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Journal Abstract Search
220 related items for PubMed ID: 16861895
1. Budding Yeast Sae2 is an In Vivo Target of the Mec1 and Tel1 Checkpoint Kinases During Meiosis. Cartagena-Lirola H, Guerini I, Viscardi V, Lucchini G, Longhese MP. Cell Cycle; 2006 Jul; 5(14):1549-59. PubMed ID: 16861895 [Abstract] [Full Text] [Related]
2. The functions of budding yeast Sae2 in the DNA damage response require Mec1- and Tel1-dependent phosphorylation. Baroni E, Viscardi V, Cartagena-Lirola H, Lucchini G, Longhese MP. Mol Cell Biol; 2004 May; 24(10):4151-65. PubMed ID: 15121837 [Abstract] [Full Text] [Related]
3. The Saccharomyces cerevisiae Sae2 protein negatively regulates DNA damage checkpoint signalling. Clerici M, Mantiero D, Lucchini G, Longhese MP. EMBO Rep; 2006 Feb; 7(2):212-8. PubMed ID: 16374511 [Abstract] [Full Text] [Related]
4. Direct and indirect control of the initiation of meiotic recombination by DNA damage checkpoint mechanisms in budding yeast. Argunhan B, Farmer S, Leung WK, Terentyev Y, Humphryes N, Tsubouchi T, Toyoizumi H, Tsubouchi H. PLoS One; 2013 Feb; 8(6):e65875. PubMed ID: 23762445 [Abstract] [Full Text] [Related]
5. Role of the Saccharomyces cerevisiae Rad53 checkpoint kinase in signaling double-strand breaks during the meiotic cell cycle. Cartagena-Lirola H, Guerini I, Manfrini N, Lucchini G, Longhese MP. Mol Cell Biol; 2008 Jul; 28(14):4480-93. PubMed ID: 18505828 [Abstract] [Full Text] [Related]
6. Phosphorylation of the axial element protein Hop1 by Mec1/Tel1 ensures meiotic interhomolog recombination. Carballo JA, Johnson AL, Sedgwick SG, Cha RS. Cell; 2008 Mar 07; 132(5):758-70. PubMed ID: 18329363 [Abstract] [Full Text] [Related]
7. Prevention of DNA Rereplication Through a Meiotic Recombination Checkpoint Response. Najor NA, Weatherford L, Brush GS. G3 (Bethesda); 2016 Dec 07; 6(12):3869-3881. PubMed ID: 27678521 [Abstract] [Full Text] [Related]
8. CDK and Mec1/Tel1-catalyzed phosphorylation of Sae2 regulate different responses to DNA damage. Yu TY, Garcia VE, Symington LS. Nucleic Acids Res; 2019 Dec 02; 47(21):11238-11249. PubMed ID: 31552432 [Abstract] [Full Text] [Related]
9. Positive regulation of meiotic DNA double-strand break formation by activation of the DNA damage checkpoint kinase Mec1(ATR). Gray S, Allison RM, Garcia V, Goldman AS, Neale MJ. Open Biol; 2013 Jul 31; 3(7):130019. PubMed ID: 23902647 [Abstract] [Full Text] [Related]
10. Uncoupling Sae2 Functions in Downregulation of Tel1 and Rad53 Signaling Activities. Colombo CV, Menin L, Ranieri R, Bonetti D, Clerici M, Longhese MP. Genetics; 2019 Feb 31; 211(2):515-530. PubMed ID: 30538107 [Abstract] [Full Text] [Related]
11. Sae2 Function at DNA Double-Strand Breaks Is Bypassed by Dampening Tel1 or Rad53 Activity. Gobbini E, Villa M, Gnugnoli M, Menin L, Clerici M, Longhese MP. PLoS Genet; 2015 Nov 31; 11(11):e1005685. PubMed ID: 26584331 [Abstract] [Full Text] [Related]
12. Mec1 Modulates Interhomolog Crossover and Interplays with Tel1 at Post Double-Strand Break Stages. Lee MS, Joo JW, Choi H, Kang HA, Kim K. J Microbiol Biotechnol; 2020 Mar 28; 30(3):469-475. PubMed ID: 31847509 [Abstract] [Full Text] [Related]
13. Three distinct modes of Mec1/ATR and Tel1/ATM activation illustrate differential checkpoint targeting during budding yeast early meiosis. Cheng YH, Chuang CN, Shen HJ, Lin FM, Wang TF. Mol Cell Biol; 2013 Aug 28; 33(16):3365-76. PubMed ID: 23775120 [Abstract] [Full Text] [Related]
14. A meiotic recombination checkpoint controlled by mitotic checkpoint genes. Lydall D, Nikolsky Y, Bishop DK, Weinert T. Nature; 1996 Oct 31; 383(6603):840-3. PubMed ID: 8893012 [Abstract] [Full Text] [Related]
15. ATR/Mec1 prevents lethal meiotic recombination initiation on partially replicated chromosomes in budding yeast. Blitzblau HG, Hochwagen A. Elife; 2013 Oct 01; 2():e00844. PubMed ID: 24137535 [Abstract] [Full Text] [Related]
16. Pch2 prevents Mec1/Tel1-mediated Hop1 phosphorylation occurring independently of Red1 in budding yeast meiosis. Lo YH, Chuang CN, Wang TF. PLoS One; 2014 Oct 01; 9(1):e85687. PubMed ID: 24465650 [Abstract] [Full Text] [Related]