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525 related items for PubMed ID: 17644079

  • 1. K201 modulates excitation-contraction coupling and spontaneous Ca2+ release in normal adult rabbit ventricular cardiomyocytes.
    Loughrey CM, Otani N, Seidler T, Craig MA, Matsuda R, Kaneko N, Smith GL.
    Cardiovasc Res; 2007 Nov 01; 76(2):236-46. PubMed ID: 17644079
    [Abstract] [Full Text] [Related]

  • 2. K201 (JTV-519) alters the spatiotemporal properties of diastolic Ca(2+) release and the associated diastolic contraction during β-adrenergic stimulation in rat ventricular cardiomyocytes.
    Elliott EB, Hasumi H, Otani N, Matsuda T, Matsuda R, Kaneko N, Smith GL, Loughrey CM.
    Basic Res Cardiol; 2011 Nov 01; 106(6):1009-22. PubMed ID: 21901290
    [Abstract] [Full Text] [Related]

  • 3. K201 (JTV519) is a Ca2+-Dependent Blocker of SERCA and a Partial Agonist of Ryanodine Receptors in Striated Muscle.
    Darcy YL, Diaz-Sylvester PL, Copello JA.
    Mol Pharmacol; 2016 Aug 01; 90(2):106-15. PubMed ID: 27235390
    [Abstract] [Full Text] [Related]

  • 4. JTV519 (K201) reduces sarcoplasmic reticulum Ca²⁺ leak and improves diastolic function in vitro in murine and human non-failing myocardium.
    Sacherer M, Sedej S, Wakuła P, Wallner M, Vos MA, Kockskämper J, Stiegler P, Sereinigg M, von Lewinski D, Antoons G, Pieske BM, Heinzel FR, CONTICA investigators.
    Br J Pharmacol; 2012 Oct 01; 167(3):493-504. PubMed ID: 22509897
    [Abstract] [Full Text] [Related]

  • 5. A novel mechanism of tandem activation of ryanodine receptors by cytosolic and SR luminal Ca2+ during excitation-contraction coupling in atrial myocytes.
    Maxwell JT, Blatter LA.
    J Physiol; 2017 Jun 15; 595(12):3835-3845. PubMed ID: 28028837
    [Abstract] [Full Text] [Related]

  • 6. Na+-dependent SR Ca2+ overload induces arrhythmogenic events in mouse cardiomyocytes with a human CPVT mutation.
    Sedej S, Heinzel FR, Walther S, Dybkova N, Wakula P, Groborz J, Gronau P, Maier LS, Vos MA, Lai FA, Napolitano C, Priori SG, Kockskämper J, Pieske B.
    Cardiovasc Res; 2010 Jul 01; 87(1):50-9. PubMed ID: 20080988
    [Abstract] [Full Text] [Related]

  • 7. S100A1 increases the gain of excitation-contraction coupling in isolated rabbit ventricular cardiomyocytes.
    Kettlewell S, Most P, Currie S, Koch WJ, Smith GL.
    J Mol Cell Cardiol; 2005 Dec 01; 39(6):900-10. PubMed ID: 16236309
    [Abstract] [Full Text] [Related]

  • 8. Overexpression of FK-506 binding protein 12.0 modulates excitation contraction coupling in adult rabbit ventricular cardiomyocytes.
    Seidler T, Loughrey CM, Zibrova D, Kettlewell S, Teucher N, Kögler H, Hasenfuss G, Smith GL.
    Circ Res; 2007 Nov 09; 101(10):1020-9. PubMed ID: 17872463
    [Abstract] [Full Text] [Related]

  • 9. Assessment of sarcoplasmic reticulum Ca(2+) flux pathways in cardiomyocytes from rabbits with infarct-induced left-ventricular dysfunction.
    Neary P, Duncan AM, Cobbe SM, Smith GL.
    Pflugers Arch; 2002 Jun 09; 444(3):360-71. PubMed ID: 12111244
    [Abstract] [Full Text] [Related]

  • 10. High-mobility group box 1 (HMGB1) impaired cardiac excitation-contraction coupling by enhancing the sarcoplasmic reticulum (SR) Ca(2+) leak through TLR4-ROS signaling in cardiomyocytes.
    Zhang C, Mo M, Ding W, Liu W, Yan D, Deng J, Luo X, Liu J.
    J Mol Cell Cardiol; 2014 Sep 09; 74():260-73. PubMed ID: 24937603
    [Abstract] [Full Text] [Related]

  • 11. Defective domain-domain interactions within the ryanodine receptor as a critical cause of diastolic Ca2+ leak in failing hearts.
    Tateishi H, Yano M, Mochizuki M, Suetomi T, Ono M, Xu X, Uchinoumi H, Okuda S, Oda T, Kobayashi S, Yamamoto T, Ikeda Y, Ohkusa T, Ikemoto N, Matsuzaki M.
    Cardiovasc Res; 2009 Feb 15; 81(3):536-45. PubMed ID: 18996969
    [Abstract] [Full Text] [Related]

  • 12. Effects of adenovirus-mediated sorcin overexpression on excitation-contraction coupling in isolated rabbit cardiomyocytes.
    Seidler T, Miller SL, Loughrey CM, Kania A, Burow A, Kettlewell S, Teucher N, Wagner S, Kögler H, Meyers MB, Hasenfuss G, Smith GL.
    Circ Res; 2003 Jul 25; 93(2):132-9. PubMed ID: 12805242
    [Abstract] [Full Text] [Related]

  • 13. Mechanistic basis of excitation-contraction coupling in human pluripotent stem cell-derived ventricular cardiomyocytes revealed by Ca2+ spark characteristics: direct evidence of functional Ca2+-induced Ca2+ release.
    Li S, Cheng H, Tomaselli GF, Li RA.
    Heart Rhythm; 2014 Jan 25; 11(1):133-40. PubMed ID: 24096168
    [Abstract] [Full Text] [Related]

  • 14. Increasing ryanodine receptor open probability alone does not produce arrhythmogenic calcium waves: threshold sarcoplasmic reticulum calcium content is required.
    Venetucci LA, Trafford AW, Eisner DA.
    Circ Res; 2007 Jan 05; 100(1):105-11. PubMed ID: 17110597
    [Abstract] [Full Text] [Related]

  • 15. Overexpression of FK506-binding protein FKBP12.6 in cardiomyocytes reduces ryanodine receptor-mediated Ca(2+) leak from the sarcoplasmic reticulum and increases contractility.
    Prestle J, Janssen PM, Janssen AP, Zeitz O, Lehnart SE, Bruce L, Smith GL, Hasenfuss G.
    Circ Res; 2001 Feb 02; 88(2):188-94. PubMed ID: 11157671
    [Abstract] [Full Text] [Related]

  • 16. Role of SERCA and the sarcoplasmic reticulum calcium content on calcium waves propagation in rat ventricular myocytes.
    Salazar-Cantú A, Pérez-Treviño P, Montalvo-Parra D, Balderas-Villalobos J, Gómez-Víquez NL, García N, Altamirano J.
    Arch Biochem Biophys; 2016 Aug 15; 604():11-9. PubMed ID: 27242324
    [Abstract] [Full Text] [Related]

  • 17. Effects of calsequestrin over-expression on excitation-contraction coupling in isolated rabbit cardiomyocytes.
    Miller SL, Currie S, Loughrey CM, Kettlewell S, Seidler T, Reynolds DF, Hasenfuss G, Smith GL.
    Cardiovasc Res; 2005 Sep 01; 67(4):667-77. PubMed ID: 15913577
    [Abstract] [Full Text] [Related]

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