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164 related items for PubMed ID: 17590433

  • 1. Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1.
    Israelson A, Zaid H, Abu-Hamad S, Nahon E, Shoshan-Barmatz V.
    Cell Calcium; 2008 Feb; 43(2):196-204. PubMed ID: 17590433
    [Abstract] [Full Text] [Related]

  • 2. The voltage-dependent anion channel-1 modulates apoptotic cell death.
    Zaid H, Abu-Hamad S, Israelson A, Nathan I, Shoshan-Barmatz V.
    Cell Death Differ; 2005 Jul; 12(7):751-60. PubMed ID: 15818409
    [Abstract] [Full Text] [Related]

  • 3. Dominant-negative VDAC1 mutants reveal oligomeric VDAC1 to be the active unit in mitochondria-mediated apoptosis.
    Mader A, Abu-Hamad S, Arbel N, Gutiérrez-Aguilar M, Shoshan-Barmatz V.
    Biochem J; 2010 Jul 01; 429(1):147-55. PubMed ID: 20420578
    [Abstract] [Full Text] [Related]

  • 4. Localization of the voltage-dependent anion channel-1 Ca2+-binding sites.
    Israelson A, Abu-Hamad S, Zaid H, Nahon E, Shoshan-Barmatz V.
    Cell Calcium; 2007 Mar 01; 41(3):235-44. PubMed ID: 16930689
    [Abstract] [Full Text] [Related]

  • 5. Hexokinase-I protection against apoptotic cell death is mediated via interaction with the voltage-dependent anion channel-1: mapping the site of binding.
    Abu-Hamad S, Zaid H, Israelson A, Nahon E, Shoshan-Barmatz V.
    J Biol Chem; 2008 May 09; 283(19):13482-90. PubMed ID: 18308720
    [Abstract] [Full Text] [Related]

  • 6. VDAC1 cysteine residues: topology and function in channel activity and apoptosis.
    Aram L, Geula S, Arbel N, Shoshan-Barmatz V.
    Biochem J; 2010 Apr 14; 427(3):445-54. PubMed ID: 20192921
    [Abstract] [Full Text] [Related]

  • 7. In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death.
    Azoulay-Zohar H, Israelson A, Abu-Hamad S, Shoshan-Barmatz V.
    Biochem J; 2004 Jan 15; 377(Pt 2):347-55. PubMed ID: 14561215
    [Abstract] [Full Text] [Related]

  • 8. The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins.
    Abu-Hamad S, Arbel N, Calo D, Arzoine L, Israelson A, Keinan N, Ben-Romano R, Friedman O, Shoshan-Barmatz V.
    J Cell Sci; 2009 Jun 01; 122(Pt 11):1906-16. PubMed ID: 19461077
    [Abstract] [Full Text] [Related]

  • 9. An N-terminal nucleotide-binding site in VDAC1: involvement in regulating mitochondrial function.
    Yehezkel G, Abu-Hamad S, Shoshan-Barmatz V.
    J Cell Physiol; 2007 Aug 01; 212(2):551-61. PubMed ID: 17503466
    [Abstract] [Full Text] [Related]

  • 10. Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo.
    Shoshan-Barmatz V, Keinan N, Abu-Hamad S, Tyomkin D, Aram L.
    Biochim Biophys Acta; 2010 Aug 01; 1797(6-7):1281-91. PubMed ID: 20214874
    [Abstract] [Full Text] [Related]

  • 11. Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase.
    Shoshan-Barmatz V, Zakar M, Rosenthal K, Abu-Hamad S.
    Biochim Biophys Acta; 2009 May 01; 1787(5):421-30. PubMed ID: 19094960
    [Abstract] [Full Text] [Related]

  • 12. A soluble form of the pilus protein FimA targets the VDAC-hexokinase complex at mitochondria to suppress host cell apoptosis.
    Sukumaran SK, Fu NY, Tin CB, Wan KF, Lee SS, Yu VC.
    Mol Cell; 2010 Mar 26; 37(6):768-83. PubMed ID: 20347420
    [Abstract] [Full Text] [Related]

  • 13. Molecular model of hexokinase binding to the outer mitochondrial membrane porin (VDAC1): Implication for the design of new cancer therapies.
    Rosano C.
    Mitochondrion; 2011 May 26; 11(3):513-9. PubMed ID: 21315184
    [Abstract] [Full Text] [Related]

  • 14. Voltage-dependent anion channel 1 is involved in endostatin-induced endothelial cell apoptosis.
    Yuan S, Fu Y, Wang X, Shi H, Huang Y, Song X, Li L, Song N, Luo Y.
    FASEB J; 2008 Aug 26; 22(8):2809-20. PubMed ID: 18381814
    [Abstract] [Full Text] [Related]

  • 15. VDAC1, having a shorter N-terminus than VDAC2 but showing the same migration in an SDS-polyacrylamide gel, is the predominant form expressed in mitochondria of various tissues.
    Yamamoto T, Yamada A, Watanabe M, Yoshimura Y, Yamazaki N, Yoshimura Y, Yamauchi T, Kataoka M, Nagata T, Terada H, Shinohara Y.
    J Proteome Res; 2006 Dec 26; 5(12):3336-44. PubMed ID: 17137335
    [Abstract] [Full Text] [Related]

  • 16. Voltage-dependent anion channel 1-based peptides interact with Bcl-2 to prevent antiapoptotic activity.
    Arbel N, Shoshan-Barmatz V.
    J Biol Chem; 2010 Feb 26; 285(9):6053-62. PubMed ID: 20037155
    [Abstract] [Full Text] [Related]

  • 17. Structure-based analysis of VDAC1: N-terminus location, translocation, channel gating and association with anti-apoptotic proteins.
    Geula S, Ben-Hail D, Shoshan-Barmatz V.
    Biochem J; 2012 Jun 15; 444(3):475-85. PubMed ID: 22397371
    [Abstract] [Full Text] [Related]

  • 18. The expression level of the voltage-dependent anion channel controls life and death of the cell.
    Abu-Hamad S, Sivan S, Shoshan-Barmatz V.
    Proc Natl Acad Sci U S A; 2006 Apr 11; 103(15):5787-92. PubMed ID: 16585511
    [Abstract] [Full Text] [Related]

  • 19. ADP/ATP carrier is required for mitochondrial outer membrane permeabilization and cytochrome c release in yeast apoptosis.
    Pereira C, Camougrand N, Manon S, Sousa MJ, Côrte-Real M.
    Mol Microbiol; 2007 Nov 11; 66(3):571-82. PubMed ID: 17822411
    [Abstract] [Full Text] [Related]

  • 20. Voltage-dependent anion channel 1-based peptides interact with hexokinase to prevent its anti-apoptotic activity.
    Arzoine L, Zilberberg N, Ben-Romano R, Shoshan-Barmatz V.
    J Biol Chem; 2009 Feb 06; 284(6):3946-55. PubMed ID: 19049977
    [Abstract] [Full Text] [Related]

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