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Journal Abstract Search

223 related items for PubMed ID: 17116693

  • 1. Helix-loop-helix protein p8, a transcriptional regulator required for cardiomyocyte hypertrophy and cardiac fibroblast matrix metalloprotease induction.
    Goruppi S, Patten RD, Force T, Kyriakis JM.
    Mol Cell Biol; 2007 Feb; 27(3):993-1006. PubMed ID: 17116693
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  • 2. IL-17 stimulates MMP-1 expression in primary human cardiac fibroblasts via p38 MAPK- and ERK1/2-dependent C/EBP-beta , NF-kappaB, and AP-1 activation.
    Cortez DM, Feldman MD, Mummidi S, Valente AJ, Steffensen B, Vincenti M, Barnes JL, Chandrasekar B.
    Am J Physiol Heart Circ Physiol; 2007 Dec; 293(6):H3356-65. PubMed ID: 17921324
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  • 3. Decreased metalloprotease 9 induction, cardiac fibrosis, and higher autophagy after pressure overload in mice lacking the transcriptional regulator p8.
    Georgescu SP, Aronovitz MJ, Iovanna JL, Patten RD, Kyriakis JM, Goruppi S.
    Am J Physiol Cell Physiol; 2011 Nov; 301(5):C1046-56. PubMed ID: 21775709
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  • 4. JunD attenuates phenylephrine-mediated cardiomyocyte hypertrophy by negatively regulating AP-1 transcriptional activity.
    Hilfiker-Kleiner D, Hilfiker A, Castellazzi M, Wollert KC, Trautwein C, Schunkert H, Drexler H.
    Cardiovasc Res; 2006 Jul 01; 71(1):108-17. PubMed ID: 16690042
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  • 5. The pro-hypertrophic basic helix-loop-helix protein p8 is degraded by the ubiquitin/proteasome system in a protein kinase B/Akt- and glycogen synthase kinase-3-dependent manner, whereas endothelin induction of p8 mRNA and renal mesangial cell hypertrophy require NFAT4.
    Goruppi S, Kyriakis JM.
    J Biol Chem; 2004 May 14; 279(20):20950-8. PubMed ID: 15016802
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  • 6. Deficiency of the transcriptional regulator p8 results in increased autophagy and apoptosis, and causes impaired heart function.
    Kong DK, Georgescu SP, Cano C, Aronovitz MJ, Iovanna JL, Patten RD, Kyriakis JM, Goruppi S.
    Mol Biol Cell; 2010 Apr 15; 21(8):1335-49. PubMed ID: 20181828
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  • 7. Dominant negative mutant of c-Jun inhibits cardiomyocyte hypertrophy induced by endothelin 1 and phenylephrine.
    Omura T, Yoshiyama M, Yoshida K, Nakamura Y, Kim S, Iwao H, Takeuchi K, Yoshikawa J.
    Hypertension; 2002 Jan 15; 39(1):81-6. PubMed ID: 11799083
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  • 11. Load-induced transcriptional activation of c-jun in rat myocardium: regulation by myocyte enhancer factor 2.
    Nadruz W, Kobarg CB, Constancio SS, Corat PD, Franchini KG.
    Circ Res; 2003 Feb 07; 92(2):243-51. PubMed ID: 12574153
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  • 13. Adenoviral-directed expression of the type 1A angiotensin receptor promotes cardiomyocyte hypertrophy via transactivation of the epidermal growth factor receptor.
    Thomas WG, Brandenburger Y, Autelitano DJ, Pham T, Qian H, Hannan RD.
    Circ Res; 2002 Feb 08; 90(2):135-42. PubMed ID: 11834705
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  • 14. COX-2 is involved in ET-1-induced hypertrophy of neonatal rat cardiomyocytes: role of NFATc3.
    Li H, Gao S, Ye J, Feng X, Cai Y, Liu Z, Lu J, Li Q, Huang X, Chen S, Liu P.
    Mol Cell Endocrinol; 2014 Feb 15; 382(2):998-1006. PubMed ID: 24291639
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  • 16. The role of endothelin-converting enzyme-1 in the development of alpha1-adrenergic-stimulated hypertrophy in cultured neonatal rat cardiac myocytes.
    Kaburagi S, Hasegawa K, Morimoto T, Araki M, Sawamura T, Masaki T, Sasayama S.
    Circulation; 1999 Jan 19; 99(2):292-8. PubMed ID: 9892597
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  • 17. Multiprotein bridging factor 1 cooperates with c-Jun and is necessary for cardiac hypertrophy in vitro.
    Busk PK, Wulf-Andersen L, Strøm CC, Enevoldsen M, Thirstrup K, Haunsø S, Sheikh SP.
    Exp Cell Res; 2003 May 15; 286(1):102-14. PubMed ID: 12729799
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