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Journal Abstract Search

118 related items for PubMed ID: 16856153

  • 21.
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  • 22. Muscle-derived but not centrally derived transgene GDNF is neuroprotective in G93A-SOD1 mouse model of ALS.
    Li W, Brakefield D, Pan Y, Hunter D, Myckatyn TM, Parsadanian A.
    Exp Neurol; 2007 Feb; 203(2):457-71. PubMed ID: 17034790
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  • 25. Motor neuron dysfunction in a mouse model of ALS: gender-dependent effect of P2X7 antagonism.
    Cervetto C, Frattaroli D, Maura G, Marcoli M.
    Toxicology; 2013 Sep 06; 311(1-2):69-77. PubMed ID: 23583883
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  • 26. Absence of p53: no effect in a transgenic mouse model of familial amyotrophic lateral sclerosis.
    Kuntz C, Kinoshita Y, Beal MF, Donehower LA, Morrison RS.
    Exp Neurol; 2000 Sep 06; 165(1):184-90. PubMed ID: 10964497
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  • 29. The lack of effect of specific overexpression of IGF-1 in the central nervous system or skeletal muscle on pathophysiology in the G93A SOD-1 mouse model of ALS.
    Messi ML, Clark HM, Prevette DM, Oppenheim RW, Delbono O.
    Exp Neurol; 2007 Sep 06; 207(1):52-63. PubMed ID: 17597610
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  • 31. Impairments to the GH-IGF-I axis in hSOD1G93A mice give insight into possible mechanisms of GH dysregulation in patients with amyotrophic lateral sclerosis.
    Steyn FJ, Ngo ST, Lee JD, Leong JW, Buckley AJ, Veldhuis JD, McCombe PA, Chen C, Bellingham MC.
    Endocrinology; 2012 Aug 06; 153(8):3735-46. PubMed ID: 22621959
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  • 32. Glutamate release induced by activation of glycine and GABA transporters in spinal cord is enhanced in a mouse model of amyotrophic lateral sclerosis.
    Raiteri L, Zappettini S, Stigliani S, Paluzzi S, Raiteri M, Bonanno G.
    Neurotoxicology; 2005 Oct 06; 26(5):883-92. PubMed ID: 15885796
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  • 35. Increase of disease duration of amyotrophic lateral sclerosis in a mouse model by transgenic small interfering RNA.
    Yokota T, Sasaguri H, Saito Y, Yamada H, Unno T, Yamamoto Y, Kubodera T, Anzai M, Mitani T, Mizusawa H.
    Arch Neurol; 2007 Jan 06; 64(1):145-6. PubMed ID: 17210827
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  • 36. Altered expression of myogenic regulatory factors in the mouse model of amyotrophic lateral sclerosis.
    Manzano R, Toivonen JM, Oliván S, Calvo AC, Moreno-Igoa M, Muñoz MJ, Zaragoza P, García-Redondo A, Osta R.
    Neurodegener Dis; 2011 Jan 06; 8(5):386-96. PubMed ID: 21346327
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  • 37. Molecular signatures of amyotrophic lateral sclerosis disease progression in hind and forelimb muscles of an SOD1(G93A) mouse model.
    Capitanio D, Vasso M, Ratti A, Grignaschi G, Volta M, Moriggi M, Daleno C, Bendotti C, Silani V, Gelfi C.
    Antioxid Redox Signal; 2012 Nov 15; 17(10):1333-50. PubMed ID: 22563797
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  • 38. Properties of slow- and fast-twitch muscle fibres in a mouse model of amyotrophic lateral sclerosis.
    Atkin JD, Scott RL, West JM, Lopes E, Quah AK, Cheema SS.
    Neuromuscul Disord; 2005 May 15; 15(5):377-88. PubMed ID: 15833433
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  • 39. Expression of the axon-guidance protein receptor Neuropilin 1 is increased in the spinal cord and decreased in muscle of a mouse model of amyotrophic lateral sclerosis.
    Körner S, Thau-Habermann N, Kefalakes E, Bursch F, Petri S.
    Eur J Neurosci; 2019 Jun 15; 49(11):1529-1543. PubMed ID: 30589468
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  • 40. Tau isoforms expression in transgenic mouse model of amyotrophic lateral sclerosis.
    Usarek E, Kuźma-Kozakiewicz M, Schwalenstöcker B, Kaźmierczak B, Münch C, Ludolph AC, Barańczyk-Kuźma A.
    Neurochem Res; 2006 May 15; 31(5):597-602. PubMed ID: 16770730
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