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396 related items for PubMed ID: 17015480

  • 1. TReP-132 is a novel progesterone receptor coactivator required for the inhibition of breast cancer cell growth and enhancement of differentiation by progesterone.
    Gizard F, Robillard R, Gross B, Barbier O, Révillion F, Peyrat JP, Torpier G, Hum DW, Staels B.
    Mol Cell Biol; 2006 Oct; 26(20):7632-44. PubMed ID: 17015480
    [Abstract] [Full Text] [Related]

  • 2. TReP-132 controls cell proliferation by regulating the expression of the cyclin-dependent kinase inhibitors p21WAF1/Cip1 and p27Kip1.
    Gizard F, Robillard R, Barbier O, Quatannens B, Faucompré A, Révillion F, Peyrat JP, Staels B, Hum DW.
    Mol Cell Biol; 2005 Jun; 25(11):4335-48. PubMed ID: 15899840
    [Abstract] [Full Text] [Related]

  • 3. Integrity of the LXXLL motif in Stat6 is required for the inhibition of breast cancer cell growth and enhancement of differentiation in the context of progesterone.
    Wei M, He Q, Yang Z, Wang Z, Zhang Q, Liu B, Gu Q, Su L, Yu Y, Zhu Z, Zhang G.
    BMC Cancer; 2014 Jan 08; 14():10. PubMed ID: 24401087
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  • 5. The antiprogestin Lonaprisan inhibits breast cancer cell proliferation by inducing p21 expression.
    Busia L, Faus H, Hoffmann J, Haendler B.
    Mol Cell Endocrinol; 2011 Feb 10; 333(1):37-46. PubMed ID: 21138753
    [Abstract] [Full Text] [Related]

  • 6. Novel signaling molecules implicated in tumor-associated fatty acid synthase-dependent breast cancer cell proliferation and survival: Role of exogenous dietary fatty acids, p53-p21WAF1/CIP1, ERK1/2 MAPK, p27KIP1, BRCA1, and NF-kappaB.
    Menendez JA, Mehmi I, Atlas E, Colomer R, Lupu R.
    Int J Oncol; 2004 Mar 10; 24(3):591-608. PubMed ID: 14767544
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  • 8. Activation of the p21WAF1/CIP1 promoter independent of p53 by the histone deacetylase inhibitor suberoylanilide hydroxamic acid (SAHA) through the Sp1 sites.
    Huang L, Sowa Y, Sakai T, Pardee AB.
    Oncogene; 2000 Nov 23; 19(50):5712-9. PubMed ID: 11126357
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  • 10. Reduction of human metastatic breast cancer cell aggressiveness on introduction of either form a or B of the progesterone receptor and then treatment with progestins.
    Sumida T, Itahana Y, Hamakawa H, Desprez PY.
    Cancer Res; 2004 Nov 01; 64(21):7886-92. PubMed ID: 15520195
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  • 11. Progestins reinitiate cell cycle progression in antiestrogen-arrested breast cancer cells through the B-isoform of progesterone receptor.
    McGowan EM, Russell AJ, Boonyaratanakornkit V, Saunders DN, Lehrbach GM, Sergio CM, Musgrove EA, Edwards DP, Sutherland RL.
    Cancer Res; 2007 Sep 15; 67(18):8942-51. PubMed ID: 17875737
    [Abstract] [Full Text] [Related]

  • 12. Taxol-induced growth arrest and apoptosis is associated with the upregulation of the Cdk inhibitor, p21WAF1/CIP1, in human breast cancer cells.
    Choi YH, Yoo YH.
    Oncol Rep; 2012 Dec 15; 28(6):2163-9. PubMed ID: 23023313
    [Abstract] [Full Text] [Related]

  • 13. Involvement of p21(WAF1/Cip1), p27(Kip1), and p18(INK4c) in troglitazone-induced cell-cycle arrest in human hepatoma cell lines.
    Koga H, Sakisaka S, Harada M, Takagi T, Hanada S, Taniguchi E, Kawaguchi T, Sasatomi K, Kimura R, Hashimoto O, Ueno T, Yano H, Kojiro M, Sata M.
    Hepatology; 2001 May 15; 33(5):1087-97. PubMed ID: 11343236
    [Abstract] [Full Text] [Related]

  • 14. Integration of progesterone receptor mediated rapid signaling and nuclear actions in breast cancer cell models: role of mitogen-activated protein kinases and cell cycle regulators.
    Faivre E, Skildum A, Pierson-Mullany L, Lange CA.
    Steroids; 2005 May 15; 70(5-7):418-26. PubMed ID: 15862825
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  • 15. Activation of protein kinase G Increases the expression of p21CIP1, p27KIP1, and histidine triad protein 1 through Sp1.
    Cen B, Deguchi A, Weinstein IB.
    Cancer Res; 2008 Jul 01; 68(13):5355-62. PubMed ID: 18593937
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  • 16. Retinoic acid inhibition of cell cycle progression in MCF-7 human breast cancer cells.
    Zhu WY, Jones CS, Kiss A, Matsukuma K, Amin S, De Luca LM.
    Exp Cell Res; 1997 Aug 01; 234(2):293-9. PubMed ID: 9260897
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  • 17. Progestin drives breast cancer growth by inducing p21(CIP1) expression through the assembly of a transcriptional complex among Stat3, progesterone receptor and ErbB-2.
    Diaz Flaqué MC, Vicario R, Proietti CJ, Izzo F, Schillaci R, Elizalde PV.
    Steroids; 2013 Jun 01; 78(6):559-67. PubMed ID: 23178160
    [Abstract] [Full Text] [Related]

  • 18. Tamoxifen-induced activation of p21Waf1/Cip1 gene transcription is mediated by Early Growth Response-1 protein through the JNK and p38 MAP kinase/Elk-1 cascades in MDA-MB-361 breast carcinoma cells.
    Kim CG, Choi BH, Son SW, Yi SJ, Shin SY, Lee YH.
    Cell Signal; 2007 Jun 01; 19(6):1290-300. PubMed ID: 17307334
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  • 19. The orphan receptor COUP-TFII regulates G2/M progression of breast cancer cells by modulating the expression/activity of p21(WAF1/CIP1), cyclin D1, and cdk2.
    Nakshatri H, Mendonca MS, Bhat-Nakshatri P, Patel NM, Goulet RJ, Cornetta K.
    Biochem Biophys Res Commun; 2000 Apr 21; 270(3):1144-53. PubMed ID: 10772965
    [Abstract] [Full Text] [Related]

  • 20. Histone deacetylase inhibitors activate INK4d gene through Sp1 site in its promoter.
    Yokota T, Matsuzaki Y, Miyazawa K, Zindy F, Roussel MF, Sakai T.
    Oncogene; 2004 Jul 08; 23(31):5340-9. PubMed ID: 15107822
    [Abstract] [Full Text] [Related]

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