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PUBMED FOR HANDHELDS

Journal Abstract Search


135 related items for PubMed ID: 17274524

  • 1. Molecular basis of neurodegenerative diseases.
    Rotilio J, Stella AM.
    Ital J Biochem; 2006; 55(3-4):189-93. PubMed ID: 17274524
    [No Abstract] [Full Text] [Related]

  • 2. ER stress and neurodegenerative diseases.
    Lindholm D, Wootz H, Korhonen L.
    Cell Death Differ; 2006 Mar; 13(3):385-92. PubMed ID: 16397584
    [Abstract] [Full Text] [Related]

  • 3. Developing therapeutics for the diseases of protein misfolding.
    May BC, Govaerts C, Cohen FE.
    Neurology; 2006 Jan 24; 66(2 Suppl 1):S118-22. PubMed ID: 16432139
    [Abstract] [Full Text] [Related]

  • 4. [Diseases associated with protein aggregation].
    Bak D, Milewski M.
    Postepy Biochem; 2005 Jan 24; 51(3):297-307. PubMed ID: 16381174
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  • 6. The environmental dependency of protein folding best explains prion and amyloid diseases.
    Kelly JW.
    Proc Natl Acad Sci U S A; 1998 Feb 03; 95(3):930-2. PubMed ID: 9448261
    [No Abstract] [Full Text] [Related]

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  • 8. Protein conformational misfolding and amyloid formation: characteristics of a new class of disorders that include Alzheimer's and Prion diseases.
    Thompson AJ, Barrow CJ.
    Curr Med Chem; 2002 Oct 03; 9(19):1751-62. PubMed ID: 12369885
    [Abstract] [Full Text] [Related]

  • 9. Mechanical stress and formation of protein aggregates in neurodegenerative disorders.
    Hachiya NS, Kozuka Y, Kaneko K.
    Med Hypotheses; 2008 Oct 03; 70(5):1034-7. PubMed ID: 17910993
    [Abstract] [Full Text] [Related]

  • 10. Roles of extracellular chaperones in amyloidosis.
    Wyatt AR, Yerbury JJ, Dabbs RA, Wilson MR.
    J Mol Biol; 2012 Aug 24; 421(4-5):499-516. PubMed ID: 22248589
    [Abstract] [Full Text] [Related]

  • 11. Protein folding and cutaneous diseases.
    Botelho MG, Lupi O.
    Int J Dermatol; 2008 Dec 24; 47(12):1225-33. PubMed ID: 19126006
    [No Abstract] [Full Text] [Related]

  • 12. Maillard reaction versus other nonenzymatic modifications in neurodegenerative processes.
    Pamplona R, Ilieva E, Ayala V, Bellmunt MJ, Cacabelos D, Dalfo E, Ferrer I, Portero-Otin M.
    Ann N Y Acad Sci; 2008 Apr 24; 1126():315-9. PubMed ID: 18079479
    [Abstract] [Full Text] [Related]

  • 13. Protein misfolding, evolution and disease.
    Dobson CM.
    Trends Biochem Sci; 1999 Sep 24; 24(9):329-32. PubMed ID: 10470028
    [No Abstract] [Full Text] [Related]

  • 14. [Chaperono- and proteinopathies: molecular basis--novel therapeutic options].
    Maier CP.
    Dtsch Med Wochenschr; 2008 Apr 24; 133(15):777-81. PubMed ID: 18382952
    [No Abstract] [Full Text] [Related]

  • 15. [Approaches from genetical analyses on human diseases: neurodegenerative diseases].
    Kakizuka A.
    Tanpakushitsu Kakusan Koso; 2000 Apr 24; 45(6 Suppl):792-7. PubMed ID: 10771635
    [No Abstract] [Full Text] [Related]

  • 16. The attention of researchers on the biological process of protein folding has turned to the formation and properties of aggregates.
    Ellis RJ.
    Semin Cell Dev Biol; 2004 Feb 24; 15(1):1-2. PubMed ID: 15036201
    [No Abstract] [Full Text] [Related]

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  • 19. Emerging roles of J proteins in neurodegenerative disorders.
    Gibbs SJ, Braun JE.
    Neurobiol Dis; 2008 Nov 24; 32(2):196-9. PubMed ID: 18760363
    [Abstract] [Full Text] [Related]

  • 20. Intrinsic disorder in proteins associated with neurodegenerative diseases.
    Uversky VN.
    Front Biosci (Landmark Ed); 2009 Jun 01; 14(14):5188-238. PubMed ID: 19482612
    [Abstract] [Full Text] [Related]


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