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210 related items for PubMed ID: 17077285

  • 1. Glucocorticoid resistance in two key models of acute lymphoblastic leukemia occurs at the level of the glucocorticoid receptor.
    Schmidt S, Irving JA, Minto L, Matheson E, Nicholson L, Ploner A, Parson W, Kofler A, Amort M, Erdel M, Hall A, Kofler R.
    FASEB J; 2006 Dec; 20(14):2600-2. PubMed ID: 17077285
    [Abstract] [Full Text] [Related]

  • 2. Glucocorticoid receptor heterozygosity combined with lack of receptor auto-induction causes glucocorticoid resistance in Jurkat acute lymphoblastic leukemia cells.
    Riml S, Schmidt S, Ausserlechner MJ, Geley S, Kofler R.
    Cell Death Differ; 2004 Jul; 11 Suppl 1():S65-72. PubMed ID: 15017388
    [Abstract] [Full Text] [Related]

  • 3. Glucocorticoid-induced glucocorticoid-receptor expression and promoter usage is not linked to glucocorticoid resistance in childhood ALL.
    Tissing WJ, Meijerink JP, Brinkhof B, Broekhuis MJ, Menezes RX, den Boer ML, Pieters R.
    Blood; 2006 Aug 01; 108(3):1045-9. PubMed ID: 16574952
    [Abstract] [Full Text] [Related]

  • 4. Glucocorticoid-induced apoptosis and glucocorticoid resistance in acute lymphoblastic leukemia.
    Ploner C, Schmidt S, Presul E, Renner K, Schröcksnadel K, Rainer J, Riml S, Kofler R.
    J Steroid Biochem Mol Biol; 2005 Feb 01; 93(2-5):153-60. PubMed ID: 15860257
    [Abstract] [Full Text] [Related]

  • 5. NFkappaB modulators in a model of glucocorticoid resistant, childhood acute lymphoblastic leukemia.
    Nicholson L, Hall AG, Redfern CP, Irving J.
    Leuk Res; 2010 Oct 01; 34(10):1366-73. PubMed ID: 20106524
    [Abstract] [Full Text] [Related]

  • 6. Resistance to glucocorticoid-induced apoptosis in human T-cell acute lymphoblastic leukemia CEM-C1 cells is due to insufficient glucocorticoid receptor expression.
    Geley S, Hartmann BL, Hala M, Strasser-Wozak EM, Kapelari K, Kofler R.
    Cancer Res; 1996 Nov 01; 56(21):5033-8. PubMed ID: 8895760
    [Abstract] [Full Text] [Related]

  • 7. BTG1 regulates glucocorticoid receptor autoinduction in acute lymphoblastic leukemia.
    van Galen JC, Kuiper RP, van Emst L, Levers M, Tijchon E, Scheijen B, Waanders E, van Reijmersdal SV, Gilissen C, van Kessel AG, Hoogerbrugge PM, van Leeuwen FN.
    Blood; 2010 Jun 10; 115(23):4810-9. PubMed ID: 20354172
    [Abstract] [Full Text] [Related]

  • 8. Rapamycin sensitizes glucocorticoid resistant acute lymphoblastic leukemia CEM-C1 cells to dexamethasone induced apoptosis through both mTOR suppression and up-regulation and activation of glucocorticoid receptor.
    Guo X, Zhou CY, Li Q, Gao J, Zhu YP, Gu L, Ma ZG.
    Biomed Environ Sci; 2013 May 10; 26(5):371-81. PubMed ID: 23611130
    [Abstract] [Full Text] [Related]

  • 9. Use of recombinant cell-permeable small peptides to modulate glucocorticoid sensitivity of acute lymphoblastic leukemia cells.
    Geng CD, Vedeckis WV.
    Biochemistry; 2010 Oct 19; 49(41):8892-901. PubMed ID: 20831260
    [Abstract] [Full Text] [Related]

  • 10. Resistance to glucocorticoid-induced apoptosis in lymphoblastic leukemia.
    Kofler R, Schmidt S, Kofler A, Ausserlechner MJ.
    J Endocrinol; 2003 Jul 19; 178(1):19-27. PubMed ID: 12844332
    [Abstract] [Full Text] [Related]

  • 11. Receptor mutation is not a common mechanism of naturally occurring glucocorticoid resistance in leukaemia cell lines.
    Beesley AH, Weller RE, Senanayake S, Welch M, Kees UR.
    Leuk Res; 2009 Feb 19; 33(2):321-5. PubMed ID: 18789525
    [Abstract] [Full Text] [Related]

  • 12. Genetic variations in the glucocorticoid receptor gene are not related to glucocorticoid resistance in childhood acute lymphoblastic leukemia.
    Tissing WJ, Meijerink JP, den Boer ML, Brinkhof B, van Rossum EF, van Wering ER, Koper JW, Sonneveld P, Pieters R.
    Clin Cancer Res; 2005 Aug 15; 11(16):6050-6. PubMed ID: 16115950
    [Abstract] [Full Text] [Related]

  • 13. Resistance of human leukemic CEM-C1 cells is overcome by synergism between glucocorticoid and protein kinase A pathways: correlation with c-Myc suppression.
    Medh RD, Saeed MF, Johnson BH, Thompson EB.
    Cancer Res; 1998 Aug 15; 58(16):3684-93. PubMed ID: 9721879
    [Abstract] [Full Text] [Related]

  • 14. The effects of microRNAs on glucocorticoid responsiveness.
    Wang H, Gou X, Jiang T, Ouyang J.
    J Cancer Res Clin Oncol; 2017 Jun 15; 143(6):1005-1011. PubMed ID: 28286901
    [Abstract] [Full Text] [Related]

  • 15. A facile, branched DNA assay to quantitatively measure glucocorticoid receptor auto-regulation in T-cell acute lymphoblastic leukemia.
    Schwartz JR, Sarvaiya PJ, Leiva LE, Velez MC, Singleton TC, Yu LC, Vedeckis WV.
    Chin J Cancer; 2012 Aug 15; 31(8):381-91. PubMed ID: 22739263
    [Abstract] [Full Text] [Related]

  • 16. Erg and AP-1 as determinants of glucocorticoid response in acute lymphoblastic leukemia.
    Chen DW, Saha V, Liu JZ, Schwartz JM, Krstic-Demonacos M.
    Oncogene; 2013 Jun 20; 32(25):3039-48. PubMed ID: 22869147
    [Abstract] [Full Text] [Related]

  • 17. Protein expression of the glucocorticoid receptor in childhood acute lymphoblastic leukemia.
    Lauten M, Cario G, Asgedom G, Welte K, Schrappe M.
    Haematologica; 2003 Nov 20; 88(11):1253-8. PubMed ID: 14607754
    [Abstract] [Full Text] [Related]

  • 18. Quantitative analysis and modeling of glucocorticoid-controlled gene expression.
    Chen DW, Lynch JT, Demonacos C, Krstic-Demonacos M, Schwartz JM.
    Pharmacogenomics; 2010 Nov 20; 11(11):1545-60. PubMed ID: 21121775
    [Abstract] [Full Text] [Related]

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  • 20. Role for microRNAs in regulating glucocorticoid response and resistance in multiple myeloma.
    Tessel MA, Benham AL, Krett NL, Rosen ST, Gunaratne PH.
    Horm Cancer; 2011 Jun 20; 2(3):182-9. PubMed ID: 21761344
    [Abstract] [Full Text] [Related]

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