.
    Aman TK, Raman IM.
    Biophys J; 2007 Mar 15; 92(6):1938-51. PubMed ID: 17189307
    [Abstract] [Full Text] [Related]

  • 16. Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth.
    Brackenbury WJ, Calhoun JD, Chen C, Miyazaki H, Nukina N, Oyama F, Ranscht B, Isom LL.
    Proc Natl Acad Sci U S A; 2010 Feb 02; 107(5):2283-8. PubMed ID: 20133873
    [Abstract] [Full Text] [Related]

  • 17. Production of resurgent current in NaV1.6-null Purkinje neurons by slowing sodium channel inactivation with beta-pompilidotoxin.
    Grieco TM, Raman IM.
    J Neurosci; 2004 Jan 07; 24(1):35-42. PubMed ID: 14715935
    [Abstract] [Full Text] [Related]

  • 18.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 19. FGF14 regulates the intrinsic excitability of cerebellar Purkinje neurons.
    Shakkottai VG, Xiao M, Xu L, Wong M, Nerbonne JM, Ornitz DM, Yamada KA.
    Neurobiol Dis; 2009 Jan 07; 33(1):81-8. PubMed ID: 18930825
    [Abstract] [Full Text] [Related]

  • 20. Robustness of burst firing in dissociated purkinje neurons with acute or long-term reductions in sodium conductance.
    Swensen AM, Bean BP.
    J Neurosci; 2005 Apr 06; 25(14):3509-20. PubMed ID: 15814781
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 21.