These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

645 related items for PubMed ID: 17872463

  • 1. Overexpression of FK-506 binding protein 12.0 modulates excitation contraction coupling in adult rabbit ventricular cardiomyocytes.
    Seidler T, Loughrey CM, Zibrova D, Kettlewell S, Teucher N, Kögler H, Hasenfuss G, Smith GL.
    Circ Res; 2007 Nov 09; 101(10):1020-9. PubMed ID: 17872463
    [Abstract] [Full Text] [Related]

  • 2. Effects of adenovirus-mediated sorcin overexpression on excitation-contraction coupling in isolated rabbit cardiomyocytes.
    Seidler T, Miller SL, Loughrey CM, Kania A, Burow A, Kettlewell S, Teucher N, Wagner S, Kögler H, Meyers MB, Hasenfuss G, Smith GL.
    Circ Res; 2003 Jul 25; 93(2):132-9. PubMed ID: 12805242
    [Abstract] [Full Text] [Related]

  • 3. Over-expression of FK506-binding protein FKBP12.6 alters excitation-contraction coupling in adult rabbit cardiomyocytes.
    Loughrey CM, Seidler T, Miller SL, Prestle J, MacEachern KE, Reynolds DF, Hasenfuss G, Smith GL.
    J Physiol; 2004 May 01; 556(Pt 3):919-34. PubMed ID: 14966299
    [Abstract] [Full Text] [Related]

  • 4. Effects of calsequestrin over-expression on excitation-contraction coupling in isolated rabbit cardiomyocytes.
    Miller SL, Currie S, Loughrey CM, Kettlewell S, Seidler T, Reynolds DF, Hasenfuss G, Smith GL.
    Cardiovasc Res; 2005 Sep 01; 67(4):667-77. PubMed ID: 15913577
    [Abstract] [Full Text] [Related]

  • 5. Conditional FKBP12.6 overexpression in mouse cardiac myocytes prevents triggered ventricular tachycardia through specific alterations in excitation-contraction coupling.
    Gellen B, Fernández-Velasco M, Briec F, Vinet L, LeQuang K, Rouet-Benzineb P, Bénitah JP, Pezet M, Palais G, Pellegrin N, Zhang A, Perrier R, Escoubet B, Marniquet X, Richard S, Jaisser F, Gómez AM, Charpentier F, Mercadier JJ.
    Circulation; 2008 Apr 08; 117(14):1778-86. PubMed ID: 18378612
    [Abstract] [Full Text] [Related]

  • 6. S100A1 increases the gain of excitation-contraction coupling in isolated rabbit ventricular cardiomyocytes.
    Kettlewell S, Most P, Currie S, Koch WJ, Smith GL.
    J Mol Cell Cardiol; 2005 Dec 08; 39(6):900-10. PubMed ID: 16236309
    [Abstract] [Full Text] [Related]

  • 7.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 8.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 9.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 10. Effects of FK506 on [Ca2+]i differ in mouse and rabbit ventricular myocytes.
    Su Z, Sugishita K, Li F, Ritter M, Barry WH.
    J Pharmacol Exp Ther; 2003 Jan 08; 304(1):334-41. PubMed ID: 12490609
    [Abstract] [Full Text] [Related]

  • 11. S100A1 decreases calcium spark frequency and alters their spatial characteristics in permeabilized adult ventricular cardiomyocytes.
    Völkers M, Loughrey CM, Macquaide N, Remppis A, DeGeorge BR, Wegner FV, Friedrich O, Fink RH, Koch WJ, Smith GL, Most P.
    Cell Calcium; 2007 Feb 08; 41(2):135-43. PubMed ID: 16919727
    [Abstract] [Full Text] [Related]

  • 12.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 13. Overexpression of FK506-binding protein FKBP12.6 in cardiomyocytes reduces ryanodine receptor-mediated Ca(2+) leak from the sarcoplasmic reticulum and increases contractility.
    Prestle J, Janssen PM, Janssen AP, Zeitz O, Lehnart SE, Bruce L, Smith GL, Hasenfuss G.
    Circ Res; 2001 Feb 02; 88(2):188-94. PubMed ID: 11157671
    [Abstract] [Full Text] [Related]

  • 14.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 15. Dyssynchronous (non-uniform) Ca2+ release in myocytes from streptozotocin-induced diabetic rats.
    Shao CH, Rozanski GJ, Patel KP, Bidasee KR.
    J Mol Cell Cardiol; 2007 Jan 02; 42(1):234-46. PubMed ID: 17027851
    [Abstract] [Full Text] [Related]

  • 16. Adenovirus-mediated FKBP12.6 overexpression induces hypertrophy and apoptosis in cultured neonatal cardiomyocytes.
    Zhong J, Chen J, Cao T, Wang L, Zhang W, Liu D, Zhu Z.
    Clin Exp Pharmacol Physiol; 2009 Feb 02; 36(2):135-40. PubMed ID: 18759859
    [Abstract] [Full Text] [Related]

  • 17. Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release.
    Maier LS, Zhang T, Chen L, DeSantiago J, Brown JH, Bers DM.
    Circ Res; 2003 May 02; 92(8):904-11. PubMed ID: 12676813
    [Abstract] [Full Text] [Related]

  • 18.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 19. Metabolic inhibition alters subcellular calcium release patterns in rat ventricular myocytes: implications for defective excitation-contraction coupling during cardiac ischemia and failure.
    Fukumoto GH, Lamp ST, Motter C, Bridge JH, Garfinkel A, Goldhaber JI.
    Circ Res; 2005 Mar 18; 96(5):551-7. PubMed ID: 15718501
    [Abstract] [Full Text] [Related]

  • 20. Protein kinase A phosphorylation at serine-2808 of the cardiac Ca2+-release channel (ryanodine receptor) does not dissociate 12.6-kDa FK506-binding protein (FKBP12.6).
    Xiao B, Sutherland C, Walsh MP, Chen SR.
    Circ Res; 2004 Mar 05; 94(4):487-95. PubMed ID: 14715536
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 33.