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Journal Abstract Search

198 related items for PubMed ID: 17341489

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  • 3. Investigation of Batten disease with the yeast Saccharomyces cerevisiae.
    Pearce DA, Sherman F.
    Mol Genet Metab; 1999 Apr; 66(4):314-9. PubMed ID: 10191120
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  • 5. btn1, the Schizosaccharomyces pombe homologue of the human Batten disease gene CLN3, regulates vacuole homeostasis.
    Gachet Y, Codlin S, Hyams JS, Mole SE.
    J Cell Sci; 2005 Dec 01; 118(Pt 23):5525-36. PubMed ID: 16291725
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  • 6. A role in vacuolar arginine transport for yeast Btn1p and for human CLN3, the protein defective in Batten disease.
    Kim Y, Ramirez-Montealegre D, Pearce DA.
    Proc Natl Acad Sci U S A; 2003 Dec 23; 100(26):15458-62. PubMed ID: 14660799
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  • 7. The subcellular location of the yeast Saccharomyces cerevisiae homologue of the protein defective in the juvenile form of Batten disease.
    Croopnick JB, Choi HC, Mueller DM.
    Biochem Biophys Res Commun; 1998 Sep 18; 250(2):335-41. PubMed ID: 9753630
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  • 8. Studies of pH regulation by Btn1p, the yeast homolog of human Cln3p.
    Pearce DA, Nosel SA, Sherman F.
    Mol Genet Metab; 1999 Apr 18; 66(4):320-3. PubMed ID: 10191121
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  • 10. Altered arginine metabolism in the central nervous system (CNS) of the Cln3-/- mouse model of juvenile Batten disease.
    Chan CH, Ramirez-Montealegre D, Pearce DA.
    Neuropathol Appl Neurobiol; 2009 Apr 18; 35(2):189-207. PubMed ID: 19284480
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  • 11. Phenotypic reversal of the btn1 defects in yeast by chloroquine: a yeast model for Batten disease.
    Pearce DA, Carr CJ, Das B, Sherman F.
    Proc Natl Acad Sci U S A; 1999 Sep 28; 96(20):11341-5. PubMed ID: 10500178
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  • 13. Saccharomyces cerevisiae lacking Btn1p modulate vacuolar ATPase activity to regulate pH imbalance in the vacuole.
    Padilla-López S, Pearce DA.
    J Biol Chem; 2006 Apr 14; 281(15):10273-80. PubMed ID: 16423829
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  • 15. pH-dependent localization of Btn1p in the yeast model for Batten disease.
    Wolfe DM, Padilla-Lopez S, Vitiello SP, Pearce DA.
    Dis Model Mech; 2011 Jan 14; 4(1):120-5. PubMed ID: 20959629
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  • 16. BTN1, a yeast gene corresponding to the human gene responsible for Batten's disease, is not essential for viability, mitochondrial function, or degradation of mitochondrial ATP synthase.
    Pearce DA, Sherman F.
    Yeast; 1997 Jun 30; 13(8):691-7. PubMed ID: 9219333
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  • 17. The Sko1p repressor and Gcn4p activator antagonistically modulate stress-regulated transcription in Saccharomyces cerevisiae.
    Pascual-Ahuir A, Serrano R, Proft M.
    Mol Cell Biol; 2001 Jan 30; 21(1):16-25. PubMed ID: 11113177
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  • 18. Nitric oxide signaling is disrupted in the yeast model for Batten disease.
    Osório NS, Carvalho A, Almeida AJ, Padilla-Lopez S, Leão C, Laranjinha J, Ludovico P, Pearce DA, Rodrigues F.
    Mol Biol Cell; 2007 Jul 30; 18(7):2755-67. PubMed ID: 17475770
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  • 19. Deletion of btn1, an orthologue of CLN3, increases glycolysis and perturbs amino acid metabolism in the fission yeast model of Batten disease.
    Pears MR, Codlin S, Haines RL, White IJ, Mortishire-Smith RJ, Mole SE, Griffin JL.
    Mol Biosyst; 2010 Jun 30; 6(6):1093-102. PubMed ID: 20485751
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  • 20. Batten disease: evaluation of CLN3 mutations on protein localization and function.
    Haskell RE, Carr CJ, Pearce DA, Bennett MJ, Davidson BL.
    Hum Mol Genet; 2000 Mar 22; 9(5):735-44. PubMed ID: 10749980
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