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Journal Abstract Search
176 related items for PubMed ID: 17360712
1. The effect of mutations in alpha-tropomyosin (E40K and E54K) that cause familial dilated cardiomyopathy on the regulatory mechanism of cardiac muscle thin filaments. Mirza M, Robinson P, Kremneva E, Copeland O, Nikolaeva O, Watkins H, Levitsky D, Redwood C, El-Mezgueldi M, Marston S. J Biol Chem; 2007 May 04; 282(18):13487-97. PubMed ID: 17360712 [Abstract] [Full Text] [Related]
2. DCM-related tropomyosin mutants E40K/E54K over-inhibit the actomyosin interaction and lead to a decrease in the number of cycling cross-bridges. Bai F, Groth HL, Kawai M. PLoS One; 2012 May 04; 7(10):e47471. PubMed ID: 23077624 [Abstract] [Full Text] [Related]
6. Effects of two familial hypertrophic cardiomyopathy mutations in alpha-tropomyosin, Asp175Asn and Glu180Gly, on the thermal unfolding of actin-bound tropomyosin. Kremneva E, Boussouf S, Nikolaeva O, Maytum R, Geeves MA, Levitsky DI. Biophys J; 2004 Dec 04; 87(6):3922-33. PubMed ID: 15454401 [Abstract] [Full Text] [Related]
8. The second half of the fourth period of tropomyosin is a key region for Ca(2+)-dependent regulation of striated muscle thin filaments. Sakuma A, Kimura-Sakiyama C, Onoue A, Shitaka Y, Kusakabe T, Miki M. Biochemistry; 2006 Aug 08; 45(31):9550-8. PubMed ID: 16878989 [Abstract] [Full Text] [Related]