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185 related items for PubMed ID: 17636114
1. Physical activity modifies the effect of SNPs in the SLC2A2 (GLUT2) and ABCC8 (SUR1) genes on the risk of developing type 2 diabetes. Kilpeläinen TO, Lakka TA, Laaksonen DE, Laukkanen O, Lindström J, Eriksson JG, Valle TT, Hämäläinen H, Aunola S, Ilanne-Parikka P, Keinänen-Kiukaanniemi S, Tuomilehto J, Uusitupa M, Laakso M, Finnish Diabetes Prevention Study Group. Physiol Genomics; 2007 Oct 22; 31(2):264-72. PubMed ID: 17636114 [Abstract] [Full Text] [Related]
2. Polymorphisms of the SUR1 (ABCC8) and Kir6.2 (KCNJ11) genes predict the conversion from impaired glucose tolerance to type 2 diabetes. The Finnish Diabetes Prevention Study. Laukkanen O, Pihlajamäki J, Lindström J, Eriksson J, Valle TT, Hämäläinen H, Ilanne-Parikka P, Keinänen-Kiukaanniemi S, Tuomilehto J, Uusitupa M, Laakso M, Finnish Diabetes Prevention Study Group. J Clin Endocrinol Metab; 2004 Dec 22; 89(12):6286-90. PubMed ID: 15579791 [Abstract] [Full Text] [Related]
3. Type 2 diabetes-associated missense polymorphisms KCNJ11 E23K and ABCC8 A1369S influence progression to diabetes and response to interventions in the Diabetes Prevention Program. Florez JC, Jablonski KA, Kahn SE, Franks PW, Dabelea D, Hamman RF, Knowler WC, Nathan DM, Altshuler D. Diabetes; 2007 Feb 22; 56(2):531-6. PubMed ID: 17259403 [Abstract] [Full Text] [Related]
4. Interaction of single nucleotide polymorphisms in ADRB2, ADRB3, TNF, IL6, IGF1R, LIPC, LEPR, and GHRL with physical activity on the risk of type 2 diabetes mellitus and changes in characteristics of the metabolic syndrome: The Finnish Diabetes Prevention Study. Kilpeläinen TO, Lakka TA, Laaksonen DE, Mager U, Salopuro T, Kubaszek A, Todorova B, Laukkanen O, Lindström J, Eriksson JG, Hämäläinen H, Aunola S, Ilanne-Parikka P, Keinänen-Kiukaanniemi S, Tuomilehto J, Laakso M, Uusitupa M, Finnish Diabetes Prevention Study Group. Metabolism; 2008 Mar 22; 57(3):428-36. PubMed ID: 18249219 [Abstract] [Full Text] [Related]
5. Polymorphisms in the SLC2A2 (GLUT2) gene are associated with the conversion from impaired glucose tolerance to type 2 diabetes: the Finnish Diabetes Prevention Study. Laukkanen O, Lindström J, Eriksson J, Valle TT, Hämäläinen H, Ilanne-Parikka P, Keinänen-Kiukaanniemi S, Tuomilehto J, Uusitupa M, Laakso M, Finnish Diabetes Prevention Study. Diabetes; 2005 Jul 22; 54(7):2256-60. PubMed ID: 15983230 [Abstract] [Full Text] [Related]
6. Effects of single nucleotide polymorphisms in K(ATP) channel genes on type 2 diabetes in a Turkish population. Gonen MS, Arikoglu H, Erkoc Kaya D, Ozdemir H, Ipekci SH, Arslan A, Kayis SA, Gogebakan B. Arch Med Res; 2012 May 22; 43(4):317-23. PubMed ID: 22704848 [Abstract] [Full Text] [Related]
7. Association of adiponectin (AdipoQ) and sulphonylurea receptor (ABCC8) gene polymorphisms with Type 2 Diabetes in North Indian population of Punjab. Matharoo K, Arora P, Bhanwer AJ. Gene; 2013 Sep 15; 527(1):228-34. PubMed ID: 23764562 [Abstract] [Full Text] [Related]
8. Candidate gene association study in type 2 diabetes indicates a role for genes involved in beta-cell function as well as insulin action. Barroso I, Luan J, Middelberg RP, Harding AH, Franks PW, Jakes RW, Clayton D, Schafer AJ, O'Rahilly S, Wareham NJ. PLoS Biol; 2003 Oct 15; 1(1):E20. PubMed ID: 14551916 [Abstract] [Full Text] [Related]
12. Common variants in the ATP-sensitive K+ channel genes KCNJ11 (Kir6.2) and ABCC8 (SUR1) in relation to glucose intolerance: population-based studies and meta-analyses. van Dam RM, Hoebee B, Seidell JC, Schaap MM, de Bruin TW, Feskens EJ. Diabet Med; 2005 May 15; 22(5):590-8. PubMed ID: 15842514 [Abstract] [Full Text] [Related]
13. Sulfonylurea receptor -1 (SUR1): genetic and metabolic evidences for a role in the susceptibility to type 2 diabetes mellitus. Reis AF, Velho G. Diabetes Metab; 2002 Feb 15; 28(1):14-9. PubMed ID: 11938023 [Abstract] [Full Text] [Related]
15. SNPs in PPARG associate with type 2 diabetes and interact with physical activity. Kilpeläinen TO, Lakka TA, Laaksonen DE, Lindström J, Eriksson JG, Valle TT, Hämäläinen H, Ilanne-Parikka P, Keinänen-Kiukaanniemi S, Lindi V, Tuomilehto J, Uusitupa M, Laakso M. Med Sci Sports Exerc; 2008 Jan 15; 40(1):25-33. PubMed ID: 18091023 [Abstract] [Full Text] [Related]
16. The common C49620T polymorphism in the sulfonylurea receptor gene (ABCC8), pancreatic beta cell function and long-term diabetic complications in obese patients with long-lasting type 2 diabetes mellitus. Stefanski A, Majkowska L, Ciechanowicz A, Frankow M, Safranow K, Parczewski M, Pilarska K. Exp Clin Endocrinol Diabetes; 2007 May 15; 115(5):317-21. PubMed ID: 17516295 [Abstract] [Full Text] [Related]
18. KCNJ11, ABCC8 and TCF7L2 polymorphisms and the response to sulfonylurea treatment in patients with type 2 diabetes: a bioinformatics assessment. Song J, Yang Y, Mauvais-Jarvis F, Wang YP, Niu T. BMC Med Genet; 2017 Jun 06; 18(1):64. PubMed ID: 28587604 [Abstract] [Full Text] [Related]
19. Large-scale association studies of variants in genes encoding the pancreatic beta-cell KATP channel subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) confirm that the KCNJ11 E23K variant is associated with type 2 diabetes. Gloyn AL, Weedon MN, Owen KR, Turner MJ, Knight BA, Hitman G, Walker M, Levy JC, Sampson M, Halford S, McCarthy MI, Hattersley AT, Frayling TM. Diabetes; 2003 Feb 06; 52(2):568-72. PubMed ID: 12540637 [Abstract] [Full Text] [Related]
20. The ATP-sensitive K(+) channel ABCC8 S1369A type 2 diabetes risk variant increases MgATPase activity. Fatehi M, Raja M, Carter C, Soliman D, Holt A, Light PE. Diabetes; 2012 Jan 06; 61(1):241-9. PubMed ID: 22187380 [Abstract] [Full Text] [Related] Page: [Next] [New Search]