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Journal Abstract Search


1060 related items for PubMed ID: 17891523

  • 1. Opening of mitochondrial permeability transition pore induces hypercontracture in Ca2+ overloaded cardiac myocytes.
    Ruiz-Meana M, Abellán A, Miró-Casas E, Garcia-Dorado D.
    Basic Res Cardiol; 2007 Nov; 102(6):542-52. PubMed ID: 17891523
    [Abstract] [Full Text] [Related]

  • 2. Role of mitochondrial re-energization and Ca2+ influx in reperfusion injury of metabolically inhibited cardiac myocytes.
    Rodrigo GC, Standen NB.
    Cardiovasc Res; 2005 Aug 01; 67(2):291-300. PubMed ID: 15885675
    [Abstract] [Full Text] [Related]

  • 3. Evidence that hydroxysafflor yellow A protects the heart against ischaemia-reperfusion injury by inhibiting mitochondrial permeability transition pore opening.
    Liu YN, Zhou ZM, Chen P.
    Clin Exp Pharmacol Physiol; 2008 Feb 01; 35(2):211-6. PubMed ID: 17941891
    [Abstract] [Full Text] [Related]

  • 4. Mitochondrial Ca2+ uptake during simulated ischemia does not affect permeability transition pore opening upon simulated reperfusion.
    Ruiz-Meana M, Garcia-Dorado D, Miró-Casas E, Abellán A, Soler-Soler J.
    Cardiovasc Res; 2006 Sep 01; 71(4):715-24. PubMed ID: 16860295
    [Abstract] [Full Text] [Related]

  • 5. Interplay between Ca2+ cycling and mitochondrial permeability transition pores promotes reperfusion-induced injury of cardiac myocytes.
    Abdallah Y, Kasseckert SA, Iraqi W, Said M, Shahzad T, Erdogan A, Neuhof C, Gündüz D, Schlüter KD, Tillmanns H, Piper HM, Reusch HP, Ladilov Y.
    J Cell Mol Med; 2011 Nov 01; 15(11):2478-85. PubMed ID: 21199327
    [Abstract] [Full Text] [Related]

  • 6. Local control of mitochondrial membrane potential, permeability transition pore and reactive oxygen species by calcium and calmodulin in rat ventricular myocytes.
    Odagiri K, Katoh H, Kawashima H, Tanaka T, Ohtani H, Saotome M, Urushida T, Satoh H, Hayashi H.
    J Mol Cell Cardiol; 2009 Jun 01; 46(6):989-97. PubMed ID: 19318235
    [Abstract] [Full Text] [Related]

  • 7. Signalling via the reperfusion injury signalling kinase (RISK) pathway links closure of the mitochondrial permeability transition pore to cardioprotection.
    Davidson SM, Hausenloy D, Duchen MR, Yellon DM.
    Int J Biochem Cell Biol; 2006 Mar 01; 38(3):414-9. PubMed ID: 16280253
    [Abstract] [Full Text] [Related]

  • 8. Intermittent high altitude hypoxia inhibits opening of mitochondrial permeability transition pores against reperfusion injury.
    Zhu WZ, Xie Y, Chen L, Yang HT, Zhou ZN.
    J Mol Cell Cardiol; 2006 Jan 01; 40(1):96-106. PubMed ID: 16288778
    [Abstract] [Full Text] [Related]

  • 9. Transient mitochondrial permeability transition pore opening mediates preconditioning-induced protection.
    Hausenloy D, Wynne A, Duchen M, Yellon D.
    Circulation; 2004 Apr 13; 109(14):1714-7. PubMed ID: 15066952
    [Abstract] [Full Text] [Related]

  • 10. Role of sarcoplasmic reticulum in mitochondrial permeability transition and cardiomyocyte death during reperfusion.
    Ruiz-Meana M, Abellán A, Miró-Casas E, Agulló E, Garcia-Dorado D.
    Am J Physiol Heart Circ Physiol; 2009 Oct 13; 297(4):H1281-9. PubMed ID: 19684187
    [Abstract] [Full Text] [Related]

  • 11. Cyclosporine A prevents apoptosis-related mitochondrial dysfunction after neonatal cardioplegic arrest.
    Oka N, Wang L, Mi W, Zhu W, Honjo O, Caldarone CA.
    J Thorac Cardiovasc Surg; 2008 Jan 13; 135(1):123-30, 130.e1-2. PubMed ID: 18179928
    [Abstract] [Full Text] [Related]

  • 12. Doxorubicin induced myocardial injury is exacerbated following ischaemic stress via opening of the mitochondrial permeability transition pore.
    Gharanei M, Hussain A, Janneh O, Maddock HL.
    Toxicol Appl Pharmacol; 2013 Apr 15; 268(2):149-56. PubMed ID: 23262186
    [Abstract] [Full Text] [Related]

  • 13. The mitochondrial permeability transition pore and the Ca2+-activated K+ channel contribute to the cardioprotection conferred by tumor necrosis factor-alpha.
    Gao Q, Zhang SZ, Cao CM, Bruce IC, Xia Q.
    Cytokine; 2005 Dec 07; 32(5):199-205. PubMed ID: 16260145
    [Abstract] [Full Text] [Related]

  • 14. Inhibition of Bcl-2 sensitizes mitochondrial permeability transition pore (MPTP) opening in ischemia-damaged mitochondria.
    Chen Q, Xu H, Xu A, Ross T, Bowler E, Hu Y, Lesnefsky EJ.
    PLoS One; 2015 Dec 07; 10(3):e0118834. PubMed ID: 25756500
    [Abstract] [Full Text] [Related]

  • 15. Adenosine A1 receptor activation reduces opening of mitochondrial permeability transition pores in hypoxic cardiomyocytes.
    Xiang F, Huang YS, Zhang DX, Chu ZG, Zhang JP, Zhang Q.
    Clin Exp Pharmacol Physiol; 2010 Mar 07; 37(3):343-9. PubMed ID: 19793110
    [Abstract] [Full Text] [Related]

  • 16. Oxidative stress, mitochondrial permeability transition pore opening and cell death during hypoxia-reoxygenation in adult cardiomyocytes.
    Assaly R, de Tassigny Ad, Paradis S, Jacquin S, Berdeaux A, Morin D.
    Eur J Pharmacol; 2012 Jan 30; 675(1-3):6-14. PubMed ID: 22173126
    [Abstract] [Full Text] [Related]

  • 17. Direct toxic effects of aqueous extract of cigarette smoke on cardiac myocytes at clinically relevant concentrations.
    Yamada S, Zhang XQ, Kadono T, Matsuoka N, Rollins D, Badger T, Rodesch CK, Barry WH.
    Toxicol Appl Pharmacol; 2009 Apr 01; 236(1):71-7. PubMed ID: 19371621
    [Abstract] [Full Text] [Related]

  • 18. Testosterone induces cytoprotection by activating ATP-sensitive K+ channels in the cardiac mitochondrial inner membrane.
    Er F, Michels G, Gassanov N, Rivero F, Hoppe UC.
    Circulation; 2004 Nov 09; 110(19):3100-7. PubMed ID: 15520315
    [Abstract] [Full Text] [Related]

  • 19. IL-6 induces PI 3-kinase and nitric oxide-dependent protection and preserves mitochondrial function in cardiomyocytes.
    Smart N, Mojet MH, Latchman DS, Marber MS, Duchen MR, Heads RJ.
    Cardiovasc Res; 2006 Jan 09; 69(1):164-77. PubMed ID: 16219301
    [Abstract] [Full Text] [Related]

  • 20. The neuroprotection conferred by activating the mitochondrial ATP-sensitive K+ channel is mediated by inhibiting the mitochondrial permeability transition pore.
    Wu L, Shen F, Lin L, Zhang X, Bruce IC, Xia Q.
    Neurosci Lett; 2006 Jul 10; 402(1-2):184-9. PubMed ID: 16678347
    [Abstract] [Full Text] [Related]


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