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421 related items for PubMed ID: 17632123
1. Protein kinase C protects preconditioned rabbit hearts by increasing sensitivity of adenosine A2b-dependent signaling during early reperfusion. Kuno A, Critz SD, Cui L, Solodushko V, Yang XM, Krahn T, Albrecht B, Philipp S, Cohen MV, Downey JM. J Mol Cell Cardiol; 2007 Sep; 43(3):262-71. PubMed ID: 17632123 [Abstract] [Full Text] [Related]
3. Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C. Kuno A, Solenkova NV, Solodushko V, Dost T, Liu Y, Yang XM, Cohen MV, Downey JM. Am J Physiol Heart Circ Physiol; 2008 Sep; 295(3):H1288-H1295. PubMed ID: 18660452 [Abstract] [Full Text] [Related]
4. Cardioprotective PKG-independent NO signaling at reperfusion. Cohen MV, Yang XM, Liu Y, Solenkova NV, Downey JM. Am J Physiol Heart Circ Physiol; 2010 Dec; 299(6):H2028-36. PubMed ID: 20852051 [Abstract] [Full Text] [Related]
5. Protein tyrosine kinase is downstream of protein kinase C for ischemic preconditioning's anti-infarct effect in the rabbit heart. Baines CP, Wang L, Cohen MV, Downey JM. J Mol Cell Cardiol; 1998 Feb; 30(2):383-92. PubMed ID: 9515015 [Abstract] [Full Text] [Related]
6. Both A2a and A2b adenosine receptors at reperfusion are necessary to reduce infarct size in mouse hearts. Methner C, Schmidt K, Cohen MV, Downey JM, Krieg T. Am J Physiol Heart Circ Physiol; 2010 Oct; 299(4):H1262-4. PubMed ID: 20709859 [Abstract] [Full Text] [Related]
7. Adenosine 2B Receptor Activation Reduces Myocardial Reperfusion Injury by Promoting Anti-Inflammatory Macrophages Differentiation via PI3K/Akt Pathway. Tian Y, Piras BA, Kron IL, French BA, Yang Z. Oxid Med Cell Longev; 2015 Oct; 2015():585297. PubMed ID: 26161239 [Abstract] [Full Text] [Related]
13. Cooperative cardioprotection through adenosine A1 and A2A receptor agonism in ischemia-reperfused isolated mouse heart. Urmaliya VB, Pouton CW, Ledent C, Short JL, White PJ. J Cardiovasc Pharmacol; 2010 Oct; 56(4):379-88. PubMed ID: 20930592 [Abstract] [Full Text] [Related]
14. NECA and bradykinin at reperfusion reduce infarction in rabbit hearts by signaling through PI3K, ERK, and NO. Yang XM, Krieg T, Cui L, Downey JM, Cohen MV. J Mol Cell Cardiol; 2004 Mar; 36(3):411-21. PubMed ID: 15010280 [Abstract] [Full Text] [Related]
17. Evidence that the acute phase of ischemic preconditioning does not require signaling by the A 2B adenosine receptor. Maas JE, Wan TC, Figler RA, Gross GJ, Auchampach JA. J Mol Cell Cardiol; 2010 Nov; 49(5):886-93. PubMed ID: 20797398 [Abstract] [Full Text] [Related]
18. Activation of RISK and SAFE pathways is not involved in the effects of Cx43 deficiency on tolerance to ischemia-reperfusion injury and preconditioning protection. Sánchez JA, Rodríguez-Sinovas A, Barba I, Miró-Casas E, Fernández-Sanz C, Ruiz-Meana M, Alburquerque-Béjar JJ, García-Dorado D. Basic Res Cardiol; 2013 May; 108(3):351. PubMed ID: 23595215 [Abstract] [Full Text] [Related]
19. Opioid receptor contributes to ischemic preconditioning through protein kinase C activation in rabbits. Miki T, Cohen MV, Downey JM. Mol Cell Biochem; 1998 Sep; 186(1-2):3-12. PubMed ID: 9774179 [Abstract] [Full Text] [Related]
20. Adenosine A2A and A2B receptors work in concert to induce a strong protection against reperfusion injury in rat hearts. Xi J, McIntosh R, Shen X, Lee S, Chanoit G, Criswell H, Zvara DA, Xu Z. J Mol Cell Cardiol; 2009 Nov; 47(5):684-90. PubMed ID: 19695259 [Abstract] [Full Text] [Related] Page: [Next] [New Search]