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221 related items for PubMed ID: 17098995

  • 1. Synergistic and strain-specific effects of bovine spongiform encephalopathy and scrapie prions in the cell-free conversion of recombinant prion protein.
    Eiden M, Palm GJ, Hinrichs W, Matthey U, Zahn R, Groschup MH.
    J Gen Virol; 2006 Dec; 87(Pt 12):3753-3761. PubMed ID: 17098995
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  • 3. Differences in proteinase K resistance and neuronal deposition of abnormal prion proteins characterize bovine spongiform encephalopathy (BSE) and scrapie strains.
    Kuczius T, Groschup MH.
    Mol Med; 1999 Jun; 5(6):406-18. PubMed ID: 10415165
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  • 4. Strain-Dependent Prion Infection in Mice Expressing Prion Protein with Deletion of Central Residues 91-106.
    Uchiyama K, Miyata H, Yamaguchi Y, Imamura M, Okazaki M, Pasiana AD, Chida J, Hara H, Atarashi R, Watanabe H, Kondoh G, Sakaguchi S.
    Int J Mol Sci; 2020 Oct 01; 21(19):. PubMed ID: 33019549
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  • 7. Amino acid sequence and prion strain specific effects on the in vitro and in vivo convertibility of ovine/murine and bovine/murine prion protein chimeras.
    Kupfer L, Eiden M, Buschmann A, Groschup MH.
    Biochim Biophys Acta; 2007 Jun 01; 1772(6):704-13. PubMed ID: 17145171
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  • 12. Strain-specific propagation of PrP(Sc) properties into baculovirus-expressed hamster PrP(C).
    Iniguez V, McKenzie D, Mirwald J, Aiken J.
    J Gen Virol; 2000 Oct 01; 81(Pt 10):2565-2571. PubMed ID: 10993947
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  • 14. Propagation of RML prions in mice expressing PrP devoid of GPI anchor leads to formation of a novel, stable prion strain.
    Mahal SP, Jablonski J, Suponitsky-Kroyter I, Oelschlegel AM, Herva ME, Oldstone M, Weissmann C.
    PLoS Pathog; 2012 Oct 01; 8(6):e1002746. PubMed ID: 22685404
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  • 15. High-resolution differentiation of transmissible spongiform encephalopathy strains by quantitative N-terminal amino acid profiling (N-TAAP) of PK-digested abnormal prion protein.
    Gielbert A, Davis LA, Sayers AR, Hope J, Gill AC, Sauer MJ.
    J Mass Spectrom; 2009 Mar 01; 44(3):384-96. PubMed ID: 19053160
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  • 16. Molecular analysis of the abnormal prion protein during coinfection of mice by bovine spongiform encephalopathy and a scrapie agent.
    Baron TG, Biacabe AG.
    J Virol; 2001 Jan 01; 75(1):107-14. PubMed ID: 11119579
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  • 17. PrP-C1 fragment in cattle brains reveals features of the transmissible spongiform encephalopathy associated PrPsc.
    Serra F, Müller J, Gray J, Lüthi R, Dudas S, Czub S, Seuberlich T.
    Brain Res; 2017 Mar 15; 1659():19-28. PubMed ID: 28119056
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  • 18. Molecular analysis of the protease-resistant prion protein in scrapie and bovine spongiform encephalopathy transmitted to ovine transgenic and wild-type mice.
    Baron T, Crozet C, Biacabe AG, Philippe S, Verchere J, Bencsik A, Madec JY, Calavas D, Samarut J.
    J Virol; 2004 Jun 15; 78(12):6243-51. PubMed ID: 15163717
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  • 19. Experimental transmission of two young and one suspended bovine spongiform encephalopathy (BSE) cases to bovinized transgenic mice.
    Yokoyama T, Masujin K, Yamakawa Y, Sata T, Murayama Y, Shu Y, Okada H, Mohri S, Shinagawa M.
    Jpn J Infect Dis; 2007 Sep 15; 60(5):317-20. PubMed ID: 17881878
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  • 20. Specific binding of normal prion protein to the scrapie form via a localized domain initiates its conversion to the protease-resistant state.
    Horiuchi M, Caughey B.
    EMBO J; 1999 Jun 15; 18(12):3193-203. PubMed ID: 10369660
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