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Journal Abstract Search

98 related items for PubMed ID: 17020133

  • 1. [Influence of stress on the activation of CaMKII in the brain].
    Suenaga T, Morinobu S, Yamawaki S.
    Nihon Shinkei Seishin Yakurigaku Zasshi; 2006 Aug; 26(4):169-75. PubMed ID: 17020133
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  • 2. Activation of calcium/calmodulin-dependent protein kinases after traumatic brain injury.
    Atkins CM, Chen S, Alonso OF, Dietrich WD, Hu BR.
    J Cereb Blood Flow Metab; 2006 Dec; 26(12):1507-18. PubMed ID: 16570077
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  • 3. Reduced basal CaMKII levels in hippocampal CA1 region: possible cause of stress-induced impairment of LTP in chronically stressed rats.
    Gerges NZ, Aleisa AM, Schwarz LA, Alkadhi KA.
    Hippocampus; 2004 Dec; 14(3):402-10. PubMed ID: 15132438
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  • 7. Propofol and ketamine-induced anesthetic depth-dependent decrease of CaMKII phosphorylation levels in rat hippocampus and cortex.
    Cui X, Li J, Li T, Ji F, Bu X, Zhang N, Zhang B.
    J Neurosurg Anesthesiol; 2009 Apr; 21(2):145-54. PubMed ID: 19295394
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  • 9. Phosphorylation of Ca2+/calmodulin-dependent protein kinase type ii and the alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionate (ampa) receptor in response to a threonine-devoid diet.
    Sharp JW, Ross CM, Koehnle TJ, Gietzen DW.
    Neuroscience; 2004 Apr; 126(4):1053-62. PubMed ID: 15207338
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  • 10. Brief seizure activity alters Ca2+/calmodulin dependent protein kinase II dephosphorylation and subcellular distribution in rat brain for several hours.
    Dong Y, Rosenberg HC.
    Neurosci Lett; 2004 Mar 04; 357(2):95-8. PubMed ID: 15036583
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  • 11. A mechanism for the inactivation of Ca2+/calmodulin-dependent protein kinase II during prolonged seizure activity and its consequence after the recovery from seizure activity in rats in vivo.
    Yamagata Y, Imoto K, Obata K.
    Neuroscience; 2006 Jul 07; 140(3):981-92. PubMed ID: 16632208
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  • 12. CaM kinase II and protein kinase C activations mediate enhancement of long-term potentiation by nefiracetam in the rat hippocampal CA1 region.
    Moriguchi S, Shioda N, Han F, Narahashi T, Fukunaga K.
    J Neurochem; 2008 Aug 07; 106(3):1092-103. PubMed ID: 18445137
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  • 13. Ca2+/calmodulin-dependent protein kinase II is reversibly autophosphorylated, inactivated and made sedimentable by acute neuronal excitation in rats in vivo.
    Yamagata Y, Obata K.
    J Neurochem; 2004 Nov 07; 91(3):745-54. PubMed ID: 15485503
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  • 14. Prolonged changes in Ca2+/calmodulin-dependent protein kinase II after a brief pentylenetetrazol seizure; potential role in kindling.
    Dong Y, Rosenberg HC.
    Epilepsy Res; 2004 Feb 07; 58(2-3):107-17. PubMed ID: 15120742
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  • 16. The role of regulatory domain interactions in UNC-43 CaMKII localization and trafficking.
    Umemura T, Rapp P, Rongo C.
    J Cell Sci; 2005 Aug 01; 118(Pt 15):3327-38. PubMed ID: 16079277
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  • 17. [Molecular mechanisms for memory formation].
    Manabe T.
    Brain Nerve; 2008 Jul 01; 60(7):707-15. PubMed ID: 18646610
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  • 18. Prenatal stress alters glutamatergic system responsiveness in adult rat prefrontal cortex.
    Fumagalli F, Pasini M, Frasca A, Drago F, Racagni G, Riva MA.
    J Neurochem; 2009 Jun 01; 109(6):1733-44. PubMed ID: 19383086
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  • 19. Calcium-calmodulin-dependent protein kinase II phosphorylation modulates PSD-95 binding to NMDA receptors.
    Gardoni F, Polli F, Cattabeni F, Di Luca M.
    Eur J Neurosci; 2006 Nov 01; 24(10):2694-704. PubMed ID: 17156196
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  • 20. [Effect of AVP(4-8) administration on Ca2+/CaM-dependent protein kinase II autophosphorylation in rat brain].
    Qiao LY, Chen XF, Gu BX, Wang TX, Du YC.
    Sheng Li Xue Bao; 1998 Apr 01; 50(2):132-8. PubMed ID: 11324526
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