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Journal Abstract Search

881 related items for PubMed ID: 16893685

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  • 3. Genomic DNA breakpoints in AML1/RUNX1 and ETO cluster with topoisomerase II DNA cleavage and DNase I hypersensitive sites in t(8;21) leukemia.
    Zhang Y, Strissel P, Strick R, Chen J, Nucifora G, Le Beau MM, Larson RA, Rowley JD.
    Proc Natl Acad Sci U S A; 2002 Mar 05; 99(5):3070-5. PubMed ID: 11867721
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  • 4. DNA structural properties of AF9 are similar to MLL and could act as recombination hot spots resulting in MLL/AF9 translocations and leukemogenesis.
    Strissel PL, Strick R, Tomek RJ, Roe BA, Rowley JD, Zeleznik-Le NJ.
    Hum Mol Genet; 2000 Jul 01; 9(11):1671-9. PubMed ID: 10861294
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  • 6. Distribution of 11q23 breakpoints within the MLL breakpoint cluster region in de novo acute leukemia and in treatment-related acute myeloid leukemia: correlation with scaffold attachment regions and topoisomerase II consensus binding sites.
    Broeker PL, Super HG, Thirman MJ, Pomykala H, Yonebayashi Y, Tanabe S, Zeleznik-Le N, Rowley JD.
    Blood; 1996 Mar 01; 87(5):1912-22. PubMed ID: 8634439
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  • 7. Analysis of balanced rearrangements of chromosome 6 in acute leukemia: clustered breakpoints in q22-q23 and possible involvement of c-MYB in a new recurrent translocation, t(6;7)(q23;q32 through 36).
    Sinclair P, Harrison CJ, Jarosová M, Foroni L.
    Haematologica; 2005 May 01; 90(5):602-11. PubMed ID: 15921375
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  • 8. Chromosome translocations: good genes gone wrong.
    Rowley JD.
    Pathol Biol (Paris); 1995 Mar 01; 43(3):197-201. PubMed ID: 7675546
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  • 9. Biochemical mechanisms of chromosomal translocations resulting from DNA double-strand breaks.
    Povirk LF.
    DNA Repair (Amst); 2006 Sep 08; 5(9-10):1199-212. PubMed ID: 16822725
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  • 10. Molecular characterization of genomic AML1-ETO fusions in childhood leukemia.
    Xiao Z, Greaves MF, Buffler P, Smith MT, Segal MR, Dicks BM, Wiencke JK, Wiemels JL.
    Leukemia; 2001 Dec 08; 15(12):1906-13. PubMed ID: 11753612
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  • 11. Structure and possible mechanisms of TEL-AML1 gene fusions in childhood acute lymphoblastic leukemia.
    Wiemels JL, Greaves M.
    Cancer Res; 1999 Aug 15; 59(16):4075-82. PubMed ID: 10463610
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  • 12. Identification of a potential "hotspot" DNA region in the RUNX1 gene targeted by mitoxantrone in therapy-related acute myeloid leukemia with t(16;21) translocation.
    Ottone T, Hasan SK, Montefusco E, Curzi P, Mays AN, Chessa L, Ferrari A, Conte E, Noguera NI, Lavorgna S, Ammatuna E, Divona M, Bovetti K, Amadori S, Grimwade D, Lo-Coco F.
    Genes Chromosomes Cancer; 2009 Mar 15; 48(3):213-21. PubMed ID: 19023877
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  • 13. DNA topoisomerase II in therapy-related acute promyelocytic leukemia.
    Mistry AR, Felix CA, Whitmarsh RJ, Mason A, Reiter A, Cassinat B, Parry A, Walz C, Wiemels JL, Segal MR, Adès L, Blair IA, Osheroff N, Peniket AJ, Lafage-Pochitaloff M, Cross NC, Chomienne C, Solomon E, Fenaux P, Grimwade D.
    N Engl J Med; 2005 Apr 14; 352(15):1529-38. PubMed ID: 15829534
    [Abstract] [Full Text] [Related]

  • 14. Bioflavonoids promote stable translocations between MLL-AF9 breakpoint cluster regions independent of normal chromosomal context: Model system to screen environmental risks.
    Bariar B, Vestal CG, Deem B, Goodenow D, Ughetta M, Engledove RW, Sahyouni M, Richardson C.
    Environ Mol Mutagen; 2019 Mar 14; 60(2):154-167. PubMed ID: 30387535
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  • 15. Clustering of genomic breakpoints at the MLL locus in therapy-related acute leukemia with t(4;11)(q21;q23).
    Hasan SK, Barba G, Metzler M, Divona M, Ottone T, Cicconi L, Falini B, Mecucci C, Lo-Coco F.
    Genes Chromosomes Cancer; 2014 Mar 14; 53(3):248-54. PubMed ID: 24310817
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  • 16. Chromosomal abnormalities in secondary MDS and AML. Relationship to drugs and radiation with specific emphasis on the balanced rearrangements.
    Andersen MK, Johansson B, Larsen SO, Pedersen-Bjergaard J.
    Haematologica; 1998 Jun 14; 83(6):483-8. PubMed ID: 9676019
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  • 17. Simple multiplex RT-PCR for identifying common fusion transcripts in childhood acute leukemia.
    Pakakasama S, Kajanachumpol S, Kanjanapongkul S, Sirachainan N, Meekaewkunchorn A, Ningsanond V, Hongeng S.
    Int J Lab Hematol; 2008 Aug 14; 30(4):286-91. PubMed ID: 18665825
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  • 18. Predisposition to therapy-related acute leukemia with balanced chromosomal translocations does not result from a major constitutive defect in DNA double-strand break end joining.
    Coiteux V, Onclercq-Delic R, Fenaux P, Amor-Guéret M.
    Leuk Res; 2007 Mar 14; 31(3):353-8. PubMed ID: 16890283
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  • 19. Inversion of chromosome 11 inv(11)(p15q22), as a recurring chromosomal aberration associated with de novo and secondary myeloid malignancies: identification of a P1 clone spanning the 11q22 breakpoint.
    Kobayashi H, Arai Y, Hosoda F, Maseki N, Hayashi Y, Eguchi H, Ohki M, Kaneko Y.
    Genes Chromosomes Cancer; 1997 Jul 14; 19(3):150-5. PubMed ID: 9218995
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  • 20. Molecular characterization of the genomic breakpoints in a case of t(3;21)(q26;q22).
    Hirai H, Ogawa S, Kurokawa M, Yazaki Y, Mitani K.
    Genes Chromosomes Cancer; 1999 Sep 14; 26(1):92-6. PubMed ID: 10441011
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