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169 related items for PubMed ID: 17498657

  • 1. Elimination of radiation-induced gamma-H2AX foci in mammalian nucleus can occur by histone exchange.
    Svetlova M, Solovjeva L, Nishi K, Nazarov I, Siino J, Tomilin N.
    Biochem Biophys Res Commun; 2007 Jun 29; 358(2):650-4. PubMed ID: 17498657
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  • 2. Dephosphorylation of histone gamma-H2AX during repair of DNA double-strand breaks in mammalian cells and its inhibition by calyculin A.
    Nazarov IB, Smirnova AN, Krutilina RI, Svetlova MP, Solovjeva LV, Nikiforov AA, Oei SL, Zalenskaya IA, Yau PM, Bradbury EM, Tomilin NV.
    Radiat Res; 2003 Sep 29; 160(3):309-17. PubMed ID: 12926989
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  • 3. The effect of calyculin A on the dephosphorylation of the histone γ-H2AX after formation of X-ray-induced DNA double-strand breaks in human blood lymphocytes.
    Kuefner MA, Brand M, Engert C, Kappey H, Uder M, Distel LV.
    Int J Radiat Biol; 2013 Jun 29; 89(6):424-32. PubMed ID: 23363014
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  • 4. Induction and quantification of gamma-H2AX foci following low and high LET-irradiation.
    Leatherbarrow EL, Harper JV, Cucinotta FA, O'Neill P.
    Int J Radiat Biol; 2006 Feb 29; 82(2):111-8. PubMed ID: 16546909
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  • 6. Photobleaching of GFP-labeled H2AX in chromatin: H2AX has low diffusional mobility in the nucleus.
    Siino JS, Nazarov IB, Svetlova MP, Solovjeva LV, Adamson RH, Zalenskaya IA, Yau PM, Bradbury EM, Tomilin NV.
    Biochem Biophys Res Commun; 2002 Oct 11; 297(5):1318-23. PubMed ID: 12372432
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  • 7. Histone H2AX phosphorylation in normal human cells irradiated with focused ultrasoft X rays: evidence for chromatin movement during repair.
    Hamada N, Schettino G, Kashino G, Vaid M, Suzuki K, Kodama S, Vojnovic B, Folkard M, Watanabe M, Michael BD, Prise KM.
    Radiat Res; 2006 Jul 11; 166(1 Pt 1):31-8. PubMed ID: 16808616
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  • 8. Imaging features that discriminate between foci induced by high- and low-LET radiation in human fibroblasts.
    Costes SV, Boissière A, Ravani S, Romano R, Parvin B, Barcellos-Hoff MH.
    Radiat Res; 2006 May 11; 165(5):505-15. PubMed ID: 16669704
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  • 9. Induction and repair of DNA double-strand breaks in human cells: dephosphorylation of histone H2AX and its inhibition by calyculin A.
    Antonelli F, Belli M, Cuttone G, Dini V, Esposito G, Simone G, Sorrentino E, Tabocchini MA.
    Radiat Res; 2005 Oct 11; 164(4 Pt 2):514-7. PubMed ID: 16187759
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  • 10. Low-dose hyper-radiosensitivity is not caused by a failure to recognize DNA double-strand breaks.
    Wykes SM, Piasentin E, Joiner MC, Wilson GD, Marples B.
    Radiat Res; 2006 May 11; 165(5):516-24. PubMed ID: 16669705
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  • 13. In vivo formation of gamma-H2AX and 53BP1 DNA repair foci in blood cells after radioiodine therapy of differentiated thyroid cancer.
    Lassmann M, Hänscheid H, Gassen D, Biko J, Meineke V, Reiners C, Scherthan H.
    J Nucl Med; 2010 Aug 11; 51(8):1318-25. PubMed ID: 20660387
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  • 14. Quantitative analysis reveals asynchronous and more than DSB-associated histone H2AX phosphorylation after exposure to ionizing radiation.
    Han J, Hendzel MJ, Allalunis-Turner J.
    Radiat Res; 2006 Mar 11; 165(3):283-92. PubMed ID: 16494516
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  • 15. Kinetics and dose-response of residual 53BP1/gamma-H2AX foci: co-localization, relationship with DSB repair and clonogenic survival.
    Marková E, Schultz N, Belyaev IY.
    Int J Radiat Biol; 2007 May 11; 83(5):319-29. PubMed ID: 17457757
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  • 20. Induction and Repair of DNA DSB as Revealed by H2AX Phosphorylation Foci in Human Fibroblasts Exposed to Low- and High-LET Radiation: Relationship with Early and Delayed Reproductive Cell Death.
    Antonelli F, Campa A, Esposito G, Giardullo P, Belli M, Dini V, Meschini S, Simone G, Sorrentino E, Gerardi S, Cirrone GA, Tabocchini MA.
    Radiat Res; 2015 Apr 11; 183(4):417-31. PubMed ID: 25844944
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