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PUBMED FOR HANDHELDS

Journal Abstract Search


221 related items for PubMed ID: 17229845

  • 1. FSTL3 deletion reveals roles for TGF-beta family ligands in glucose and fat homeostasis in adults.
    Mukherjee A, Sidis Y, Mahan A, Raher MJ, Xia Y, Rosen ED, Bloch KD, Thomas MK, Schneyer AL.
    Proc Natl Acad Sci U S A; 2007 Jan 23; 104(4):1348-53. PubMed ID: 17229845
    [Abstract] [Full Text] [Related]

  • 2. Follistatin and follistatin like-3 differentially regulate adiposity and glucose homeostasis.
    Brown ML, Bonomi L, Ungerleider N, Zina J, Kimura F, Mukherjee A, Sidis Y, Schneyer A.
    Obesity (Silver Spring); 2011 Oct 23; 19(10):1940-9. PubMed ID: 21546932
    [Abstract] [Full Text] [Related]

  • 3. Over-expression of Follistatin-like 3 attenuates fat accumulation and improves insulin sensitivity in mice.
    Brandt C, Hansen RH, Hansen JB, Olsen CH, Galle P, Mandrup-Poulsen T, Gehl J, Pedersen BK, Hojman P.
    Metabolism; 2015 Feb 23; 64(2):283-95. PubMed ID: 25456456
    [Abstract] [Full Text] [Related]

  • 4. Differential synthesis and action of TGFß superfamily ligands in mouse and rat islets.
    Brown ML, Kimura F, Bonomi LM, Ungerleider NA, Schneyer AL.
    Islets; 2011 Feb 23; 3(6):367-75. PubMed ID: 21964310
    [Abstract] [Full Text] [Related]

  • 5. Biological activity of follistatin isoforms and follistatin-like-3 is dependent on differential cell surface binding and specificity for activin, myostatin, and bone morphogenetic proteins.
    Sidis Y, Mukherjee A, Keutmann H, Delbaere A, Sadatsuki M, Schneyer A.
    Endocrinology; 2006 Jul 23; 147(7):3586-97. PubMed ID: 16627583
    [Abstract] [Full Text] [Related]

  • 6. Activin Enhances α- to β-Cell Transdifferentiation as a Source For β-Cells In Male FSTL3 Knockout Mice.
    Brown ML, Andrzejewski D, Burnside A, Schneyer AL.
    Endocrinology; 2016 Mar 23; 157(3):1043-54. PubMed ID: 26727106
    [Abstract] [Full Text] [Related]

  • 7. FSTL3-Neutralizing Antibodies Enhance Glucose-Responsive Insulin Secretion in Dysfunctional Male Mouse and Human Islets.
    Brown ML, Lopez A, Meyer N, Richter A, Thompson TB.
    Endocrinology; 2021 Oct 01; 162(10):. PubMed ID: 33539535
    [Abstract] [Full Text] [Related]

  • 8. Synexpression group analyses identify new functions of FSTL3, a TGFβ ligand inhibitor.
    Robertson RD, Mukherjee A.
    Biochem Biophys Res Commun; 2012 Oct 26; 427(3):568-73. PubMed ID: 23022195
    [Abstract] [Full Text] [Related]

  • 9. Activin B regulates islet composition and islet mass but not whole body glucose homeostasis or insulin sensitivity.
    Bonomi L, Brown M, Ungerleider N, Muse M, Matzuk MM, Schneyer A.
    Am J Physiol Endocrinol Metab; 2012 Sep 01; 303(5):E587-96. PubMed ID: 22739106
    [Abstract] [Full Text] [Related]

  • 10. Follistatin-like 3 (FSTL3) mediated silencing of transforming growth factor β (TGFβ) signaling is essential for testicular aging and regulating testis size.
    Oldknow KJ, Seebacher J, Goswami T, Villen J, Pitsillides AA, O'Shaughnessy PJ, Gygi SP, Schneyer AL, Mukherjee A.
    Endocrinology; 2013 Mar 01; 154(3):1310-20. PubMed ID: 23407452
    [Abstract] [Full Text] [Related]

  • 11. Overexpression of follistatin-like 3 in gonads causes defects in gonadal development and function in transgenic mice.
    Xia Y, Sidis Y, Schneyer A.
    Mol Endocrinol; 2004 Apr 01; 18(4):979-94. PubMed ID: 14739256
    [Abstract] [Full Text] [Related]

  • 12. Follistatin-derived peptide expression in muscle decreases adipose tissue mass and prevents hepatic steatosis.
    Nakatani M, Kokubo M, Ohsawa Y, Sunada Y, Tsuchida K.
    Am J Physiol Endocrinol Metab; 2011 Mar 01; 300(3):E543-53. PubMed ID: 21205933
    [Abstract] [Full Text] [Related]

  • 13. Activins A and B Regulate Fate-Determining Gene Expression in Islet Cell Lines and Islet Cells From Male Mice.
    Andrzejewski D, Brown ML, Ungerleider N, Burnside A, Schneyer AL.
    Endocrinology; 2015 Jul 01; 156(7):2440-50. PubMed ID: 25961841
    [Abstract] [Full Text] [Related]

  • 14. Activin A and follistatin-like 3 determine the susceptibility of heart to ischemic injury.
    Oshima Y, Ouchi N, Shimano M, Pimentel DR, Papanicolaou KN, Panse KD, Tsuchida K, Lara-Pezzi E, Lee SJ, Walsh K.
    Circulation; 2009 Oct 20; 120(16):1606-15. PubMed ID: 19805648
    [Abstract] [Full Text] [Related]

  • 15. Alterations of maternal serum and placental follistatin-like 3 and myostatin in pre-eclampsia.
    Guo J, Tian T, Lu D, Xia G, Wang H, Dong M.
    J Obstet Gynaecol Res; 2012 Jul 20; 38(7):988-96. PubMed ID: 22568578
    [Abstract] [Full Text] [Related]

  • 16. Differential actions of follistatin and follistatin-like 3.
    Schneyer A, Sidis Y, Xia Y, Saito S, del Re E, Lin HY, Keutmann H.
    Mol Cell Endocrinol; 2004 Oct 15; 225(1-2):25-8. PubMed ID: 15451564
    [Abstract] [Full Text] [Related]

  • 17. Structure of myostatin·follistatin-like 3: N-terminal domains of follistatin-type molecules exhibit alternate modes of binding.
    Cash JN, Angerman EB, Kattamuri C, Nolan K, Zhao H, Sidis Y, Keutmann HT, Thompson TB.
    J Biol Chem; 2012 Jan 06; 287(2):1043-53. PubMed ID: 22052913
    [Abstract] [Full Text] [Related]

  • 18. Increased activin bioavailability enhances hepatic insulin sensitivity while inducing hepatic steatosis in male mice.
    Ungerleider NA, Bonomi LM, Brown ML, Schneyer AL.
    Endocrinology; 2013 Jun 06; 154(6):2025-33. PubMed ID: 23533219
    [Abstract] [Full Text] [Related]

  • 19. Heparin and activin-binding determinants in follistatin and FSTL3.
    Sidis Y, Schneyer AL, Keutmann HT.
    Endocrinology; 2005 Jan 06; 146(1):130-6. PubMed ID: 15471966
    [Abstract] [Full Text] [Related]

  • 20. Characterization of follistatin-type domains and their contribution to myostatin and activin A antagonism.
    Cash JN, Angerman EB, Keutmann HT, Thompson TB.
    Mol Endocrinol; 2012 Jul 06; 26(7):1167-78. PubMed ID: 22593183
    [Abstract] [Full Text] [Related]


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