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Journal Abstract Search

175 related items for PubMed ID: 17569888

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  • 5. Increased cyclooxygenase-2 expression in hypothalamic paraventricular nucleus in rats with heart failure: role of nuclear factor kappaB.
    Yu Y, Kang YM, Zhang ZH, Wei SG, Chu Y, Weiss RM, Felder RB.
    Hypertension; 2007 Mar; 49(3):511-8. PubMed ID: 17242297
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  • 9. TNF-α receptor 1 knockdown in the subfornical organ ameliorates sympathetic excitation and cardiac hemodynamics in heart failure rats.
    Yu Y, Wei SG, Weiss RM, Felder RB.
    Am J Physiol Heart Circ Physiol; 2017 Oct 01; 313(4):H744-H756. PubMed ID: 28710070
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  • 10. ERK1/2 MAPK signaling in hypothalamic paraventricular nucleus contributes to sympathetic excitation in rats with heart failure after myocardial infarction.
    Yu Y, Wei SG, Zhang ZH, Weiss RM, Felder RB.
    Am J Physiol Heart Circ Physiol; 2016 Mar 15; 310(6):H732-9. PubMed ID: 26801309
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  • 12. Endoplasmic reticulum stress increases brain MAPK signaling, inflammation and renin-angiotensin system activity and sympathetic nerve activity in heart failure.
    Wei SG, Yu Y, Weiss RM, Felder RB.
    Am J Physiol Heart Circ Physiol; 2016 Oct 01; 311(4):H871-H880. PubMed ID: 27496879
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  • 16. Blood-borne interleukin-1β acts on the subfornical organ to upregulate the sympathoexcitatory milieu of the hypothalamic paraventricular nucleus.
    Wei SG, Yu Y, Felder RB.
    Am J Physiol Regul Integr Comp Physiol; 2018 Mar 01; 314(3):R447-R458. PubMed ID: 29167166
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  • 20. Partially silencing brain toll-like receptor 4 prevents in part left ventricular remodeling with sympathoinhibition in rats with myocardial infarction-induced heart failure.
    Ogawa K, Hirooka Y, Kishi T, Ide T, Sunagawa K.
    PLoS One; 2013 Mar 01; 8(7):e69053. PubMed ID: 23874864
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