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Journal Abstract Search


382 related items for PubMed ID: 16924481

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  • 4. Mutations in the genes encoding the pancreatic beta-cell KATP channel subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) in diabetes mellitus and hyperinsulinism.
    Gloyn AL, Siddiqui J, Ellard S.
    Hum Mutat; 2006 Mar; 27(3):220-31. PubMed ID: 16416420
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  • 11. Destabilization of ATP-sensitive potassium channel activity by novel KCNJ11 mutations identified in congenital hyperinsulinism.
    Lin YW, Bushman JD, Yan FF, Haidar S, MacMullen C, Ganguly A, Stanley CA, Shyng SL.
    J Biol Chem; 2008 Apr 04; 283(14):9146-56. PubMed ID: 18250167
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  • 12. Update of mutations in the genes encoding the pancreatic beta-cell K(ATP) channel subunits Kir6.2 (KCNJ11) and sulfonylurea receptor 1 (ABCC8) in diabetes mellitus and hyperinsulinism.
    Flanagan SE, Clauin S, Bellanné-Chantelot C, de Lonlay P, Harries LW, Gloyn AL, Ellard S.
    Hum Mutat; 2009 Feb 04; 30(2):170-80. PubMed ID: 18767144
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  • 13. Regulated expression of adenosine triphosphate-sensitive potassium channel subunits in pancreatic beta-cells.
    Moritz W, Leech CA, Ferrer J, Habener JF.
    Endocrinology; 2001 Jan 04; 142(1):129-38. PubMed ID: 11145575
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  • 15. Restitution of defective glucose-stimulated insulin release of sulfonylurea type 1 receptor knockout mice by acetylcholine.
    Doliba NM, Qin W, Vatamaniuk MZ, Li C, Zelent D, Najafi H, Buettger CW, Collins HW, Carr RD, Magnuson MA, Matschinsky FM.
    Am J Physiol Endocrinol Metab; 2004 May 04; 286(5):E834-43. PubMed ID: 14736703
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  • 18. Sulfonylurea receptor 1 mutations that cause opposite insulin secretion defects with chemical chaperone exposure.
    Pratt EB, Yan FF, Gay JW, Stanley CA, Shyng SL.
    J Biol Chem; 2009 Mar 20; 284(12):7951-9. PubMed ID: 19151370
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  • 19. The structure and function of the ATP-sensitive K+ channel in insulin-secreting pancreatic beta-cells.
    Miki T, Nagashima K, Seino S.
    J Mol Endocrinol; 1999 Apr 20; 22(2):113-23. PubMed ID: 10194514
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  • 20. Clinical and molecular characterization of a dominant form of congenital hyperinsulinism caused by a mutation in the high-affinity sulfonylurea receptor.
    Thornton PS, MacMullen C, Ganguly A, Ruchelli E, Steinkrauss L, Crane A, Aguilar-Bryan L, Stanley CA.
    Diabetes; 2003 Sep 20; 52(9):2403-10. PubMed ID: 12941782
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