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Journal Abstract Search

986 related items for PubMed ID: 17460373

  • 1. Developmental activity of the renin-angiotensin system during the "critical period" modulates later L-NAME-induced hypertension and renal injury.
    Ishiguro K, Sasamura H, Sakamaki Y, Itoh H, Saruta T.
    Hypertens Res; 2007 Jan; 30(1):63-75. PubMed ID: 17460373
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  • 2. Vaccination against the angiotensin type 1 receptor for the prevention of L-NAME-induced nephropathy.
    Azegami T, Sasamura H, Hayashi K, Itoh H.
    Hypertens Res; 2012 May; 35(5):492-9. PubMed ID: 22158111
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  • 3. Positive influence of AT(1) receptor antagonism upon the impaired celiprolol-induced vasodilatation in aorta from spontaneously hypertensive rats.
    Sauvaget F, Mallem MY, Bucas V, Gogny M, Desfontis JC, Noireaud J.
    Eur J Pharmacol; 2010 Oct 10; 644(1-3):169-75. PubMed ID: 20637193
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  • 4. Angiotensin II type 1 receptor blocker prevents atrial structural remodeling in rats with hypertension induced by chronic nitric oxide inhibition.
    Okazaki H, Minamino T, Tsukamoto O, Kim J, Okada K, Myoishi M, Wakeno M, Takashima S, Mochizuki N, Kitakaze M.
    Hypertens Res; 2006 Apr 10; 29(4):277-84. PubMed ID: 16778335
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  • 5. Tempol or candesartan prevents high-fat diet-induced hypertension and renal damage in spontaneously hypertensive rats.
    Chung S, Park CW, Shin SJ, Lim JH, Chung HW, Youn DY, Kim HW, Kim BS, Lee JH, Kim GH, Chang YS.
    Nephrol Dial Transplant; 2010 Feb 10; 25(2):389-99. PubMed ID: 19749146
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  • 6. Nitric oxide synthase inhibition accelerates the pressor response to low-dose angiotensin II, exacerbates target organ damage, and induces renin escape.
    Hu L, Sealey JE, Chen R, Zhou Y, Merali C, Shi Y, Laragh JH, Catanzaro DF.
    Am J Hypertens; 2004 May 10; 17(5 Pt 1):395-403. PubMed ID: 15110897
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  • 7. Persistent cardiovascular effects of chronic renin-angiotensin system inhibition following withdrawal in adult spontaneously hypertensive rats.
    Paull JR, Widdop RE.
    J Hypertens; 2001 Aug 10; 19(8):1393-402. PubMed ID: 11518847
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  • 8. Effects of an ARB on endothelial progenitor cell function and cardiovascular oxidation in hypertension.
    Yu Y, Fukuda N, Yao EH, Matsumoto T, Kobayashi N, Suzuki R, Tahira Y, Ueno T, Matsumoto K.
    Am J Hypertens; 2008 Jan 10; 21(1):72-7. PubMed ID: 18091747
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  • 9. Defective nitric oxide production impairs angiotensin II-induced Na-K-ATPase regulation in spontaneously hypertensive rats.
    Javkhedkar AA, Lokhandwala MF, Banday AA.
    Am J Physiol Renal Physiol; 2012 Jan 01; 302(1):F47-51. PubMed ID: 21900450
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  • 10. Systemic Aldosterone, But Not Angiotensin II, Plays a Pivotal Role in the Pathogenesis of Renal Injury in Chronic Nitric Oxide-Deficient Male Rats.
    Suehiro T, Tsuruya K, Ikeda H, Toyonaga J, Yamada S, Noguchi H, Tokumoto M, Kitazono T.
    Endocrinology; 2015 Jul 01; 156(7):2657-66. PubMed ID: 25872005
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  • 13. Angiotensin II type 2 receptors contribute to vascular responses in spontaneously hypertensive rats treated with angiotensin II type 1 receptor antagonists.
    Cosentino F, Savoia C, De Paolis P, Francia P, Russo A, Maffei A, Venturelli V, Schiavoni M, Lembo G, Volpe M.
    Am J Hypertens; 2005 Apr 01; 18(4 Pt 1):493-9. PubMed ID: 15831358
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  • 19. Angiotensin II blockade improves mitochondrial function in spontaneously hypertensive rats.
    De Cavanagh EM, Toblli JE, Ferder L, Piotrkowski B, Stella I, Fraga CG, Inserra F.
    Cell Mol Biol (Noisy-le-grand); 2005 Nov 08; 51(6):573-8. PubMed ID: 16309582
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