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Journal Abstract Search

159 related items for PubMed ID: 16940421

  • 1. Selective leukemic-cell killing by a novel functional class of thalidomide analogs.
    Ge Y, Montano I, Rustici G, Freebern WJ, Haggerty CM, Cui W, Ponciano-Jackson D, Chandramouli GV, Gardner ER, Figg WD, Abu-Asab M, Tsokos M, Jackson SH, Gardner K.
    Blood; 2006 Dec 15; 108(13):4126-35. PubMed ID: 16940421
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  • 2. Combinatorial antileukemic disruption of oxidative homeostasis and mitochondrial stability by the redox reactive thalidomide 2-(2,4-difluoro-phenyl)-4,5,6,7-tetrafluoro-1H-isoindole-1,3(2H)-dione (CPS49) and flavopiridol.
    Ge Y, Byun JS, De Luca P, Gueron G, Yabe IM, Sadiq-Ali SG, Figg WD, Quintero J, Haggerty CM, Li QQ, De Siervi A, Gardner K.
    Mol Pharmacol; 2008 Sep 15; 74(3):872-83. PubMed ID: 18556456
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  • 5. Human regulatory T cells rapidly suppress T cell receptor-induced Ca(2+), NF-κB, and NFAT signaling in conventional T cells.
    Schmidt A, Oberle N, Weiss EM, Vobis D, Frischbutter S, Baumgrass R, Falk CS, Haag M, Brügger B, Lin H, Mayr GW, Reichardt P, Gunzer M, Suri-Payer E, Krammer PH.
    Sci Signal; 2011 Dec 20; 4(204):ra90. PubMed ID: 22375050
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  • 10. Targeting Oncogenic Nuclear Factor Kappa B Signaling with Redox-Active Agents for Cancer Treatment.
    Fouani L, Kovacevic Z, Richardson DR.
    Antioxid Redox Signal; 2019 Mar 10; 30(8):1096-1123. PubMed ID: 29161883
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  • 11. Activation of NF-κB and respiratory burst following Aspergillus fumigatus stimulation of macrophages.
    Sun H, Xu XY, Tian XL, Shao HT, Wu XD, Wang Q, Su X, Shi Y.
    Immunobiology; 2014 Jan 10; 219(1):25-36. PubMed ID: 23886693
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  • 12. Thiol dependent NF-κB suppression and inhibition of T-cell mediated adaptive immune responses by a naturally occurring steroidal lactone Withaferin A.
    Gambhir L, Checker R, Sharma D, Thoh M, Patil A, Degani M, Gota V, Sandur SK.
    Toxicol Appl Pharmacol; 2015 Dec 01; 289(2):297-312. PubMed ID: 26408225
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  • 13. Basiliolides, a class of tetracyclic C19 dilactones from Thapsia garganica, release Ca(2+) from the endoplasmic reticulum and regulate the activity of the transcription factors nuclear factor of activated T cells, nuclear factor-kappaB, and activator protein 1 in T lymphocytes.
    Navarrete C, Sancho R, Caballero FJ, Pollastro F, Fiebich BL, Sterner O, Appendino G, Muñoz E.
    J Pharmacol Exp Ther; 2006 Oct 01; 319(1):422-30. PubMed ID: 16840645
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  • 15. Endoplasmic reticulum stress-mediated apoptosis of EBV-transformed B cells by cross-linking of CD70 is dependent upon generation of reactive oxygen species and activation of p38 MAPK and JNK pathway.
    Park GB, Kim YS, Lee HK, Song H, Cho DH, Lee WJ, Hur DY.
    J Immunol; 2010 Dec 15; 185(12):7274-84. PubMed ID: 21078900
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  • 16. Dual role of mitochondrial reactive oxygen species in hypoxia signaling: activation of nuclear factor-{kappa}B via c-SRC and oxidant-dependent cell death.
    Lluis JM, Buricchi F, Chiarugi P, Morales A, Fernandez-Checa JC.
    Cancer Res; 2007 Aug 01; 67(15):7368-77. PubMed ID: 17671207
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  • 17. Protective role of a coumarin-derived schiff base scaffold against tertiary butyl hydroperoxide (TBHP)-induced oxidative impairment and cell death via MAPKs, NF-κB and mitochondria-dependent pathways.
    Ghosh M, Manna P, Sil PC.
    Free Radic Res; 2011 May 01; 45(5):620-37. PubMed ID: 21391895
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  • 18. Immunosuppressive activity of endovanilloids: N-arachidonoyl-dopamine inhibits activation of the NF-kappa B, NFAT, and activator protein 1 signaling pathways.
    Sancho R, Macho A, de La Vega L, Calzado MA, Fiebich BL, Appendino G, Muñoz E.
    J Immunol; 2004 Feb 15; 172(4):2341-51. PubMed ID: 14764703
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  • 19. Organometallic nucleosides induce non-classical leukemic cell death that is mitochondrial-ROS dependent and facilitated by TCL1-oncogene burden.
    Prinz C, Vasyutina E, Lohmann G, Schrader A, Romanski S, Hirschhäuser C, Mayer P, Frias C, Herling CD, Hallek M, Schmalz HG, Prokop A, Mougiakakos D, Herling M.
    Mol Cancer; 2015 Jun 04; 14():114. PubMed ID: 26041471
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  • 20. Helenalin suppresses essential immune functions of activated CD4+ T cells by multiple mechanisms.
    Berges C, Fuchs D, Opelz G, Daniel V, Naujokat C.
    Mol Immunol; 2009 Sep 04; 46(15):2892-901. PubMed ID: 19656571
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