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Journal Abstract Search

228 related items for PubMed ID: 17597610

  • 1. The lack of effect of specific overexpression of IGF-1 in the central nervous system or skeletal muscle on pathophysiology in the G93A SOD-1 mouse model of ALS.
    Messi ML, Clark HM, Prevette DM, Oppenheim RW, Delbono O.
    Exp Neurol; 2007 Sep; 207(1):52-63. PubMed ID: 17597610
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  • 2. Muscle-derived but not centrally derived transgene GDNF is neuroprotective in G93A-SOD1 mouse model of ALS.
    Li W, Brakefield D, Pan Y, Hunter D, Myckatyn TM, Parsadanian A.
    Exp Neurol; 2007 Feb; 203(2):457-71. PubMed ID: 17034790
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  • 4. Impairments to the GH-IGF-I axis in hSOD1G93A mice give insight into possible mechanisms of GH dysregulation in patients with amyotrophic lateral sclerosis.
    Steyn FJ, Ngo ST, Lee JD, Leong JW, Buckley AJ, Veldhuis JD, McCombe PA, Chen C, Bellingham MC.
    Endocrinology; 2012 Aug; 153(8):3735-46. PubMed ID: 22621959
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  • 6. Activation of the stress-activated MAP kinase, p38, but not JNK in cortical motor neurons during early presymptomatic stages of amyotrophic lateral sclerosis in transgenic mice.
    Holasek SS, Wengenack TM, Kandimalla KK, Montano C, Gregor DM, Curran GL, Poduslo JF.
    Brain Res; 2005 May 31; 1045(1-2):185-98. PubMed ID: 15910777
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  • 8. Absence of p53: no effect in a transgenic mouse model of familial amyotrophic lateral sclerosis.
    Kuntz C, Kinoshita Y, Beal MF, Donehower LA, Morrison RS.
    Exp Neurol; 2000 Sep 31; 165(1):184-90. PubMed ID: 10964497
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  • 10. Therapeutic benefit of intrathecal injection of insulin-like growth factor-1 in a mouse model of Amyotrophic Lateral Sclerosis.
    Nagano I, Ilieva H, Shiote M, Murakami T, Yokoyama M, Shoji M, Abe K.
    J Neurol Sci; 2005 Aug 15; 235(1-2):61-8. PubMed ID: 15990113
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  • 11. L-carnitine suppresses the onset of neuromuscular degeneration and increases the life span of mice with familial amyotrophic lateral sclerosis.
    Kira Y, Nishikawa M, Ochi A, Sato E, Inoue M.
    Brain Res; 2006 Jan 27; 1070(1):206-14. PubMed ID: 16412993
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  • 12. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.
    Watanabe M, Dykes-Hoberg M, Culotta VC, Price DL, Wong PC, Rothstein JD.
    Neurobiol Dis; 2001 Dec 27; 8(6):933-41. PubMed ID: 11741389
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  • 14. Disease-dependent reciprocal phosphorylation of serine and tyrosine residues of c-Met/HGF receptor contributes disease retardation of a transgenic mouse model of ALS.
    Kadoyama K, Funakoshi H, Ohya-Shimada W, Nakamura T, Matsumoto K, Matsuyama S, Nakamura T.
    Neurosci Res; 2009 Oct 27; 65(2):194-200. PubMed ID: 19595710
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  • 16. Systemic administration of scAAV9-IGF1 extends survival in SOD1G93A ALS mice via inhibiting p38 MAPK and the JNK-mediated apoptosis pathway.
    Wang W, Wen D, Duan W, Yin J, Cui C, Wang Y, Li Z, Liu Y, Li C.
    Brain Res Bull; 2018 May 27; 139():203-210. PubMed ID: 29499331
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  • 17. Matrix metalloproteinase-9 regulates TNF-alpha and FasL expression in neuronal, glial cells and its absence extends life in a transgenic mouse model of amyotrophic lateral sclerosis.
    Kiaei M, Kipiani K, Calingasan NY, Wille E, Chen J, Heissig B, Rafii S, Lorenzl S, Beal MF.
    Exp Neurol; 2007 May 27; 205(1):74-81. PubMed ID: 17362932
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  • 18. Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
    Hegedus J, Putman CT, Gordon T.
    Neurobiol Dis; 2007 Nov 27; 28(2):154-64. PubMed ID: 17766128
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  • 19. Local expression of mIgf-1 modulates ubiquitin, caspase and CDK5 expression in skeletal muscle of an ALS mouse model.
    Dobrowolny G, Aucello M, Molinaro M, Musarò A.
    Neurol Res; 2008 Mar 27; 30(2):131-6. PubMed ID: 18397603
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