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Journal Abstract Search

289 related items for PubMed ID: 17259403

  • 1. Type 2 diabetes-associated missense polymorphisms KCNJ11 E23K and ABCC8 A1369S influence progression to diabetes and response to interventions in the Diabetes Prevention Program.
    Florez JC, Jablonski KA, Kahn SE, Franks PW, Dabelea D, Hamman RF, Knowler WC, Nathan DM, Altshuler D.
    Diabetes; 2007 Feb; 56(2):531-6. PubMed ID: 17259403
    [Abstract] [Full Text] [Related]

  • 2. Haplotype structure and genotype-phenotype correlations of the sulfonylurea receptor and the islet ATP-sensitive potassium channel gene region.
    Florez JC, Burtt N, de Bakker PI, Almgren P, Tuomi T, Holmkvist J, Gaudet D, Hudson TJ, Schaffner SF, Daly MJ, Hirschhorn JN, Groop L, Altshuler D.
    Diabetes; 2004 May; 53(5):1360-8. PubMed ID: 15111507
    [Abstract] [Full Text] [Related]

  • 3. Large-scale association studies of variants in genes encoding the pancreatic beta-cell KATP channel subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) confirm that the KCNJ11 E23K variant is associated with type 2 diabetes.
    Gloyn AL, Weedon MN, Owen KR, Turner MJ, Knight BA, Hitman G, Walker M, Levy JC, Sampson M, Halford S, McCarthy MI, Hattersley AT, Frayling TM.
    Diabetes; 2003 Feb; 52(2):568-72. PubMed ID: 12540637
    [Abstract] [Full Text] [Related]

  • 4. Polymorphisms of the SUR1 (ABCC8) and Kir6.2 (KCNJ11) genes predict the conversion from impaired glucose tolerance to type 2 diabetes. The Finnish Diabetes Prevention Study.
    Laukkanen O, Pihlajamäki J, Lindström J, Eriksson J, Valle TT, Hämäläinen H, Ilanne-Parikka P, Keinänen-Kiukaanniemi S, Tuomilehto J, Uusitupa M, Laakso M, Finnish Diabetes Prevention Study Group.
    J Clin Endocrinol Metab; 2004 Dec; 89(12):6286-90. PubMed ID: 15579791
    [Abstract] [Full Text] [Related]

  • 5. Common variants in the ATP-sensitive K+ channel genes KCNJ11 (Kir6.2) and ABCC8 (SUR1) in relation to glucose intolerance: population-based studies and meta-analyses.
    van Dam RM, Hoebee B, Seidell JC, Schaap MM, de Bruin TW, Feskens EJ.
    Diabet Med; 2005 May; 22(5):590-8. PubMed ID: 15842514
    [Abstract] [Full Text] [Related]

  • 6. Physical activity modifies the effect of SNPs in the SLC2A2 (GLUT2) and ABCC8 (SUR1) genes on the risk of developing type 2 diabetes.
    Kilpeläinen TO, Lakka TA, Laaksonen DE, Laukkanen O, Lindström J, Eriksson JG, Valle TT, Hämäläinen H, Aunola S, Ilanne-Parikka P, Keinänen-Kiukaanniemi S, Tuomilehto J, Uusitupa M, Laakso M, Finnish Diabetes Prevention Study Group.
    Physiol Genomics; 2007 Oct 22; 31(2):264-72. PubMed ID: 17636114
    [Abstract] [Full Text] [Related]

  • 7. Effects of single nucleotide polymorphisms in K(ATP) channel genes on type 2 diabetes in a Turkish population.
    Gonen MS, Arikoglu H, Erkoc Kaya D, Ozdemir H, Ipekci SH, Arslan A, Kayis SA, Gogebakan B.
    Arch Med Res; 2012 May 22; 43(4):317-23. PubMed ID: 22704848
    [Abstract] [Full Text] [Related]

  • 8. KCNJ11, ABCC8 and TCF7L2 polymorphisms and the response to sulfonylurea treatment in patients with type 2 diabetes: a bioinformatics assessment.
    Song J, Yang Y, Mauvais-Jarvis F, Wang YP, Niu T.
    BMC Med Genet; 2017 Jun 06; 18(1):64. PubMed ID: 28587604
    [Abstract] [Full Text] [Related]

  • 9. Association of KCNJ11 and ABCC8 genetic polymorphisms with response to repaglinide in Chinese diabetic patients.
    He YY, Zhang R, Shao XY, Hu C, Wang CR, Lu JX, Bao YQ, Jia WP, Xiang KS.
    Acta Pharmacol Sin; 2008 Aug 06; 29(8):983-9. PubMed ID: 18664331
    [Abstract] [Full Text] [Related]

  • 10. Effect of genetic variants in KCNJ11, ABCC8, PPARG and HNF4A loci on the susceptibility of type 2 diabetes in Chinese Han population.
    Wang F, Han XY, Ren Q, Zhang XY, Han LC, Luo YY, Zhou XH, Ji LN.
    Chin Med J (Engl); 2009 Oct 20; 122(20):2477-82. PubMed ID: 20079163
    [Abstract] [Full Text] [Related]

  • 11. Genetic variations in the pancreatic ATP-sensitive potassium channel, beta-cell dysfunction, and susceptibility to type 2 diabetes.
    Chistiakov DA, Potapov VA, Khodirev DC, Shamkhalova MS, Shestakova MV, Nosikov VV.
    Acta Diabetol; 2009 Mar 20; 46(1):43-9. PubMed ID: 18758683
    [Abstract] [Full Text] [Related]

  • 12. SNPs in the KCNJ11-ABCC8 gene locus are associated with type 2 diabetes and blood pressure levels in the Japanese population.
    Sakamoto Y, Inoue H, Keshavarz P, Miyawaki K, Yamaguchi Y, Moritani M, Kunika K, Nakamura N, Yoshikawa T, Yasui N, Shiota H, Tanahashi T, Itakura M.
    J Hum Genet; 2007 Mar 20; 52(10):781-793. PubMed ID: 17823772
    [Abstract] [Full Text] [Related]

  • 13. Role of common sequence variants in insulin secretion in familial type 2 diabetic kindreds: the sulfonylurea receptor, glucokinase, and hepatocyte nuclear factor 1alpha genes.
    Elbein SC, Sun J, Scroggin E, Teng K, Hasstedt SJ.
    Diabetes Care; 2001 Mar 20; 24(3):472-8. PubMed ID: 11289470
    [Abstract] [Full Text] [Related]

  • 14. Prevalence of permanent neonatal diabetes in Slovakia and successful replacement of insulin with sulfonylurea therapy in KCNJ11 and ABCC8 mutation carriers.
    Stanik J, Gasperikova D, Paskova M, Barak L, Javorkova J, Jancova E, Ciljakova M, Hlava P, Michalek J, Flanagan SE, Pearson E, Hattersley AT, Ellard S, Klimes I.
    J Clin Endocrinol Metab; 2007 Apr 20; 92(4):1276-82. PubMed ID: 17213273
    [Abstract] [Full Text] [Related]

  • 15. Coexpression of the type 2 diabetes susceptibility gene variants KCNJ11 E23K and ABCC8 S1369A alter the ATP and sulfonylurea sensitivities of the ATP-sensitive K(+) channel.
    Hamming KS, Soliman D, Matemisz LC, Niazi O, Lang Y, Gloyn AL, Light PE.
    Diabetes; 2009 Oct 20; 58(10):2419-24. PubMed ID: 19587354
    [Abstract] [Full Text] [Related]

  • 16. Kir6.2 variant E23K increases ATP-sensitive K+ channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance.
    Villareal DT, Koster JC, Robertson H, Akrouh A, Miyake K, Bell GI, Patterson BW, Nichols CG, Polonsky KS.
    Diabetes; 2009 Aug 20; 58(8):1869-78. PubMed ID: 19491206
    [Abstract] [Full Text] [Related]

  • 17. KCNJ11 gene E23K variant and therapeutic response to sulfonylureas.
    Javorsky M, Klimcakova L, Schroner Z, Zidzik J, Babjakova E, Fabianova M, Kozarova M, Tkacova R, Salagovic J, Tkac I.
    Eur J Intern Med; 2012 Apr 20; 23(3):245-9. PubMed ID: 22385882
    [Abstract] [Full Text] [Related]

  • 18. The E23K variant of Kir6.2 associates with impaired post-OGTT serum insulin response and increased risk of type 2 diabetes.
    Nielsen EM, Hansen L, Carstensen B, Echwald SM, Drivsholm T, Glümer C, Thorsteinsson B, Borch-Johnsen K, Hansen T, Pedersen O.
    Diabetes; 2003 Feb 20; 52(2):573-7. PubMed ID: 12540638
    [Abstract] [Full Text] [Related]

  • 19. Genetics of type 2 diabetes mellitus and other specific types of diabetes; its role in treatment modalities.
    Kota SK, Meher LK, Jammula S, Kota SK, Modi KD.
    Diabetes Metab Syndr; 2012 Feb 20; 6(1):54-8. PubMed ID: 23014256
    [Abstract] [Full Text] [Related]

  • 20. A novel ABCC8 mutation illustrates the variability of the diabetes phenotypes associated with a single mutation.
    Klee P, Bellanné-Chantelot C, Depret G, Llano JP, Paget C, Nicolino M.
    Diabetes Metab; 2012 Apr 20; 38(2):179-82. PubMed ID: 22326206
    [Abstract] [Full Text] [Related]

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