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Journal Abstract Search

189 related items for PubMed ID: 16860487

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  • 2. Parvalbumin deficiency in fast-twitch muscles leads to increased 'slow-twitch type' mitochondria, but does not affect the expression of fiber specific proteins.
    Racay P, Gregory P, Schwaller B.
    FEBS J; 2006 Jan; 273(1):96-108. PubMed ID: 16367751
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  • 3. Differences in locomotor behavior revealed in mice deficient for the calcium-binding proteins parvalbumin, calbindin D-28k or both.
    Farré-Castany MA, Schwaller B, Gregory P, Barski J, Mariethoz C, Eriksson JL, Tetko IV, Wolfer D, Celio MR, Schmutz I, Albrecht U, Villa AE.
    Behav Brain Res; 2007 Mar 28; 178(2):250-61. PubMed ID: 17275105
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  • 7. Ectopic parvalbumin expression in mouse forebrain neurons increases excitotoxic injury provoked by ibotenic acid injection into the striatum.
    Maetzler W, Nitsch C, Bendfeldt K, Racay P, Vollenweider F, Schwaller B.
    Exp Neurol; 2004 Mar 28; 186(1):78-88. PubMed ID: 14980812
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  • 8. Mutational analysis of dendritic Ca2+ kinetics in rodent Purkinje cells: role of parvalbumin and calbindin D28k.
    Schmidt H, Stiefel KM, Racay P, Schwaller B, Eilers J.
    J Physiol; 2003 Aug 15; 551(Pt 1):13-32. PubMed ID: 12813159
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  • 9. Compensatory regulation of Cav2.1 Ca2+ channels in cerebellar Purkinje neurons lacking parvalbumin and calbindin D-28k.
    Kreiner L, Christel CJ, Benveniste M, Schwaller B, Lee A.
    J Neurophysiol; 2010 Jan 15; 103(1):371-81. PubMed ID: 19906882
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  • 14. Microcalcification after excitotoxicity is enhanced in transgenic mice expressing parvalbumin in all neurones, may commence in neuronal mitochondria and undergoes structural modifications over time.
    Maetzler W, Stünitz H, Bendfeldt K, Vollenweider F, Schwaller B, Nitsch C.
    Neuropathol Appl Neurobiol; 2009 Apr 15; 35(2):165-77. PubMed ID: 19284478
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  • 15. Dual role of calbindin-D28K in vesicular catecholamine release from mouse chromaffin cells.
    Westerink RH, Rook MB, Beekwilder JP, Wadman WJ.
    J Neurochem; 2006 Oct 15; 99(2):628-40. PubMed ID: 16824046
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  • 16. Absence of parvalbumin increases mitochondria volume and branching of dendrites in inhibitory Pvalb neurons in vivo: a point of convergence of autism spectrum disorder (ASD) risk gene phenotypes.
    Janickova L, Rechberger KF, Wey L, Schwaller B.
    Mol Autism; 2020 Jun 09; 11(1):47. PubMed ID: 32517751
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