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3. The "Down syndrome critical region" is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome. Belichenko NP, Belichenko PV, Kleschevnikov AM, Salehi A, Reeves RH, Mobley WC. J Neurosci; 2009 May 06; 29(18):5938-48. PubMed ID: 19420260 [Abstract] [Full Text] [Related]
5. A chromosome 21 critical region does not cause specific Down syndrome phenotypes. Olson LE, Richtsmeier JT, Leszl J, Reeves RH. Science; 2004 Oct 22; 306(5696):687-90. PubMed ID: 15499018 [Abstract] [Full Text] [Related]
9. Experimental parameters affecting the Morris water maze performance of a mouse model of Down syndrome. Stasko MR, Costa AC. Behav Brain Res; 2004 Sep 23; 154(1):1-17. PubMed ID: 15302106 [Abstract] [Full Text] [Related]
12. Normalization of Dyrk1A expression by AAV2/1-shDyrk1A attenuates hippocampal-dependent defects in the Ts65Dn mouse model of Down syndrome. Altafaj X, Martín ED, Ortiz-Abalia J, Valderrama A, Lao-Peregrín C, Dierssen M, Fillat C. Neurobiol Dis; 2013 Apr 23; 52():117-27. PubMed ID: 23220201 [Abstract] [Full Text] [Related]
14. Mental retardation and associated neurological dysfunctions in Down syndrome: a consequence of dysregulation in critical chromosome 21 genes and associated molecular pathways. Rachidi M, Lopes C. Eur J Paediatr Neurol; 2008 May 23; 12(3):168-82. PubMed ID: 17933568 [Abstract] [Full Text] [Related]
15. Deficits of neuronal density in CA1 and synaptic density in the dentate gyrus, CA3 and CA1, in a mouse model of Down syndrome. Kurt MA, Kafa MI, Dierssen M, Davies DC. Brain Res; 2004 Oct 01; 1022(1-2):101-9. PubMed ID: 15353219 [Abstract] [Full Text] [Related]