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Journal Abstract Search

397 related items for PubMed ID: 17932326

  • 21.
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  • 22. Effects of cardiomyopathy-linked mutations K15N and R21H in tropomyosin on thin-filament regulation and pointed-end dynamics.
    Ly T, Pappas CT, Johnson D, Schlecht W, Colpan M, Galkin VE, Gregorio CC, Dong WJ, Kostyukova AS.
    Mol Biol Cell; 2019 Jan 15; 30(2):268-281. PubMed ID: 30462572
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  • 25. Mutations in Troponin that cause HCM, DCM AND RCM: what can we learn about thin filament function?
    Willott RH, Gomes AV, Chang AN, Parvatiyar MS, Pinto JR, Potter JD.
    J Mol Cell Cardiol; 2010 May 15; 48(5):882-92. PubMed ID: 19914256
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  • 26. Alpha-tropomyosin mutations in inherited cardiomyopathies.
    Redwood C, Robinson P.
    J Muscle Res Cell Motil; 2013 Aug 15; 34(3-4):285-94. PubMed ID: 24005378
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  • 28. Desensitization of myofilaments to Ca2+ as a therapeutic target for hypertrophic cardiomyopathy with mutations in thin filament proteins.
    Alves ML, Dias FAL, Gaffin RD, Simon JN, Montminy EM, Biesiadecki BJ, Hinken AC, Warren CM, Utter MS, Davis RT, Sakthivel S, Robbins J, Wieczorek DF, Solaro RJ, Wolska BM.
    Circ Cardiovasc Genet; 2014 Apr 15; 7(2):132-143. PubMed ID: 24585742
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  • 30. HCM and DCM cardiomyopathy-linked α-tropomyosin mutations influence off-state stability and crossbridge interaction on thin filaments.
    Farman GP, Rynkiewicz MJ, Orzechowski M, Lehman W, Moore JR.
    Arch Biochem Biophys; 2018 Jun 01; 647():84-92. PubMed ID: 29626422
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  • 31. Effects of the cardiomyopathy-causing E244D mutation of troponin T on the structures of cardiac thin filaments studied by small-angle X-ray scattering.
    Matsuo T, Kono F, Fujiwara S.
    J Struct Biol; 2019 Feb 01; 205(2):196-205. PubMed ID: 30599212
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  • 32. Functional Characterization of Cardiac Actin Mutants Causing Hypertrophic (p.A295S) and Dilated Cardiomyopathy (p.R312H and p.E361G).
    Hassoun R, Erdmann C, Schmitt S, Fujita-Becker S, Mügge A, Schröder RR, Geyer M, Borbor M, Jaquet K, Hamdani N, Mannherz HG.
    Int J Mol Sci; 2022 Apr 18; 23(8):. PubMed ID: 35457283
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  • 33. Human actin mutations associated with hypertrophic and dilated cardiomyopathies demonstrate distinct thin filament regulatory properties in vitro.
    Debold EP, Saber W, Cheema Y, Bookwalter CS, Trybus KM, Warshaw DM, Vanburen P.
    J Mol Cell Cardiol; 2010 Feb 18; 48(2):286-92. PubMed ID: 19799913
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  • 35. A novel alpha-tropomyosin mutation associates with dilated and non-compaction cardiomyopathy and diminishes actin binding.
    van de Meerakker JB, Christiaans I, Barnett P, Lekanne Deprez RH, Ilgun A, Mook OR, Mannens MM, Lam J, Wilde AA, Moorman AF, Postma AV.
    Biochim Biophys Acta; 2013 Apr 18; 1833(4):833-9. PubMed ID: 23147248
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  • 38. A novel mutant cardiac troponin C disrupts molecular motions critical for calcium binding affinity and cardiomyocyte contractility.
    Lim CC, Yang H, Yang M, Wang CK, Shi J, Berg EA, Pimentel DR, Gwathmey JK, Hajjar RJ, Helmes M, Costello CE, Huo S, Liao R.
    Biophys J; 2008 May 01; 94(9):3577-89. PubMed ID: 18212018
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  • 39. Two mutations in troponin I that cause hypertrophic cardiomyopathy have contrasting effects on cardiac muscle contractility.
    Burton D, Abdulrazzak H, Knott A, Elliott K, Redwood C, Watkins H, Marston S, Ashley C.
    Biochem J; 2002 Mar 01; 362(Pt 2):443-51. PubMed ID: 11853553
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  • 40. The effect of mutations in alpha-tropomyosin (E40K and E54K) that cause familial dilated cardiomyopathy on the regulatory mechanism of cardiac muscle thin filaments.
    Mirza M, Robinson P, Kremneva E, Copeland O, Nikolaeva O, Watkins H, Levitsky D, Redwood C, El-Mezgueldi M, Marston S.
    J Biol Chem; 2007 May 04; 282(18):13487-97. PubMed ID: 17360712
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