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363 related items for PubMed ID: 1749429

  • 1. Oncogenic and transcriptional cooperation with Ha-Ras requires phosphorylation of c-Jun on serines 63 and 73.
    Smeal T, Binetruy B, Mercola DA, Birrer M, Karin M.
    Nature; 1991 Dec 12; 354(6353):494-6. PubMed ID: 1749429
    [Abstract] [Full Text] [Related]

  • 2. The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation site.
    Bost F, Caron L, Vial E, Montreau N, Marchetti I, Dejong V, Defize L, Castellazzi M, Binétruy B.
    Oncogene; 2001 Nov 01; 20(50):7425-9. PubMed ID: 11704873
    [Abstract] [Full Text] [Related]

  • 3. c-Fos transcriptional activity stimulated by H-Ras-activated protein kinase distinct from JNK and ERK.
    Deng T, Karin M.
    Nature; 1994 Sep 08; 371(6493):171-5. PubMed ID: 8072547
    [Abstract] [Full Text] [Related]

  • 4. Ha-Ras augments c-Jun activity and stimulates phosphorylation of its activation domain.
    Binétruy B, Smeal T, Karin M.
    Nature; 1991 May 09; 351(6322):122-7. PubMed ID: 1903181
    [Abstract] [Full Text] [Related]

  • 5. Down-regulation of c-Fos/c-Jun AP-1 dimer activity by sumoylation.
    Bossis G, Malnou CE, Farras R, Andermarcher E, Hipskind R, Rodriguez M, Schmidt D, Muller S, Jariel-Encontre I, Piechaczyk M.
    Mol Cell Biol; 2005 Aug 09; 25(16):6964-79. PubMed ID: 16055710
    [Abstract] [Full Text] [Related]

  • 6. Mitogenesis of quiescent chick fibroblasts by v-Src: dependence on events at the membrane leading to early changes in AP-1.
    Catling AD, Wyke JA, Frame MC.
    Oncogene; 1993 Jul 09; 8(7):1875-86. PubMed ID: 8510932
    [Abstract] [Full Text] [Related]

  • 7. Structure and transcriptional regulation of BKJ, a novel AP-1 target gene activated during jun- or fos-induced fibroblast transformation.
    Hartl M, Bister K.
    Oncogene; 1998 Dec 03; 17(22):2901-13. PubMed ID: 9879996
    [Abstract] [Full Text] [Related]

  • 8. The inhibitory activity of a transdominant c-jun mutant fused to the ligand binding domain of the estrogen receptor.
    Kim S, Brown PH, Birrer MJ.
    Oncogene; 1996 Mar 07; 12(5):1043-53. PubMed ID: 8649795
    [Abstract] [Full Text] [Related]

  • 9. B-ATF functions as a negative regulator of AP-1 mediated transcription and blocks cellular transformation by Ras and Fos.
    Echlin DR, Tae HJ, Mitin N, Taparowsky EJ.
    Oncogene; 2000 Mar 30; 19(14):1752-63. PubMed ID: 10777209
    [Abstract] [Full Text] [Related]

  • 10. Suppression of oncogene-induced transformation by a deletion mutant of c-jun.
    Brown PH, Alani R, Preis LH, Szabo E, Birrer MJ.
    Oncogene; 1993 Apr 30; 8(4):877-86. PubMed ID: 8455942
    [Abstract] [Full Text] [Related]

  • 11. junB promoter regulation: Ras mediated transactivation by c-Ets-1 and c-Ets-2.
    Coffer P, de Jonge M, Mettouchi A, Binetruy B, Ghysdael J, Kruijer W.
    Oncogene; 1994 Mar 30; 9(3):911-21. PubMed ID: 8108135
    [Abstract] [Full Text] [Related]

  • 12. High levels of phosphorylated c-Jun, Fra-1, Fra-2 and ATF-2 proteins correlate with malignant phenotypes in the multistage mouse skin carcinogenesis model.
    Zoumpourlis V, Papassava P, Linardopoulos S, Gillespie D, Balmain A, Pintzas A.
    Oncogene; 2000 Aug 17; 19(35):4011-21. PubMed ID: 10962557
    [Abstract] [Full Text] [Related]

  • 13. Oncoprotein-mediated signalling cascade stimulates c-Jun activity by phosphorylation of serines 63 and 73.
    Smeal T, Binetruy B, Mercola D, Grover-Bardwick A, Heidecker G, Rapp UR, Karin M.
    Mol Cell Biol; 1992 Aug 17; 12(8):3507-13. PubMed ID: 1630458
    [Abstract] [Full Text] [Related]

  • 14. JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun.
    Weiss C, Schneider S, Wagner EF, Zhang X, Seto E, Bohmann D.
    EMBO J; 2003 Jul 15; 22(14):3686-95. PubMed ID: 12853483
    [Abstract] [Full Text] [Related]

  • 15. Amino acid substitutions modulate the effect of Jun on transformation, transcriptional activation and DNA replication.
    Morgan IM, Asano M, Håvarstein LS, Ishikawa H, Hiiragi T, Ito Y, Vogt PK.
    Oncogene; 1993 May 15; 8(5):1135-40. PubMed ID: 8479738
    [Abstract] [Full Text] [Related]

  • 16. The v-Jun oncoprotein replaces p39 c-Jun as the predominant AP-1 constituent in ASV17-transformed fibroblasts: implications for SAPK/JNK-mediated signal transduction.
    Kilbey A, Black EJ, Unlu M, Gillespie DA.
    Oncogene; 1996 Jun 06; 12(11):2409-18. PubMed ID: 8649782
    [Abstract] [Full Text] [Related]

  • 17. Expression of dominant negative Jun inhibits elevated AP-1 and NF-kappaB transactivation and suppresses anchorage independent growth of HPV immortalized human keratinocytes.
    Li JJ, Rhim JS, Schlegel R, Vousden KH, Colburn NH.
    Oncogene; 1998 May 28; 16(21):2711-21. PubMed ID: 9652737
    [Abstract] [Full Text] [Related]

  • 18. Induction of p202, a modulator of apoptosis, during oncogenic transformation of NIH 3T3 cells by activated H-Ras (Q61L) contributes to cell survival.
    Xin H, Geng Y, Pramanik R, Choubey D.
    J Cell Biochem; 2003 Jan 01; 88(1):191-204. PubMed ID: 12461788
    [Abstract] [Full Text] [Related]

  • 19. Oncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylation.
    Behrens A, Jochum W, Sibilia M, Wagner EF.
    Oncogene; 2000 May 18; 19(22):2657-63. PubMed ID: 10851065
    [Abstract] [Full Text] [Related]

  • 20. Mutation of a phosphorylation site in the DNA-binding domain is required for redox-independent transactivation of AP1-dependent genes by v-Jun.
    Oehler T, Pintzas A, Stumm S, Darling A, Gillespie D, Angel P.
    Oncogene; 1993 May 18; 8(5):1141-7. PubMed ID: 8479739
    [Abstract] [Full Text] [Related]

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